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Items: 1 to 20 of 118

1.

Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

Ju ST, Panka DJ, Cui H, Ettinger R, el-Khatib M, Sherr DH, Stanger BZ, Marshak-Rothstein A.

Nature. 1995 Feb 2;373(6513):444-8.

PMID:
7530337
2.

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

Brunner T, Mogil RJ, LaFace D, Yoo NJ, Mahboubi A, Echeverri F, Martin SJ, Force WR, Lynch DH, Ware CF, et al.

Nature. 1995 Feb 2;373(6513):441-4.

PMID:
7530336
3.
4.

Regulation of T-cell death genes: selective inhibition of FasL- but not Fas-mediated function.

Cui H, Sherr DH, el-Khatib M, Matsui K, Panka DJ, Marshak-Rothstein A, Ju ST.

Cell Immunol. 1996 Feb 1;167(2):276-84.

PMID:
8603437
5.

Fas ligand-mediated cytotoxicity is directly responsible for apoptosis of normal CD4+ T cells responding to a bacterial superantigen.

Ettinger R, Panka DJ, Wang JK, Stanger BZ, Ju ST, Marshak-Rothstein A.

J Immunol. 1995 May 1;154(9):4302-8.

PMID:
7536768
6.
7.
8.

Cloning and expression of a short Fas ligand: A new alternatively spliced product of the mouse Fas ligand gene.

Ayroldi E, D'Adamio F, Zollo O, Agostini M, Moraca R, Cannarile L, Migliorati G, Delfino DV, Riccardi C.

Blood. 1999 Nov 15;94(10):3456-67.

9.

Fas ligand mediates activation-induced cell death in human T lymphocytes.

Alderson MR, Tough TW, Davis-Smith T, Braddy S, Falk B, Schooley KA, Goodwin RG, Smith CA, Ramsdell F, Lynch DH.

J Exp Med. 1995 Jan 1;181(1):71-7.

10.

Two adjacent trimeric Fas ligands are required for Fas signaling and formation of a death-inducing signaling complex.

Holler N, Tardivel A, Kovacsovics-Bankowski M, Hertig S, Gaide O, Martinon F, Tinel A, Deperthes D, Calderara S, Schulthess T, Engel J, Schneider P, Tschopp J.

Mol Cell Biol. 2003 Feb;23(4):1428-40.

11.

Fungal metabolite FR901228 inhibits c-Myc and Fas ligand expression.

Wang R, Brunner T, Zhang L, Shi Y.

Oncogene. 1998 Sep 24;17(12):1503-8.

12.

Negative regulation of CD95 ligand gene expression by vitamin D3 in T lymphocytes.

Cippitelli M, Fionda C, Di Bona D, Di Rosa F, Lupo A, Piccoli M, Frati L, Santoni A.

J Immunol. 2002 Feb 1;168(3):1154-66.

13.

The Fas death factor.

Nagata S, Golstein P.

Science. 1995 Mar 10;267(5203):1449-56. Review.

PMID:
7533326
15.

Cell death induced by the Fas/Fas ligand pathway and its role in pathology.

Waring P, Müllbacher A.

Immunol Cell Biol. 1999 Aug;77(4):312-7. Review.

PMID:
10457197
16.

Activation-induced apoptosis and cell surface expression of Fas (CD95) ligand are reciprocally regulated by retinoic acid receptor alpha and gamma and involve nur77 in T cells.

Tóth R, Szegezdi E, Reichert U, Bernardon JM, Michel S, Ancian P, Kis-Tóth K, Macsári Z, Fésüs L, Szondy Z.

Eur J Immunol. 2001 May;31(5):1382-91.

PMID:
11465095
17.

The molecular mechanism of FasL-mediated cytotoxicity by CD4+ Th1 clones.

el-Khatib M, Stanger BZ, Dogan H, Cui H, Ju ST.

Cell Immunol. 1995 Jul;163(2):237-44.

PMID:
7541726
19.

A novel gene product that couples TCR signaling to Fas(CD95) expression in activation-induced cell death.

Park CG, Lee SY, Kandala G, Lee SY, Choi Y.

Immunity. 1996 Jun;4(6):583-91.

20.

Characterization of lpr-derived T cell hybridomas: Fas-deficient hybridomas are deathless, growth-arrested, and cytotoxic upon activation.

Cui H, el-Khatib M, Sherr DH, Ettinger R, Sy MS, Marshak-Rothstein A, Ju ST.

Cell Immunol. 1996 Feb 1;167(2):302-12.

PMID:
8603440

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