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Items: 14

1.

A three-dimensional skin equivalent reflecting some aspects of in vivo aged skin.

Diekmann J, Alili L, Scholz O, Giesen M, Holtkötter O, Brenneisen P.

Exp Dermatol. 2016 Jan;25(1):56-61. doi: 10.1111/exd.12866. Epub 2015 Nov 23.

PMID:
26440058
2.

Effect of Fe3O4 Nanoparticles on Skin Tumor Cells and Dermal Fibroblasts.

Alili L, Chapiro S, Marten GU, Schmidt AM, Zanger K, Brenneisen P.

Biomed Res Int. 2015;2015:530957. doi: 10.1155/2015/530957. Epub 2015 May 21.

3.

Redox-active cerium oxide nanoparticles protect human dermal fibroblasts from PQ-induced damage.

von Montfort C, Alili L, Teuber-Hanselmann S, Brenneisen P.

Redox Biol. 2015;4:1-5. doi: 10.1016/j.redox.2014.11.007. Epub 2014 Nov 22.

4.

A drug-induced accelerated senescence (DIAS) is a possibility to study aging in time lapse.

Alili L, Diekmann J, Giesen M, Holtkötter O, Brenneisen P.

Age (Dordr). 2014 Jun;36(3):9658. doi: 10.1007/s11357-014-9658-8. Epub 2014 May 16.

5.

Combination of conventional chemotherapeutics with redox-active cerium oxide nanoparticles--a novel aspect in cancer therapy.

Sack M, Alili L, Karaman E, Das S, Gupta A, Seal S, Brenneisen P.

Mol Cancer Ther. 2014 Jul;13(7):1740-9. doi: 10.1158/1535-7163.MCT-13-0950. Epub 2014 May 13.

6.

Fibroblast-to-myofibroblast switch is mediated by NAD(P)H oxidase generated reactive oxygen species.

Alili L, Sack M, Puschmann K, Brenneisen P.

Biosci Rep. 2014 Feb 1;34(1). pii: e00089. Print 2014 Feb 1.

7.

Downregulation of tumor growth and invasion by redox-active nanoparticles.

Alili L, Sack M, von Montfort C, Giri S, Das S, Carroll KS, Zanger K, Seal S, Brenneisen P.

Antioxid Redox Signal. 2013 Sep 10;19(8):765-78. doi: 10.1089/ars.2012.4831. Epub 2013 Jan 24.

8.

Combined cytotoxic and anti-invasive properties of redox-active nanoparticles in tumor-stroma interactions.

Alili L, Sack M, Karakoti AS, Teuber S, Puschmann K, Hirst SM, Reilly CM, Zanger K, Stahl W, Das S, Seal S, Brenneisen P.

Biomaterials. 2011 Apr;32(11):2918-29. doi: 10.1016/j.biomaterials.2010.12.056. Epub 2011 Jan 26.

PMID:
21269688
9.

Selenoprotein P expression is controlled through interaction of the coactivator PGC-1alpha with FoxO1a and hepatocyte nuclear factor 4alpha transcription factors.

Speckmann B, Walter PL, Alili L, Reinehr R, Sies H, Klotz LO, Steinbrenner H.

Hepatology. 2008 Dec;48(6):1998-2006. doi: 10.1002/hep.22526.

PMID:
18972406
10.

Stromal resistance of fibroblasts against oxidative damage: involvement of tumor cell-secreted platelet-derived growth factor (PDGF) and phosphoinositide 3-kinase (PI3K) activation.

Werth C, Stuhlmann D, Cat B, Steinbrenner H, Alili L, Sies H, Brenneisen P.

Carcinogenesis. 2008 Feb;29(2):404-10. doi: 10.1093/carcin/bgm296. Epub 2008 Jan 3.

PMID:
18174235
11.

Post-translational processing of selenoprotein P: implications of glycosylation for its utilisation by target cells.

Steinbrenner H, Alili L, Stuhlmann D, Sies H, Brenneisen P.

Biol Chem. 2007 Oct;388(10):1043-51.

PMID:
17937618
12.

Selenoprotein P protects endothelial cells from oxidative damage by stimulation of glutathione peroxidase expression and activity.

Steinbrenner H, Bilgic E, Alili L, Sies H, Brenneisen P.

Free Radic Res. 2006 Sep;40(9):936-43.

PMID:
17015273
13.

Enhancement of tumor invasion depends on transdifferentiation of skin fibroblasts mediated by reactive oxygen species.

Cat B, Stuhlmann D, Steinbrenner H, Alili L, Holtkötter O, Sies H, Brenneisen P.

J Cell Sci. 2006 Jul 1;119(Pt 13):2727-38. Epub 2006 Jun 6.

14.

Involvement of selenoprotein P in protection of human astrocytes from oxidative damage.

Steinbrenner H, Alili L, Bilgic E, Sies H, Brenneisen P.

Free Radic Biol Med. 2006 May 1;40(9):1513-23. Epub 2006 Jan 13.

PMID:
16632112

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