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Items: 6

1.

HIV-1 seroreversion in HIV-1-infected children: do genetic determinants play a role?

Asang C, Laws HJ, Adams O, Enczmann J, Feiterna-Sperling C, Notheis G, Buchholz B, Borkhardt A, Neubert J.

AIDS. 2014 Feb 20;28(4):543-7. doi: 10.1097/QAD.0000000000000065.

PMID:
24056069
2.

Next-generation-sequencing-spectratyping reveals public T-cell receptor repertoires in pediatric very severe aplastic anemia and identifies a β chain CDR3 sequence associated with hepatitis-induced pathogenesis.

Krell PF, Reuther S, Fischer U, Keller T, Weber S, Gombert M, Schuster FR, Asang C, Stepensky P, Strahm B, Meisel R, Stoye J, Borkhardt A.

Haematologica. 2013 Sep;98(9):1388-96. doi: 10.3324/haematol.2012.069708. Epub 2013 May 28.

3.

The HIV-1 major splice donor D1 is activated by splicing enhancer elements within the leader region and the p17-inhibitory sequence.

Asang C, Erkelenz S, Schaal H.

Virology. 2012 Oct 10;432(1):133-45. doi: 10.1016/j.virol.2012.06.004. Epub 2012 Jun 30.

4.

Insights into the selective activation of alternatively used splice acceptors by the human immunodeficiency virus type-1 bidirectional splicing enhancer.

Asang C, Hauber I, Schaal H.

Nucleic Acids Res. 2008 Mar;36(5):1450-63. doi: 10.1093/nar/gkm1147. Epub 2008 Jan 18.

5.
6.

A novel approach to describe a U1 snRNA binding site.

Freund M, Asang C, Kammler S, Konermann C, Krummheuer J, Hipp M, Meyer I, Gierling W, Theiss S, Preuss T, Schindler D, Kjems J, Schaal H.

Nucleic Acids Res. 2003 Dec 1;31(23):6963-75.

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