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Items: 17

1.

Type I interferon induces CXCL13 to support ectopic germinal center formation.

Denton AE, Innocentin S, Carr EJ, Bradford BM, Lafouresse F, Mabbott NA, Mörbe U, Ludewig B, Groom JR, Good-Jacobson KL, Linterman MA.

J Exp Med. 2019 Feb 5. pii: jem.20181216. doi: 10.1084/jem.20181216. [Epub ahead of print]

PMID:
30723095
2.

Oral Prion Neuroinvasion Occurs Independently of PrPC Expression in the Gut Epithelium.

Marshall A, Bradford BM, Clarke AR, Manson JC, Mabbott NA.

J Virol. 2018 Sep 12;92(19). pii: e01010-18. doi: 10.1128/JVI.01010-18. Print 2018 Oct 1.

3.

Increased susceptibility to oral Trichuris muris infection in the specific absence of CXCR5+ CD11c+ cells.

Bradford BM, Donaldson DS, Forman R, Else KJ, Mabbott NA.

Parasite Immunol. 2018 Aug;40(8):e12566. doi: 10.1111/pim.12566.

4.

Prion disease pathogenesis in the absence of the commensal microbiota.

Bradford BM, Tetlow L, Mabbott NA.

J Gen Virol. 2017 Jul;98(7):1943-1952. doi: 10.1099/jgv.0.000860. Epub 2017 Jul 15.

5.

Oral Prion Disease Pathogenesis Is Impeded in the Specific Absence of CXCR5-Expressing Dendritic Cells.

Bradford BM, Reizis B, Mabbott NA.

J Virol. 2017 Apr 28;91(10). pii: e00124-17. doi: 10.1128/JVI.00124-17. Print 2017 May 15.

6.

Prion pathogenesis is unaltered following down-regulation of SIGN-R1.

Bradford BM, Brown KL, Mabbott NA.

Virology. 2016 Oct;497:337-345. doi: 10.1016/j.virol.2016.08.005. Epub 2016 Aug 11.

7.

The Good, the Bad, and the Ugly of Dendritic Cells during Prion Disease.

Mabbott NA, Bradford BM.

J Immunol Res. 2015;2015:168574. doi: 10.1155/2015/168574. Epub 2015 Nov 30. Review.

8.

Reciprocal regulation of lymphoid tissue development in the large intestine by IL-25 and IL-23.

Donaldson DS, Bradford BM, Artis D, Mabbott NA.

Mucosal Immunol. 2015 May;8(3):582-95. doi: 10.1038/mi.2014.90. Epub 2014 Sep 24.

9.

Human prion diseases and the risk of their transmission during anatomical dissection.

Bradford BM, Piccardo P, Ironside JW, Mabbott NA.

Clin Anat. 2014 Sep;27(6):821-32. doi: 10.1002/ca.22403. Epub 2014 Apr 17. Review.

PMID:
24740900
10.

Aging and the mucosal immune system in the intestine.

Mabbott NA, Kobayashi A, Sehgal A, Bradford BM, Pattison M, Donaldson DS.

Biogerontology. 2015 Apr;16(2):133-45. doi: 10.1007/s10522-014-9498-z. Epub 2014 Apr 5. Review.

PMID:
24705962
11.

Peripheral prion disease pathogenesis is unaltered in the absence of sialoadhesin (Siglec-1/CD169).

Bradford BM, Crocker PR, Mabbott NA.

Immunology. 2014 Sep;143(1):120-9. doi: 10.1111/imm.12294.

12.

Pleiotropic effects of extended blockade of CSF1R signaling in adult mice.

Sauter KA, Pridans C, Sehgal A, Tsai YT, Bradford BM, Raza S, Moffat L, Gow DJ, Beard PM, Mabbott NA, Smith LB, Hume DA.

J Leukoc Biol. 2014 Aug;96(2):265-74. doi: 10.1189/jlb.2A0114-006R. Epub 2014 Mar 20.

13.

Prion disease and the innate immune system.

Bradford BM, Mabbott NA.

Viruses. 2012 Dec;4(12):3389-419. Review.

14.

Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen.

McCulloch L, Brown KL, Bradford BM, Hopkins J, Bailey M, Rajewsky K, Manson JC, Mabbott NA.

PLoS Pathog. 2011 Dec;7(12):e1002402. doi: 10.1371/journal.ppat.1002402. Epub 2011 Dec 1.

16.

Glycosylation of PrPC determines timing of neuroinvasion and targeting in the brain following transmissible spongiform encephalopathy infection by a peripheral route.

Cancellotti E, Bradford BM, Tuzi NL, Hickey RD, Brown D, Brown KL, Barron RM, Kisielewski D, Piccardo P, Manson JC.

J Virol. 2010 Apr;84(7):3464-75. doi: 10.1128/JVI.02374-09. Epub 2010 Jan 27.

17.

Dramatic reduction of PrP C level and glycosylation in peripheral nerves following PrP knock-out from Schwann cells does not prevent transmissible spongiform encephalopathy neuroinvasion.

Bradford BM, Tuzi NL, Feltri ML, McCorquodale C, Cancellotti E, Manson JC.

J Neurosci. 2009 Dec 9;29(49):15445-54. doi: 10.1523/JNEUROSCI.4195-09.2009.

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