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Items: 17

1.

Flu Shows the Power of Diversity.

Martin BE, Brooke CB.

Cell. 2019 Jan 10;176(1-2):9-10. doi: 10.1016/j.cell.2018.12.017.

PMID:
30633911
2.

Causes and Consequences of Spatial Within-Host Viral Spread.

Gallagher ME, Brooke CB, Ke R, Koelle K.

Viruses. 2018 Nov 13;10(11). pii: E627. doi: 10.3390/v10110627. Review.

3.

Influenza A Virus Superinfection Potential Is Regulated by Viral Genomic Heterogeneity.

Sun J, Brooke CB.

MBio. 2018 Oct 30;9(5). pii: e01761-18. doi: 10.1128/mBio.01761-18.

4.

The parts are greater than the whole: the role of semi-infectious particles in influenza A virus biology.

Diefenbacher M, Sun J, Brooke CB.

Curr Opin Virol. 2018 Dec;33:42-46. doi: 10.1016/j.coviro.2018.07.002. Epub 2018 Jul 24. Review.

PMID:
30053722
5.

Correction: Influenza A virus hemagglutinin glycosylation compensates for antibody escape fitness costs.

Kosik I, Ince WL, Gentles LE, Oler AJ, Kosikova M, Angel M, Magadán JG, Xie H, Brooke CB, Yewdell JW.

PLoS Pathog. 2018 Jun 20;14(6):e1007141. doi: 10.1371/journal.ppat.1007141. eCollection 2018 Jun.

6.

Influenza A virus hemagglutinin glycosylation compensates for antibody escape fitness costs.

Kosik I, Ince WL, Gentles LE, Oler AJ, Kosikova M, Angel M, Magadán JG, Xie H, Brooke CB, Yewdell JW.

PLoS Pathog. 2018 Jan 18;14(1):e1006796. doi: 10.1371/journal.ppat.1006796. eCollection 2018 Jan. Erratum in: PLoS Pathog. 2018 Jun 20;14(6):e1007141.

7.

Population Diversity and Collective Interactions during Influenza Virus Infection.

Brooke CB.

J Virol. 2017 Oct 27;91(22). pii: e01164-17. doi: 10.1128/JVI.01164-17. Print 2017 Nov 15. Review.

8.

Influenza A virus nucleoprotein selectively decreases neuraminidase gene-segment packaging while enhancing viral fitness and transmissibility.

Brooke CB, Ince WL, Wei J, Bennink JR, Yewdell JW.

Proc Natl Acad Sci U S A. 2014 Nov 25;111(47):16854-9. doi: 10.1073/pnas.1415396111. Epub 2014 Nov 10.

9.
10.

Defining influenza A virus hemagglutinin antigenic drift by sequential monoclonal antibody selection.

Das SR, Hensley SE, Ince WL, Brooke CB, Subba A, Delboy MG, Russ G, Gibbs JS, Bennink JR, Yewdell JW.

Cell Host Microbe. 2013 Mar 13;13(3):314-23. doi: 10.1016/j.chom.2013.02.008.

11.

Host versus flu: antibodies win a round?

Brooke CB, Yewdell JW.

Nat Struct Mol Biol. 2013 Mar;20(3):245-6. doi: 10.1038/nsmb.2524. No abstract available.

12.

Most influenza a virions fail to express at least one essential viral protein.

Brooke CB, Ince WL, Wrammert J, Ahmed R, Wilson PC, Bennink JR, Yewdell JW.

J Virol. 2013 Mar;87(6):3155-62. doi: 10.1128/JVI.02284-12. Epub 2013 Jan 2. Erratum in: J Virol. 2013 Jul;87(14):8267.

13.

Monocytes, viruses and metaphors: hanging the Trojan horse.

Yewdell JW, Brooke CB.

Cell Cycle. 2012 May 1;11(9):1748-9. doi: 10.4161/cc.20311. Epub 2012 May 1. No abstract available.

14.

Viral infection triggers rapid differentiation of human blood monocytes into dendritic cells.

Hou W, Gibbs JS, Lu X, Brooke CB, Roy D, Modlin RL, Bennink JR, Yewdell JW.

Blood. 2012 Mar 29;119(13):3128-31. doi: 10.1182/blood-2011-09-379479. Epub 2012 Feb 6.

15.

Early activation of the host complement system is required to restrict central nervous system invasion and limit neuropathology during Venezuelan equine encephalitis virus infection.

Brooke CB, Schäfer A, Matsushima GK, White LJ, Johnston RE.

J Gen Virol. 2012 Apr;93(Pt 4):797-806. doi: 10.1099/vir.0.038281-0. Epub 2011 Dec 28.

16.

The role of the blood-brain barrier during Venezuelan equine encephalitis virus infection.

Schäfer A, Brooke CB, Whitmore AC, Johnston RE.

J Virol. 2011 Oct;85(20):10682-90. doi: 10.1128/JVI.05032-11. Epub 2011 Aug 17.

17.

T cells facilitate recovery from Venezuelan equine encephalitis virus-induced encephalomyelitis in the absence of antibody.

Brooke CB, Deming DJ, Whitmore AC, White LJ, Johnston RE.

J Virol. 2010 May;84(9):4556-68. doi: 10.1128/JVI.02545-09. Epub 2010 Feb 24.

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