Format
Sort by
Items per page

Send to

Choose Destination

Search results

Items: 17

1.

Aberrant accrual of BIN1 near Alzheimer's disease amyloid deposits in transgenic models.

De Rossi P, Andrew RJ, Musial TF, Buggia-Prevot V, Xu G, Ponnusamy M, Ly H, Krause SV, Rice RC, de l'Estoile V, Valin T, Salem S, Despa F, Borchelt DR, Bindokas VP, Nicholson DA, Thinakaran G.

Brain Pathol. 2019 Jul;29(4):485-501. doi: 10.1111/bpa.12687. Epub 2018 Dec 27.

2.

BIN1 localization is distinct from Tau tangles in Alzheimer's disease.

De Rossi P, Buggia-Prevot V, Andrew RJ, Krause SV, Woo E, Nelson PT, Pytel P, Thinakaran G.

Matters (Zur). 2017 Jan;2017. doi: 10.19185/matters.201611000018. Epub 2017 Jan 12.

3.

Lack of BACE1 S-palmitoylation reduces amyloid burden and mitigates memory deficits in transgenic mouse models of Alzheimer's disease.

Andrew RJ, Fernandez CG, Stanley M, Jiang H, Nguyen P, Rice RC, Buggia-Prévot V, De Rossi P, Vetrivel KS, Lamb R, Argemi A, Allaert ES, Rathbun EM, Krause SV, Wagner SL, Parent AT, Holtzman DM, Thinakaran G.

Proc Natl Acad Sci U S A. 2017 Nov 7;114(45):E9665-E9674. doi: 10.1073/pnas.1708568114. Epub 2017 Oct 23.

4.

Predominant expression of Alzheimer's disease-associated BIN1 in mature oligodendrocytes and localization to white matter tracts.

De Rossi P, Buggia-Prévot V, Clayton BL, Vasquez JB, van Sanford C, Andrew RJ, Lesnick R, Botté A, Deyts C, Salem S, Rao E, Rice RC, Parent A, Kar S, Popko B, Pytel P, Estus S, Thinakaran G.

Mol Neurodegener. 2016 Aug 3;11(1):59. doi: 10.1186/s13024-016-0124-1.

5.

Presynaptic dystrophic neurites surrounding amyloid plaques are sites of microtubule disruption, BACE1 elevation, and increased Aβ generation in Alzheimer's disease.

Sadleir KR, Kandalepas PC, Buggia-Prévot V, Nicholson DA, Thinakaran G, Vassar R.

Acta Neuropathol. 2016 Aug;132(2):235-56. doi: 10.1007/s00401-016-1558-9. Epub 2016 Mar 18.

6.

Nuclear factor-κB regulates βAPP and β- and γ-secretases differently at physiological and supraphysiological Aβ concentrations.

Chami L, Buggia-Prévot V, Duplan E, Del Prete D, Chami M, Peyron JF, Checler F.

J Biol Chem. 2015 Dec 11;290(50):29758. doi: 10.1074/jbc.A115.333054. No abstract available.

7.

Significance of transcytosis in Alzheimer's disease: BACE1 takes the scenic route to axons.

Buggia-Prévot V, Thinakaran G.

Bioessays. 2015 Aug;37(8):888-98. doi: 10.1002/bies.201500019. Epub 2015 Jun 30.

8.

Overexpression of the Insulin-Like Growth Factor II Receptor Increases β-Amyloid Production and Affects Cell Viability.

Wang Y, Buggia-Prévot V, Zavorka ME, Bleackley RC, MacDonald RG, Thinakaran G, Kar S.

Mol Cell Biol. 2015 Jul;35(14):2368-84. doi: 10.1128/MCB.01338-14. Epub 2015 May 4.

9.

[Alzheimer disease and endosomal traffic in neurons].

Buggia-Prévot V.

Med Sci (Paris). 2014 Jun-Jul;30(6-7):613-5. doi: 10.1051/medsci/20143006005. Epub 2014 Jul 11. French. No abstract available.

10.

Sorting the role of SORLA in Alzheimer's disease.

Buggia-Prévot V, Thinakaran G.

Sci Transl Med. 2014 Feb 12;6(223):223fs8. doi: 10.1126/scitranslmed.3008562.

PMID:
24523319
11.

Axonal BACE1 dynamics and targeting in hippocampal neurons: a role for Rab11 GTPase.

Buggia-Prévot V, Fernandez CG, Riordan S, Vetrivel KS, Roseman J, Waters J, Bindokas VP, Vassar R, Thinakaran G.

Mol Neurodegener. 2014 Jan 4;9:1. doi: 10.1186/1750-1326-9-1.

12.

A function for EHD family proteins in unidirectional retrograde dendritic transport of BACE1 and Alzheimer's disease Aβ production.

Buggia-Prévot V, Fernandez CG, Udayar V, Vetrivel KS, Elie A, Roseman J, Sasse VA, Lefkow M, Meckler X, Bhattacharyya S, George M, Kar S, Bindokas VP, Parent AT, Rajendran L, Band H, Vassar R, Thinakaran G.

Cell Rep. 2013 Dec 26;5(6):1552-63. doi: 10.1016/j.celrep.2013.12.006.

13.

A paired RNAi and RabGAP overexpression screen identifies Rab11 as a regulator of β-amyloid production.

Udayar V, Buggia-Prévot V, Guerreiro RL, Siegel G, Rambabu N, Soohoo AL, Ponnusamy M, Siegenthaler B, Bali J; AESG, Simons M, Ries J, Puthenveedu MA, Hardy J, Thinakaran G, Rajendran L.

Cell Rep. 2013 Dec 26;5(6):1536-51. doi: 10.1016/j.celrep.2013.12.005.

14.

Ca2+ influx through store-operated Ca2+ channels reduces Alzheimer disease β-amyloid peptide secretion.

Zeiger W, Vetrivel KS, Buggia-Prévot V, Nguyen PD, Wagner SL, Villereal ML, Thinakaran G.

J Biol Chem. 2013 Sep 13;288(37):26955-66. doi: 10.1074/jbc.M113.473355. Epub 2013 Jul 31.

15.

Nuclear factor-κB regulates βAPP and β- and γ-secretases differently at physiological and supraphysiological Aβ concentrations.

Chami L, Buggia-Prévot V, Duplan E, Del Prete D, Chami M, Peyron JF, Checler F.

J Biol Chem. 2012 Jul 13;287(29):24573-84. doi: 10.1074/jbc.M111.333054. Epub 2012 May 31. Erratum in: J Biol Chem. 2013 Jul 5;288(27):19645. J Biol Chem. 2015 Dec 11;290(50):29758. Delprete, Dolores [corrected to Del Prete, Dolores].

16.

De novo and long-term l-Dopa induce both common and distinct striatal gene profiles in the hemiparkinsonian rat.

El Atifi-Borel M, Buggia-Prevot V, Platet N, Benabid AL, Berger F, Sgambato-Faure V.

Neurobiol Dis. 2009 May;34(2):340-50. doi: 10.1016/j.nbd.2009.02.002. Epub 2009 Feb 20.

PMID:
19233275
17.

NFkappaB-dependent control of BACE1 promoter transactivation by Abeta42.

Buggia-Prevot V, Sevalle J, Rossner S, Checler F.

J Biol Chem. 2008 Apr 11;283(15):10037-47. doi: 10.1074/jbc.M706579200. Epub 2008 Feb 8.

Supplemental Content

Loading ...
Support Center