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Items: 10

1.

Observing the cell in its native state: Imaging subcellular dynamics in multicellular organisms.

Liu TL, Upadhyayula S, Milkie DE, Singh V, Wang K, Swinburne IA, Mosaliganti KR, Collins ZM, Hiscock TW, Shea J, Kohrman AQ, Medwig TN, Dambournet D, Forster R, Cunniff B, Ruan Y, Yashiro H, Scholpp S, Meyerowitz EM, Hockemeyer D, Drubin DG, Martin BL, Matus DQ, Koyama M, Megason SG, Kirchhausen T, Betzig E.

Science. 2018 Apr 20;360(6386). pii: eaaq1392. doi: 10.1126/science.aaq1392.

PMID:
29674564
2.

Miro1-mediated mitochondrial positioning shapes intracellular energy gradients required for cell migration.

Schuler MH, Lewandowska A, Caprio GD, Skillern W, Upadhyayula S, Kirchhausen T, Shaw JM, Cunniff B.

Mol Biol Cell. 2017 Aug 1;28(16):2159-2169. doi: 10.1091/mbc.E16-10-0741. Epub 2017 Jun 14.

3.

AMPK activity regulates trafficking of mitochondria to the leading edge during cell migration and matrix invasion.

Cunniff B, McKenzie AJ, Heintz NH, Howe AK.

Mol Biol Cell. 2016 Sep 1;27(17):2662-74. doi: 10.1091/mbc.E16-05-0286. Epub 2016 Jul 6.

4.

Disabling Mitochondrial Peroxide Metabolism via Combinatorial Targeting of Peroxiredoxin 3 as an Effective Therapeutic Approach for Malignant Mesothelioma.

Cunniff B, Newick K, Nelson KJ, Wozniak AN, Beuschel S, Leavitt B, Bhave A, Butnor K, Koenig A, Chouchani ET, James AM, Haynes AC, Lowther WT, Murphy MP, Shukla A, Heintz NH.

PLoS One. 2015 May 26;10(5):e0127310. doi: 10.1371/journal.pone.0127310. eCollection 2015.

5.

Peroxiredoxin 3 levels regulate a mitochondrial redox setpoint in malignant mesothelioma cells.

Cunniff B, Wozniak AN, Sweeney P, DeCosta K, Heintz NH.

Redox Biol. 2014;3:79-87. doi: 10.1016/j.redox.2014.11.003. Epub 2014 Nov 18.

6.

Resolution of oxidative stress by thioredoxin reductase: Cysteine versus selenocysteine.

Cunniff B, Snider GW, Fredette N, Stumpff J, Hondal RJ, Heintz NH.

Redox Biol. 2014 Feb 19;2:475-84. doi: 10.1016/j.redox.2014.01.021. eCollection 2014.

7.

A direct and continuous assay for the determination of thioredoxin reductase activity in cell lysates.

Cunniff B, Snider GW, Fredette N, Hondal RJ, Heintz NH.

Anal Biochem. 2013 Dec 1;443(1):34-40. doi: 10.1016/j.ab.2013.08.013. Epub 2013 Aug 21.

8.

Mitochondrial-targeted nitroxides disrupt mitochondrial architecture and inhibit expression of peroxiredoxin 3 and FOXM1 in malignant mesothelioma cells.

Cunniff B, Benson K, Stumpff J, Newick K, Held P, Taatjes D, Joseph J, Kalyanaraman B, Heintz NH.

J Cell Physiol. 2013 Apr;228(4):835-45. doi: 10.1002/jcp.24232.

9.

Peroxiredoxin 3 is a redox-dependent target of thiostrepton in malignant mesothelioma cells.

Newick K, Cunniff B, Preston K, Held P, Arbiser J, Pass H, Mossman B, Shukla A, Heintz N.

PLoS One. 2012;7(6):e39404. doi: 10.1371/journal.pone.0039404. Epub 2012 Jun 25.

10.

Oxidative processing of latent Fas in the endoplasmic reticulum controls the strength of apoptosis.

Anathy V, Roberson E, Cunniff B, Nolin JD, Hoffman S, Spiess P, Guala AS, Lahue KG, Goldman D, Flemer S, van der Vliet A, Heintz NH, Budd RC, Tew KD, Janssen-Heininger YM.

Mol Cell Biol. 2012 Sep;32(17):3464-78. doi: 10.1128/MCB.00125-12. Epub 2012 Jul 2.

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