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Items: 5

1.

A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection.

Bristol JA, Djavadian R, Albright ER, Coleman CB, Ohashi M, Hayes M, Romero-Masters JC, Barlow EA, Farrell PJ, Rochford R, Kalejta RF, Johannsen EC, Kenney SC.

PLoS Pathog. 2018 Jul 27;14(7):e1007179. doi: 10.1371/journal.ppat.1007179. eCollection 2018 Jul.

2.

Canonical and Variant Forms of Histone H3 Are Deposited onto the Human Cytomegalovirus Genome during Lytic and Latent Infections.

Albright ER, Kalejta RF.

J Virol. 2016 Oct 28;90(22):10309-10320. doi: 10.1128/JVI.01220-16. Print 2016 Nov 15.

3.

Long and Short Isoforms of the Human Cytomegalovirus UL138 Protein Silence IE Transcription and Promote Latency.

Lee SH, Caviness K, Albright ER, Lee JH, Gelbmann CB, Rak M, Goodrum F, Kalejta RF.

J Virol. 2016 Sep 29;90(20):9483-94. doi: 10.1128/JVI.01547-16. Print 2016 Oct 15.

4.

Cellular defense against latent colonization foiled by human cytomegalovirus UL138 protein.

Lee SH, Albright ER, Lee JH, Jacobs D, Kalejta RF.

Sci Adv. 2015 Nov 27;1(10):e1501164. doi: 10.1126/sciadv.1501164. eCollection 2015 Nov.

5.

Myeloblastic cell lines mimic some but not all aspects of human cytomegalovirus experimental latency defined in primary CD34+ cell populations.

Albright ER, Kalejta RF.

J Virol. 2013 Sep;87(17):9802-12. doi: 10.1128/JVI.01436-13. Epub 2013 Jul 3.

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