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Items: 10

1.

Human pericyte-endothelial cell interactions in co-culture models mimicking the diabetic retinal microvascular environment.

Tarallo S, Beltramo E, Berrone E, Porta M.

Acta Diabetol. 2012 Dec;49 Suppl 1:S141-51. doi: 10.1007/s00592-012-0390-5. Epub 2012 Apr 19.

2.

Essential role of TGF-beta signaling in glucose-induced cell hypertrophy.

Wu L, Derynck R.

Dev Cell. 2009 Jul;17(1):35-48. doi: 10.1016/j.devcel.2009.05.010.

3.

TGF-beta signaling: a tale of two responses.

Rahimi RA, Leof EB.

J Cell Biochem. 2007 Oct 15;102(3):593-608. Review.

PMID:
17729308
4.
5.

TLR4 links innate immunity and fatty acid-induced insulin resistance.

Shi H, Kokoeva MV, Inouye K, Tzameli I, Yin H, Flier JS.

J Clin Invest. 2006 Nov;116(11):3015-25. Epub 2006 Oct 19.

6.

Akt-dependent transformation: there is more to growth than just surviving.

Plas DR, Thompson CB.

Oncogene. 2005 Nov 14;24(50):7435-42. Review.

PMID:
16288290
7.

Mediators of diabetic renal disease: the case for tgf-Beta as the major mediator.

Ziyadeh FN.

J Am Soc Nephrol. 2004 Jan;15 Suppl 1:S55-7. Review.

8.

In the absence of extrinsic signals, nutrient utilization by lymphocytes is insufficient to maintain either cell size or viability.

Rathmell JC, Vander Heiden MG, Harris MH, Frauwirth KA, Thompson CB.

Mol Cell. 2000 Sep;6(3):683-92.

9.

Molecular mechanisms of diabetic renal hypertrophy.

Wolf G, Ziyadeh FN.

Kidney Int. 1999 Aug;56(2):393-405. Review.

10.

Size control in animal development.

Conlon I, Raff M.

Cell. 1999 Jan 22;96(2):235-44. Review. No abstract available.

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