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Items: 1 to 20 of 40

1.

Models of membrane-bound Alzheimer's Abeta peptide assemblies.

Shafrir Y, Durell S, Arispe N, Guy HR.

Proteins. 2010 Dec;78(16):3473-87. doi: 10.1002/prot.22853.

2.

Amyloid Beta annular protofibrils in cell processes and synapses accumulate with aging and Alzheimer-associated genetic modification.

Kokubo H, Kayed R, Glabe CG, Staufenbiel M, Saido TC, Iwata N, Yamaguchi H.

Int J Alzheimers Dis. 2009 Jul 14;2009. pii: 689285. doi: 10.4061/2009/689285.

3.

Truncated beta-amyloid peptide channels provide an alternative mechanism for Alzheimer's Disease and Down syndrome.

Jang H, Arce FT, Ramachandran S, Capone R, Azimova R, Kagan BL, Nussinov R, Lal R.

Proc Natl Acad Sci U S A. 2010 Apr 6;107(14):6538-43. doi: 10.1073/pnas.0914251107.

4.

Annular protofibrils are a structurally and functionally distinct type of amyloid oligomer.

Kayed R, Pensalfini A, Margol L, Sokolov Y, Sarsoza F, Head E, Hall J, Glabe C.

J Biol Chem. 2009 Feb 13;284(7):4230-7. doi: 10.1074/jbc.M808591200.

5.

Fibril specific, conformation dependent antibodies recognize a generic epitope common to amyloid fibrils and fibrillar oligomers that is absent in prefibrillar oligomers.

Kayed R, Head E, Sarsoza F, Saing T, Cotman CW, Necula M, Margol L, Wu J, Breydo L, Thompson JL, Rasool S, Gurlo T, Butler P, Glabe CG.

Mol Neurodegener. 2007 Sep 26;2:18.

6.

GM1-ganglioside-induced Abeta assembly on synaptic membranes of cultured neurons.

Yamamoto N, Fukata Y, Fukata M, Yanagisawa K.

Biochim Biophys Acta. 2007 May;1768(5):1128-37.

7.

Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid beta-peptide.

Haass C, Selkoe DJ.

Nat Rev Mol Cell Biol. 2007 Feb;8(2):101-12. Review.

PMID:
17245412
8.

Conformation-dependent anti-amyloid oligomer antibodies.

Kayed R, Glabe CG.

Methods Enzymol. 2006;413:326-44.

PMID:
17046404
9.

Are amyloid diseases caused by protein aggregates that mimic bacterial pore-forming toxins?

Lashuel HA, Lansbury PT Jr.

Q Rev Biophys. 2006 May;39(2):167-201. Review.

PMID:
16978447
10.

Prefibrillar amyloid aggregates could be generic toxins in higher organisms.

Baglioni S, Casamenti F, Bucciantini M, Luheshi LM, Taddei N, Chiti F, Dobson CM, Stefani M.

J Neurosci. 2006 Aug 2;26(31):8160-7.

11.

Protein aggregation and its consequences for human disease.

Dobson CM.

Protein Pept Lett. 2006;13(3):219-27. Review.

PMID:
16515449
12.

Common mechanisms of amyloid oligomer pathogenesis in degenerative disease.

Glabe CG.

Neurobiol Aging. 2006 Apr;27(4):570-5. Review.

PMID:
16481071
13.

Common structure and toxic function of amyloid oligomers implies a common mechanism of pathogenesis.

Glabe CG, Kayed R.

Neurology. 2006 Jan 24;66(2 Suppl 1):S74-8. Review.

PMID:
16432151
14.

Opinion: What is the role of protein aggregation in neurodegeneration?

Ross CA, Poirier MA.

Nat Rev Mol Cell Biol. 2005 Nov;6(11):891-8. Review.

PMID:
16167052
16.

Intraneuronal Abeta causes the onset of early Alzheimer's disease-related cognitive deficits in transgenic mice.

Billings LM, Oddo S, Green KN, McGaugh JL, LaFerla FM.

Neuron. 2005 Mar 3;45(5):675-88.

17.

Pore-forming protein toxins: from structure to function.

Parker MW, Feil SC.

Prog Biophys Mol Biol. 2005 May;88(1):91-142. Review.

PMID:
15561302
18.

Annular alpha-synuclein species from purified multiple system atrophy inclusions.

Pountney DL, Lowe R, Quilty M, Vickers JC, Voelcker NH, Gai WP.

J Neurochem. 2004 Jul;90(2):502-12.

19.
20.

The human islet amyloid polypeptide forms transient membrane-active prefibrillar assemblies.

Porat Y, Kolusheva S, Jelinek R, Gazit E.

Biochemistry. 2003 Sep 23;42(37):10971-7.

PMID:
12974632
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