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Items: 5

1.

Modeling Axonal Defects in Hereditary Spastic Paraplegia with Human Pluripotent Stem Cells.

Denton KR, Xu C, Shah H, Li XJ.

Front Biol (Beijing). 2016 Oct;11(5):339-354. Epub 2016 Sep 28.

2.

Abnormal mitochondrial transport and morphology as early pathological changes in human models of spinal muscular atrophy.

Xu CC, Denton KR, Wang ZB, Zhang X, Li XJ.

Dis Model Mech. 2016 Jan;9(1):39-49. doi: 10.1242/dmm.021766. Epub 2015 Nov 19.

3.

Modeling Axonal Phenotypes with Human Pluripotent Stem Cells.

Denton KR, Xu CC, Li XJ.

Methods Mol Biol. 2016;1353:309-21. doi: 10.1007/7651_2014_167.

PMID:
25520289
4.

Pharmacologic rescue of axon growth defects in a human iPSC model of hereditary spastic paraplegia SPG3A.

Zhu PP, Denton KR, Pierson TM, Li XJ, Blackstone C.

Hum Mol Genet. 2014 Nov 1;23(21):5638-48. doi: 10.1093/hmg/ddu280. Epub 2014 Jun 6.

5.

Loss of spastin function results in disease-specific axonal defects in human pluripotent stem cell-based models of hereditary spastic paraplegia.

Denton KR, Lei L, Grenier J, Rodionov V, Blackstone C, Li XJ.

Stem Cells. 2014 Feb;32(2):414-23. doi: 10.1002/stem.1569.

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