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Items: 1 to 20 of 43

1.

Endotoxin-induced uveitis is primarily dependent on radiation-resistant cells and on MyD88 but not TRIF.

Kezic J, Taylor S, Gupta S, Planck SR, Rosenzweig HL, Rosenbaum JT.

J Leukoc Biol. 2011 Aug;90(2):305-11. doi: 10.1189/jlb.0111036. Epub 2011 May 24.

2.

Dectin-1 and NOD2 mediate cathepsin activation in zymosan-induced arthritis in mice.

Rosenzweig HL, Clowers JS, Nunez G, Rosenbaum JT, Davey MP.

Inflamm Res. 2011 Jul;60(7):705-14. doi: 10.1007/s00011-011-0324-7. Epub 2011 Mar 20.

3.

Deal watch: XOMA and Servier to develop anti-IL-1β antibody for inflammatory diseases.

[No authors listed]

Nat Rev Drug Discov. 2011 Mar;10(3):166. doi: 10.1038/nrd3390. No abstract available.

PMID:
21358727
4.

Tissue-based class control: the other side of tolerance.

Matzinger P, Kamala T.

Nat Rev Immunol. 2011 Mar;11(3):221-30. doi: 10.1038/nri2940. Review.

PMID:
21350581
5.

The genetic basis of spondyloarthritis.

Reveille JD.

Ann Rheum Dis. 2011 Mar;70 Suppl 1:i44-50. doi: 10.1136/ard.2010.140574. Review.

PMID:
21339218
6.

Interleukin-1 in the pathogenesis and treatment of inflammatory diseases.

Dinarello CA.

Blood. 2011 Apr 7;117(14):3720-32. doi: 10.1182/blood-2010-07-273417. Epub 2011 Feb 8. Review.

7.

IL-37 is a fundamental inhibitor of innate immunity.

Nold MF, Nold-Petry CA, Zepp JA, Palmer BE, Bufler P, Dinarello CA.

Nat Immunol. 2010 Nov;11(11):1014-22. doi: 10.1038/ni.1944. Epub 2010 Oct 10.

8.

Interleukin-1β and interleukin-6 in arthritis animal models: roles in the early phase of transition from acute to chronic inflammation and relevance for human rheumatoid arthritis.

Ferraccioli G, Bracci-Laudiero L, Alivernini S, Gremese E, Tolusso B, De Benedetti F.

Mol Med. 2010 Nov-Dec;16(11-12):552-7. doi: 10.2119/molmed.2010.00067. Epub 2010 Aug 2. Review.

9.

Monogenic autoinflammatory diseases: new insights into clinical aspects and pathogenesis.

Henderson C, Goldbach-Mansky R.

Curr Opin Rheumatol. 2010 Sep;22(5):567-78. doi: 10.1097/BOR.0b013e32833ceff4. Review.

10.

TNF, but not IL-6 and IL-17, is crucial for the development of T cell-independent psoriasis-like dermatitis in Il1rn-/- mice.

Nakajima A, Matsuki T, Komine M, Asahina A, Horai R, Nakae S, Ishigame H, Kakuta S, Saijo S, Iwakura Y.

J Immunol. 2010 Aug 1;185(3):1887-93. doi: 10.4049/jimmunol.1001227. Epub 2010 Jul 7.

11.

IL-1 pathways in inflammation and human diseases.

Gabay C, Lamacchia C, Palmer G.

Nat Rev Rheumatol. 2010 Apr;6(4):232-41. doi: 10.1038/nrrheum.2010.4. Epub 2010 Feb 23. Review.

PMID:
20177398
12.

Nucleotide-binding oligomerization domain 2 and Toll-like receptor 2 function independently in a murine model of arthritis triggered by intraarticular peptidoglycan.

Rosenzweig HL, Jann MJ, Vance EE, Planck SR, Rosenbaum JT, Davey MP.

Arthritis Rheum. 2010 Apr;62(4):1051-9. doi: 10.1002/art.27335.

13.

Autoinflammation: the prominent role of IL-1 in monogenic autoinflammatory diseases and implications for common illnesses.

Goldbach-Mansky R, Kastner DL.

J Allergy Clin Immunol. 2009 Dec;124(6):1141-9; quiz 1150-1. doi: 10.1016/j.jaci.2009.11.016. Review.

14.

Local interleukin-1-driven joint pathology is dependent on toll-like receptor 4 activation.

Abdollahi-Roodsaz S, Joosten LA, Koenders MI, van den Brand BT, van de Loo FA, van den Berg WB.

Am J Pathol. 2009 Nov;175(5):2004-13. doi: 10.2353/ajpath.2009.090262. Epub 2009 Oct 15.

15.

An autoinflammatory disease due to homozygous deletion of the IL1RN locus.

Reddy S, Jia S, Geoffrey R, Lorier R, Suchi M, Broeckel U, Hessner MJ, Verbsky J.

N Engl J Med. 2009 Jun 4;360(23):2438-44. doi: 10.1056/NEJMoa0809568.

16.

An autoinflammatory disease with deficiency of the interleukin-1-receptor antagonist.

Aksentijevich I, Masters SL, Ferguson PJ, Dancey P, Frenkel J, van Royen-Kerkhoff A, Laxer R, Tedgård U, Cowen EW, Pham TH, Booty M, Estes JD, Sandler NG, Plass N, Stone DL, Turner ML, Hill S, Butman JA, Schneider R, Babyn P, El-Shanti HI, Pope E, Barron K, Bing X, Laurence A, Lee CC, Chapelle D, Clarke GI, Ohson K, Nicholson M, Gadina M, Yang B, Korman BD, Gregersen PK, van Hagen PM, Hak AE, Huizing M, Rahman P, Douek DC, Remmers EF, Kastner DL, Goldbach-Mansky R.

N Engl J Med. 2009 Jun 4;360(23):2426-37. doi: 10.1056/NEJMoa0807865.

17.

Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*).

Masters SL, Simon A, Aksentijevich I, Kastner DL.

Annu Rev Immunol. 2009;27:621-68. doi: 10.1146/annurev.immunol.25.022106.141627. Review.

18.

Lentiviral-vector-mediated expression of murine IL-1 receptor antagonist or IL-10 reduces the severity of endotoxin-induced uveitis.

Trittibach P, Barker SE, Broderick CA, Natkunarajah M, Duran Y, Robbie SJ, Bainbridge JW, Smith AJ, Sarra GM, Dick AD, Ali RR.

Gene Ther. 2008 Nov;15(22):1478-88. doi: 10.1038/gt.2008.109. Epub 2008 Jun 26.

19.

Activation of NOD2 in vivo induces IL-1beta production in the eye via caspase-1 but results in ocular inflammation independently of IL-1 signaling.

Rosenzweig HL, Martin TM, Planck SR, Galster K, Jann MM, Davey MP, Kobayashi K, Flavell RA, Rosenbaum JT.

J Leukoc Biol. 2008 Aug;84(2):529-36. doi: 10.1189/jlb.0108015. Epub 2008 May 21.

20.

NOD2, the gene responsible for familial granulomatous uveitis, in a mouse model of uveitis.

Rosenzweig HL, Martin TM, Jann MM, Planck SR, Davey MP, Kobayashi K, Flavell RA, Rosenbaum JT.

Invest Ophthalmol Vis Sci. 2008 Apr;49(4):1518-24. doi: 10.1167/iovs.07-1174.

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