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Items: 8


Mice lacking sodium channel beta1 subunits display defects in neuronal excitability, sodium channel expression, and nodal architecture.

Chen C, Westenbroek RE, Xu X, Edwards CA, Sorenson DR, Chen Y, McEwen DP, O'Malley HA, Bharucha V, Meadows LS, Knudsen GA, Vilaythong A, Noebels JL, Saunders TL, Scheuer T, Shrager P, Catterall WA, Isom LL.

J Neurosci. 2004 Apr 21;24(16):4030-42.


A novel form of autosomal recessive pure hereditary spastic paraplegia maps to chromosome 13q14.

Hodgkinson CA, Bohlega S, Abu-Amero SN, Cupler E, Kambouris M, Meyer BF, Bharucha VA.

Neurology. 2002 Dec 24;59(12):1905-9.


Reduced sodium channel density, altered voltage dependence of inactivation, and increased susceptibility to seizures in mice lacking sodium channel beta 2-subunits.

Chen C, Bharucha V, Chen Y, Westenbroek RE, Brown A, Malhotra JD, Jones D, Avery C, Gillespie PJ 3rd, Kazen-Gillespie KA, Kazarinova-Noyes K, Shrager P, Saunders TL, Macdonald RL, Ransom BR, Scheuer T, Catterall WA, Isom LL.

Proc Natl Acad Sci U S A. 2002 Dec 24;99(26):17072-7. Epub 2002 Dec 12.


Characterization of the cis-acting elements of the mouse myelin P2 promoter.

Bharucha VA, Peden KW, Subach BR, Narayanan V, Tennekoon GI.

J Neurosci Res. 1993 Dec 1;36(5):508-19.


Increased P0 glycoprotein gene expression in primary and transfected rat Schwann cells after treatment with axolemma-enriched fraction.

Knight RM, Fossom LH, Neuberger TJ, Attema BL, Tennekoon G, Bharucha V, DeVries GH.

J Neurosci Res. 1993 May 1;35(1):38-45.


GM1 ganglioside treatment reduces functional deficits associated with cortical focal ischemia.

Bharucha VA, Wakade CG, Mahadik SP, Karpiak SE.

Exp Neurol. 1991 Oct;114(1):136-9. No abstract available.


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