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Items: 5


Inhibition of class I histone deacetylases abrogates TGFβ expression and development of fibrosis during chronic pancreatitis.

Bombardo M, Chen R, Malagola E, Saponara E, Hills AP, Graf R, Sonda S.

Mol Pharmacol. 2018 Jun 7. pii: mol.117.110924. doi: 10.1124/mol.117.110924. [Epub ahead of print]


Class I histone deacetylase inhibition improves pancreatitis outcome by limiting leukocyte recruitment and acinar-to-ductal metaplasia.

Bombardo M, Saponara E, Malagola E, Chen R, Seleznik GM, Haumaitre C, Quilichini E, Zabel A, Reding T, Graf R, Sonda S.

Br J Pharmacol. 2017 Nov;174(21):3865-3880. doi: 10.1111/bph.13984. Epub 2017 Sep 20.


Ibuprofen and diclofenac treatments reduce proliferation of pancreatic acinar cells upon inflammatory injury and mitogenic stimulation.

Bombardo M, Malagola E, Chen R, Rudnicka A, Graf R, Sonda S.

Br J Pharmacol. 2018 Jan;175(2):335-347. doi: 10.1111/bph.13867. Epub 2017 Jun 16.


Inactivation of TGFβ receptor II signalling in pancreatic epithelial cells promotes acinar cell proliferation, acinar-to-ductal metaplasia and fibrosis during pancreatitis.

Grabliauskaite K, Saponara E, Reding T, Bombardo M, Seleznik GM, Malagola E, Zabel A, Faso C, Sonda S, Graf R.

J Pathol. 2016 Feb;238(3):434-45. doi: 10.1002/path.4666. Epub 2015 Nov 28.


Serotonin promotes acinar dedifferentiation following pancreatitis-induced regeneration in the adult pancreas.

Saponara E, Grabliauskaite K, Bombardo M, Buzzi R, Silva AB, Malagola E, Tian Y, Hehl AB, Schraner EM, Seleznik GM, Zabel A, Reding T, Sonda S, Graf R.

J Pathol. 2015 Dec;237(4):495-507. doi: 10.1002/path.4595. Epub 2015 Sep 10.

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