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Items: 1 to 20 of 29

1.

Real-time fluorescence detection of ERAD substrate retrotranslocation in a mammalian in vitro system.

Wahlman J, DeMartino GN, Skach WR, Bulleid NJ, Brodsky JL, Johnson AE.

Cell. 2007 Jun 1;129(5):943-55.

2.

Derlin-1 promotes the efficient degradation of the cystic fibrosis transmembrane conductance regulator (CFTR) and CFTR folding mutants.

Sun F, Zhang R, Gong X, Geng X, Drain PF, Frizzell RA.

J Biol Chem. 2006 Dec 1;281(48):36856-63. Epub 2006 Sep 5.

3.

Downregulation of protein disulfide isomerase inhibits infection by the mouse polyomavirus.

Gilbert J, Ou W, Silver J, Benjamin T.

J Virol. 2006 Nov;80(21):10868-70. Epub 2006 Aug 23.

4.

Murine polyomavirus requires the endoplasmic reticulum protein Derlin-2 to initiate infection.

Lilley BN, Gilbert JM, Ploegh HL, Benjamin TL.

J Virol. 2006 Sep;80(17):8739-44.

5.

Sequential quality-control checkpoints triage misfolded cystic fibrosis transmembrane conductance regulator.

Younger JM, Chen L, Ren HY, Rosser MF, Turnbull EL, Fan CY, Patterson C, Cyr DM.

Cell. 2006 Aug 11;126(3):571-82.

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A complex of Yos9p and the HRD ligase integrates endoplasmic reticulum quality control into the degradation machinery.

Gauss R, Jarosch E, Sommer T, Hirsch C.

Nat Cell Biol. 2006 Aug;8(8):849-54. Epub 2006 Jul 16.

PMID:
16845381
9.

Protein disulfide isomerase-like proteins play opposing roles during retrotranslocation.

Forster ML, Sivick K, Park YN, Arvan P, Lencer WI, Tsai B.

J Cell Biol. 2006 Jun 19;173(6):853-9.

10.

EDEM is involved in retrotranslocation of ricin from the endoplasmic reticulum to the cytosol.

Slominska-Wojewodzka M, Gregers TF, Wälchli S, Sandvig K.

Mol Biol Cell. 2006 Apr;17(4):1664-75. Epub 2006 Feb 1.

11.

Derlin-2 and Derlin-3 are regulated by the mammalian unfolded protein response and are required for ER-associated degradation.

Oda Y, Okada T, Yoshida H, Kaufman RJ, Nagata K, Mori K.

J Cell Biol. 2006 Jan 30;172(3):383-93.

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Protein disulphide-isomerase reduces ricin to its A and B chains in the endoplasmic reticulum.

Spooner RA, Watson PD, Marsden CJ, Smith DC, Moore KA, Cook JP, Lord JM, Roberts LM.

Biochem J. 2004 Oct 15;383(Pt 2):285-93.

14.

A membrane protein complex mediates retro-translocation from the ER lumen into the cytosol.

Ye Y, Shibata Y, Yun C, Ron D, Rapoport TA.

Nature. 2004 Jun 24;429(6994):841-7.

15.

A membrane protein required for dislocation of misfolded proteins from the ER.

Lilley BN, Ploegh HL.

Nature. 2004 Jun 24;429(6994):834-40.

16.

Molecular characterization of the principal substrate binding site of the ubiquitous folding catalyst protein disulfide isomerase.

Pirneskoski A, Klappa P, Lobell M, Williamson RA, Byrne L, Alanen HI, Salo KE, Kivirikko KI, Freedman RB, Ruddock LW.

J Biol Chem. 2004 Mar 12;279(11):10374-81. Epub 2003 Dec 18.

17.

The intracellular voyage of cholera toxin: going retro.

Lencer WI, Tsai B.

Trends Biochem Sci. 2003 Dec;28(12):639-45. Review.

PMID:
14659695
18.

Gangliosides that associate with lipid rafts mediate transport of cholera and related toxins from the plasma membrane to endoplasmic reticulm.

Fujinaga Y, Wolf AA, Rodighiero C, Wheeler H, Tsai B, Allen L, Jobling MG, Rapoport T, Holmes RK, Lencer WI.

Mol Biol Cell. 2003 Dec;14(12):4783-93. Epub 2003 Sep 17.

19.

Role of ubiquitination in retro-translocation of cholera toxin and escape of cytosolic degradation.

Rodighiero C, Tsai B, Rapoport TA, Lencer WI.

EMBO Rep. 2002 Dec;3(12):1222-7. Epub 2002 Nov 21.

20.

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