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Items: 4

1.

Pseudomonas aeruginosa Induced Airway Epithelial Injury Drives Fibroblast Activation: A Mechanism in Chronic Lung Allograft Dysfunction.

Borthwick LA, Suwara MI, Carnell SC, Green NJ, Mahida R, Dixon D, Gillespie CS, Cartwright TN, Horabin J, Walker A, Olin E, Rangar M, Gardner A, Mann J, Corris PA, Mann DA, Fisher AJ.

Am J Transplant. 2016 Jun;16(6):1751-65. doi: 10.1111/ajt.13690. Epub 2016 Feb 26.

2.

How reliable are sino-nasal cell lines for studying the pathophysiology of chronic rhinosinusitis?

Ball SL, Suwara MI, Borthwick LA, Wilson JA, Mann DA, Fisher AJ.

Ann Otol Rhinol Laryngol. 2015 Jun;124(6):437-42. doi: 10.1177/0003489414565003. Epub 2014 Dec 23.

3.

Mechanistic differences between phenotypes of chronic lung allograft dysfunction after lung transplantation.

Suwara MI, Vanaudenaerde BM, Verleden SE, Vos R, Green NJ, Ward C, Borthwick LA, Vandermeulen E, Lordan J, Van Raemdonck DE, Corris PA, Verleden GM, Fisher AJ.

Transpl Int. 2014 Aug;27(8):857-67. doi: 10.1111/tri.12341. Epub 2014 Jun 17.

4.

IL-1α released from damaged epithelial cells is sufficient and essential to trigger inflammatory responses in human lung fibroblasts.

Suwara MI, Green NJ, Borthwick LA, Mann J, Mayer-Barber KD, Barron L, Corris PA, Farrow SN, Wynn TA, Fisher AJ, Mann DA.

Mucosal Immunol. 2014 May;7(3):684-93. doi: 10.1038/mi.2013.87. Epub 2013 Oct 30.

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