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Items: 1 to 20 of 31

1.

H2AX phosphorylation within the G1 phase after UV irradiation depends on nucleotide excision repair and not DNA double-strand breaks.

Marti TM, Hefner E, Feeney L, Natale V, Cleaver JE.

Proc Natl Acad Sci U S A. 2006 Jun 27;103(26):9891-6. Epub 2006 Jun 20.

2.

Opposing effects of the UV lesion repair protein XPA and UV bypass polymerase eta on ATR checkpoint signaling.

Bomgarden RD, Lupardus PJ, Soni DV, Yee MC, Ford JM, Cimprich KA.

EMBO J. 2006 Jun 7;25(11):2605-14. Epub 2006 May 4. Erratum in: EMBO J. 2006 Oct 18;25(20):5036.

3.

p8/TTD-A as a repair-specific TFIIH subunit.

Coin F, Proietti De Santis L, Nardo T, Zlobinskaya O, Stefanini M, Egly JM.

Mol Cell. 2006 Jan 20;21(2):215-26.

4.

XPA protein as a limiting factor for nucleotide excision repair and UV sensitivity in human cells.

Köberle B, Roginskaya V, Wood RD.

DNA Repair (Amst). 2006 May 10;5(5):641-8. Epub 2006 Jan 18.

PMID:
16413230
5.
6.

Transcriptome analysis reveals cyclobutane pyrimidine dimers as a major source of UV-induced DNA breaks.

Garinis GA, Mitchell JR, Moorhouse MJ, Hanada K, de Waard H, Vandeputte D, Jans J, Brand K, Smid M, van der Spek PJ, Hoeijmakers JH, Kanaar R, van der Horst GT.

EMBO J. 2005 Nov 16;24(22):3952-62. Epub 2005 Oct 27.

7.

Transcription-associated breaks in xeroderma pigmentosum group D cells from patients with combined features of xeroderma pigmentosum and Cockayne syndrome.

Theron T, Fousteri MI, Volker M, Harries LW, Botta E, Stefanini M, Fujimoto M, Andressoo JO, Mitchell J, Jaspers NG, McDaniel LD, Mullenders LH, Lehmann AR.

Mol Cell Biol. 2005 Sep;25(18):8368-78.

8.

UV-sensitive syndrome.

Spivak G.

Mutat Res. 2005 Sep 4;577(1-2):162-9.

PMID:
15916784
9.

Unwind and slow down: checkpoint activation by helicase and polymerase uncoupling.

Cortez D.

Genes Dev. 2005 May 1;19(9):1007-12. No abstract available.

10.

A new, tenth subunit of TFIIH is responsible for the DNA repair syndrome trichothiodystrophy group A.

Giglia-Mari G, Coin F, Ranish JA, Hoogstraten D, Theil A, Wijgers N, Jaspers NG, Raams A, Argentini M, van der Spek PJ, Botta E, Stefanini M, Egly JM, Aebersold R, Hoeijmakers JH, Vermeulen W.

Nat Genet. 2004 Jul;36(7):714-9. Epub 2004 Jun 27.

PMID:
15220921
11.

UV-induced ataxia-telangiectasia-mutated and Rad3-related (ATR) activation requires replication stress.

Ward IM, Minn K, Chen J.

J Biol Chem. 2004 Mar 12;279(11):9677-80. Epub 2004 Jan 23.

12.

Physical and functional interactions between nucleotide excision repair and DNA damage checkpoint.

Giannattasio M, Lazzaro F, Longhese MP, Plevani P, Muzi-Falconi M.

EMBO J. 2004 Jan 28;23(2):429-38. Epub 2004 Jan 15.

13.

True XP group E patients have a defective UV-damaged DNA binding protein complex and mutations in DDB2 which reveal the functional domains of its p48 product.

Rapić-Otrin V, Navazza V, Nardo T, Botta E, McLenigan M, Bisi DC, Levine AS, Stefanini M.

Hum Mol Genet. 2003 Jul 1;12(13):1507-22.

PMID:
12812979
14.

Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes.

Zou L, Elledge SJ.

Science. 2003 Jun 6;300(5625):1542-8.

15.

A splicing mutation affecting expression of ataxia-telangiectasia and Rad3-related protein (ATR) results in Seckel syndrome.

O'Driscoll M, Ruiz-Perez VL, Woods CG, Jeggo PA, Goodship JA.

Nat Genet. 2003 Apr;33(4):497-501. Epub 2003 Mar 17.

PMID:
12640452
16.

The founding members of xeroderma pigmentosum group G produce XPG protein with severely impaired endonuclease activity.

Lalle P, Nouspikel T, Constantinou A, Thorel F, Clarkson SG.

J Invest Dermatol. 2002 Feb;118(2):344-51.

17.

The xeroderma pigmentosum group D (XPD) gene: one gene, two functions, three diseases.

Lehmann AR.

Genes Dev. 2001 Jan 1;15(1):15-23. Review. No abstract available.

18.

The DNA damage response: putting checkpoints in perspective.

Zhou BB, Elledge SJ.

Nature. 2000 Nov 23;408(6811):433-9. Review.

PMID:
11100718
19.
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