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Items: 20

1.

The conduction pore of a cardiac potassium channel.

Tai KK, Goldstein SA.

Nature. 1998 Feb 5;391(6667):605-8.

PMID:
9468141
2.

Molecular mechanism and functional significance of the MinK control of the KvLQT1 channel activity.

Romey G, Attali B, Chouabe C, Abitbol I, Guillemare E, Barhanin J, Lazdunski M.

J Biol Chem. 1997 Jul 4;272(27):16713-6.

3.

Molecular basis of the long-QT syndrome associated with deafness.

Splawski I, Timothy KW, Vincent GM, Atkinson DL, Keating MT.

N Engl J Med. 1997 May 29;336(22):1562-7. No abstract available.

4.

KvLQT1, a voltage-gated potassium channel responsible for human cardiac arrhythmias.

Yang WP, Levesque PC, Little WA, Conder ML, Shalaby FY, Blanar MA.

Proc Natl Acad Sci U S A. 1997 Apr 15;94(8):4017-21.

5.

A novel mutation in the potassium channel gene KVLQT1 causes the Jervell and Lange-Nielsen cardioauditory syndrome.

Neyroud N, Tesson F, Denjoy I, Leibovici M, Donger C, Barhanin J, Fauré S, Gary F, Coumel P, Petit C, Schwartz K, Guicheney P.

Nat Genet. 1997 Feb;15(2):186-9.

PMID:
9020846
6.

Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel.

Sanguinetti MC, Curran ME, Zou A, Shen J, Spector PS, Atkinson DL, Keating MT.

Nature. 1996 Nov 7;384(6604):80-3.

PMID:
8900283
7.

K(V)LQT1 and lsK (minK) proteins associate to form the I(Ks) cardiac potassium current.

Barhanin J, Lesage F, Guillemare E, Fink M, Lazdunski M, Romey G.

Nature. 1996 Nov 7;384(6604):78-80.

PMID:
8900282
8.

Fast inactivation causes rectification of the IKr channel.

Spector PS, Curran ME, Zou A, Keating MT, Sanguinetti MC.

J Gen Physiol. 1996 May;107(5):611-9.

9.

Evidence for two components of delayed rectifier K+ current in human ventricular myocytes.

Li GR, Feng J, Yue L, Carrier M, Nattel S.

Circ Res. 1996 Apr;78(4):689-96.

10.

The inward rectification mechanism of the HERG cardiac potassium channel.

Smith PL, Baukrowitz T, Yellen G.

Nature. 1996 Feb 29;379(6568):833-6.

PMID:
8587608
11.

Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias.

Wang Q, Curran ME, Splawski I, Burn TC, Millholland JM, VanRaay TJ, Shen J, Timothy KW, Vincent GM, de Jager T, Schwartz PJ, Toubin JA, Moss AJ, Atkinson DL, Landes GM, Connors TD, Keating MT.

Nat Genet. 1996 Jan;12(1):17-23.

PMID:
8528244
13.

Effects of external cations and mutations in the pore region on C-type inactivation of Shaker potassium channels.

López-Barneo J, Hoshi T, Heinemann SH, Aldrich RW.

Receptors Channels. 1993;1(1):61-71.

PMID:
8081712
14.
15.

Oogenesis in Xenopus laevis (Daudin). I. Stages of oocyte development in laboratory maintained animals.

Dumont JN.

J Morphol. 1972 Feb;136(2):153-79. No abstract available.

PMID:
4109871
16.
18.

Biophysical and molecular mechanisms of Shaker potassium channel inactivation.

Hoshi T, Zagotta WN, Aldrich RW.

Science. 1990 Oct 26;250(4980):533-8.

PMID:
2122519
19.

Tetraethylammonium blockade distinguishes two inactivation mechanisms in voltage-activated K+ channels.

Choi KL, Aldrich RW, Yellen G.

Proc Natl Acad Sci U S A. 1991 Jun 15;88(12):5092-5.

20.

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