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Items: 6

1.

Calcium-dependent Nr4a1 expression in mouse Leydig cells requires distinct AP1/CRE and MEF2 elements.

Abdou HS, Robert NM, Tremblay JJ.

J Mol Endocrinol. 2016 Apr;56(3):151-61. doi: 10.1530/JME-15-0202. Epub 2015 Dec 8.

PMID:
26647388
2.

A cell-autonomous molecular cascade initiated by AMP-activated protein kinase represses steroidogenesis.

Abdou HS, Bergeron F, Tremblay JJ.

Mol Cell Biol. 2014 Dec 1;34(23):4257-71. doi: 10.1128/MCB.00734-14. Epub 2014 Sep 15.

3.

Inactivation of histone deacetylase 1 (HDAC1) but not HDAC2 is required for the glucocorticoid-dependent CCAAT/enhancer-binding protein α (C/EBPα) expression and preadipocyte differentiation.

Kuzmochka C, Abdou HS, Haché RJ, Atlas E.

Endocrinology. 2014 Dec;155(12):4762-73. doi: 10.1210/en.2014-1565. Epub 2014 Sep 9.

PMID:
25203139
5.

The calcium signaling pathway regulates leydig cell steroidogenesis through a transcriptional cascade involving the nuclear receptor NR4A1 and the steroidogenic acute regulatory protein.

Abdou HS, Villeneuve G, Tremblay JJ.

Endocrinology. 2013 Jan;154(1):511-20. doi: 10.1210/en.2012-1767. Epub 2012 Nov 26.

PMID:
23183170
6.

Liver-enriched inhibitory protein (LIP) actively inhibits preadipocyte differentiation through histone deacetylase 1 (HDAC1).

Abdou HS, Atlas E, Haché RJ.

J Biol Chem. 2011 Jun 17;286(24):21488-99. doi: 10.1074/jbc.M110.211540. Epub 2011 Apr 25.

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