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Items: 1 to 20 of 103

1.

Glia-specific autophagy dysfunction in ALS.

Strohm L, Behrends C.

Semin Cell Dev Biol. 2019 May 28. pii: S1084-9521(19)30020-5. doi: 10.1016/j.semcdb.2019.05.024. [Epub ahead of print] Review.

PMID:
31132469
2.

Circulating brain-enriched microRNAs as novel biomarkers for detection and differentiation of neurodegenerative diseases.

Sheinerman KS, Toledo JB, Tsivinsky VG, Irwin D, Grossman M, Weintraub D, Hurtig HI, Chen-Plotkin A, Wolk DA, McCluskey LF, Elman LB, Trojanowski JQ, Umansky SR.

Alzheimers Res Ther. 2017 Nov 9;9(1):89. doi: 10.1186/s13195-017-0316-0.

3.

Pathogenic mutation in the ALS/FTD gene, CCNF, causes elevated Lys48-linked ubiquitylation and defective autophagy.

Lee A, Rayner SL, Gwee SSL, De Luca A, Shahheydari H, Sundaramoorthy V, Ragagnin A, Morsch M, Radford R, Galper J, Freckleton S, Shi B, Walker AK, Don EK, Cole NJ, Yang S, Williams KL, Yerbury JJ, Blair IP, Atkin JD, Molloy MP, Chung RS.

Cell Mol Life Sci. 2018 Jan;75(2):335-354. doi: 10.1007/s00018-017-2632-8. Epub 2017 Aug 29.

PMID:
28852778
4.

Autophagy and apoptosis dysfunction in neurodegenerative disorders.

Ghavami S, Shojaei S, Yeganeh B, Ande SR, Jangamreddy JR, Mehrpour M, Christoffersson J, Chaabane W, Moghadam AR, Kashani HH, Hashemi M, Owji AA, Łos MJ.

Prog Neurobiol. 2014 Jan;112:24-49. doi: 10.1016/j.pneurobio.2013.10.004. Epub 2013 Nov 6. Review.

5.

Impaired protein degradation in FTLD and related disorders.

Götzl JK, Lang CM, Haass C, Capell A.

Ageing Res Rev. 2016 Dec;32:122-139. doi: 10.1016/j.arr.2016.04.008. Epub 2016 May 7. Review.

PMID:
27166223
6.

TDP-43-The key to understanding amyotrophic lateral sclerosis.

Xu Z, Yang C.

Rare Dis. 2014 Oct 30;2(1):e944443. doi: 10.4161/21675511.2014.944443. eCollection 2014.

7.

Differential roles of the ubiquitin proteasome system and autophagy in the clearance of soluble and aggregated TDP-43 species.

Scotter EL, Vance C, Nishimura AL, Lee YB, Chen HJ, Urwin H, Sardone V, Mitchell JC, Rogelj B, Rubinsztein DC, Shaw CE.

J Cell Sci. 2014 Mar 15;127(Pt 6):1263-78. doi: 10.1242/jcs.140087. Epub 2014 Jan 14.

8.

Pathomechanisms of TDP-43 in neurodegeneration.

Gao J, Wang L, Huntley ML, Perry G, Wang X.

J Neurochem. 2018 Feb 27. doi: 10.1111/jnc.14327. [Epub ahead of print] Review.

9.

Disturbance of proteasomal and autophagic protein degradation pathways by amyotrophic lateral sclerosis-linked mutations in ubiquilin 2.

Osaka M, Ito D, Suzuki N.

Biochem Biophys Res Commun. 2016 Apr 1;472(2):324-31. doi: 10.1016/j.bbrc.2016.02.107. Epub 2016 Mar 2.

PMID:
26944018
10.

Therapeutic potentials of plant iridoids in Alzheimer's and Parkinson's diseases: A review.

Dinda B, Dinda M, Kulsi G, Chakraborty A, Dinda S.

Eur J Med Chem. 2019 May 1;169:185-199. doi: 10.1016/j.ejmech.2019.03.009. Epub 2019 Mar 8. Review.

PMID:
30877973
11.

The established and emerging roles of astrocytes and microglia in amyotrophic lateral sclerosis and frontotemporal dementia.

Radford RA, Morsch M, Rayner SL, Cole NJ, Pountney DL, Chung RS.

Front Cell Neurosci. 2015 Oct 27;9:414. doi: 10.3389/fncel.2015.00414. eCollection 2015. Review.

12.

The role of heat shock proteins in Amyotrophic Lateral Sclerosis: The therapeutic potential of Arimoclomol.

Kalmar B, Lu CH, Greensmith L.

Pharmacol Ther. 2014 Jan;141(1):40-54. doi: 10.1016/j.pharmthera.2013.08.003. Epub 2013 Aug 23. Review.

PMID:
23978556
13.

The most prevalent genetic cause of ALS-FTD, C9orf72 synergizes the toxicity of ATXN2 intermediate polyglutamine repeats through the autophagy pathway.

Ciura S, Sellier C, Campanari ML, Charlet-Berguerand N, Kabashi E.

Autophagy. 2016 Aug 2;12(8):1406-8. doi: 10.1080/15548627.2016.1189070. Epub 2016 May 31.

14.

Degradation of misfolded proteins in neurodegenerative diseases: therapeutic targets and strategies.

Ciechanover A, Kwon YT.

Exp Mol Med. 2015 Mar 13;47:e147. doi: 10.1038/emm.2014.117. Review.

15.

Implications of Microglia in Amyotrophic Lateral Sclerosis and Frontotemporal Dementia.

Haukedal H, Freude K.

J Mol Biol. 2019 Apr 19;431(9):1818-1829. doi: 10.1016/j.jmb.2019.02.004. Epub 2019 Feb 11. Review.

PMID:
30763568
16.

Autophagy as a common pathway in amyotrophic lateral sclerosis.

Nguyen DKH, Thombre R, Wang J.

Neurosci Lett. 2019 Apr 1;697:34-48. doi: 10.1016/j.neulet.2018.04.006. Epub 2018 Apr 4. Review.

PMID:
29626651
17.

Interactions of pathological proteins in neurodegenerative diseases.

Spires-Jones TL, Attems J, Thal DR.

Acta Neuropathol. 2017 Aug;134(2):187-205. doi: 10.1007/s00401-017-1709-7. Epub 2017 Apr 11. Review.

18.

Impaired autophagy and APP processing in Alzheimer's disease: The potential role of Beclin 1 interactome.

Salminen A, Kaarniranta K, Kauppinen A, Ojala J, Haapasalo A, Soininen H, Hiltunen M.

Prog Neurobiol. 2013 Jul-Aug;106-107:33-54. doi: 10.1016/j.pneurobio.2013.06.002. Epub 2013 Jul 1. Review.

PMID:
23827971
19.

Possible concurrence of TDP-43, tau and other proteins in amyotrophic lateral sclerosis/frontotemporal lobar degeneration.

Takeda T.

Neuropathology. 2018 Feb;38(1):72-81. doi: 10.1111/neup.12428. Epub 2017 Sep 27. Review.

PMID:
28960544
20.

Is amyotrophic lateral sclerosis/frontotemporal dementia an autophagy disease?

Deng Z, Sheehan P, Chen S, Yue Z.

Mol Neurodegener. 2017 Dec 28;12(1):90. doi: 10.1186/s13024-017-0232-6. Review.

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