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Items: 1 to 20 of 115

1.

X-linked IAP is a direct inhibitor of cell-death proteases.

Deveraux QL, Takahashi R, Salvesen GS, Reed JC.

Nature. 1997 Jul 17;388(6639):300-4.

PMID:
9230442
2.

IAP-family protein survivin inhibits caspase activity and apoptosis induced by Fas (CD95), Bax, caspases, and anticancer drugs.

Tamm I, Wang Y, Sausville E, Scudiero DA, Vigna N, Oltersdorf T, Reed JC.

Cancer Res. 1998 Dec 1;58(23):5315-20.

3.

Smac induces cytochrome c release and apoptosis independently from Bax/Bcl-x(L) in a strictly caspase-3-dependent manner in human carcinoma cells.

Hasenjäger A, Gillissen B, Müller A, Normand G, Hemmati PG, Schuler M, Dörken B, Daniel PT.

Oncogene. 2004 Jun 3;23(26):4523-35.

PMID:
15064710
4.

The Drosophila inhibitor of apoptosis D-IAP1 suppresses cell death induced by the caspase drICE.

Kaiser WJ, Vucic D, Miller LK.

FEBS Lett. 1998 Nov 27;440(1-2):243-8.

5.

Conversion of Bcl-2 to a Bax-like death effector by caspases.

Cheng EH, Kirsch DG, Clem RJ, Ravi R, Kastan MB, Bedi A, Ueno K, Hardwick JM.

Science. 1997 Dec 12;278(5345):1966-8.

6.

A single BIR domain of XIAP sufficient for inhibiting caspases.

Takahashi R, Deveraux Q, Tamm I, Welsh K, Assa-Munt N, Salvesen GS, Reed JC.

J Biol Chem. 1998 Apr 3;273(14):7787-90.

7.

Fas-induced activation of the cell death-related protease CPP32 Is inhibited by Bcl-2 and by ICE family protease inhibitors.

Armstrong RC, Aja T, Xiang J, Gaur S, Krebs JF, Hoang K, Bai X, Korsmeyer SJ, Karanewsky DS, Fritz LC, Tomaselli KJ.

J Biol Chem. 1996 Jul 12;271(28):16850-5.

8.

Cellular, biochemical, and genetic analysis of mechanism of small molecule IAP inhibitors.

Wang Z, Cuddy M, Samuel T, Welsh K, Schimmer A, Hanaii F, Houghten R, Pinilla C, Reed JC.

J Biol Chem. 2004 Nov 12;279(46):48168-76. Epub 2004 Aug 27.

9.

Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c.

Rossé T, Olivier R, Monney L, Rager M, Conus S, Fellay I, Jansen B, Borner C.

Nature. 1998 Jan 29;391(6666):496-9.

PMID:
9461218
10.

Transforming growth factor-beta-mediated apoptosis in the Ramos B-lymphoma cell line is accompanied by caspase activation and Bcl-XL downregulation.

Saltzman A, Munro R, Searfoss G, Franks C, Jaye M, Ivashchenko Y.

Exp Cell Res. 1998 Jul 10;242(1):244-54.

PMID:
9665822
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17.

XIAP inhibition of caspase-3 preserves its association with the Apaf-1 apoptosome and prevents CD95- and Bax-induced apoptosis.

Bratton SB, Lewis J, Butterworth M, Duckett CS, Cohen GM.

Cell Death Differ. 2002 Sep;9(9):881-92.

18.

IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases.

Deveraux QL, Roy N, Stennicke HR, Van Arsdale T, Zhou Q, Srinivasula SM, Alnemri ES, Salvesen GS, Reed JC.

EMBO J. 1998 Apr 15;17(8):2215-23.

19.

Neutralization of Smac/Diablo by inhibitors of apoptosis (IAPs). A caspase-independent mechanism for apoptotic inhibition.

Wilkinson JC, Wilkinson AS, Scott FL, Csomos RA, Salvesen GS, Duckett CS.

J Biol Chem. 2004 Dec 3;279(49):51082-90. Epub 2004 Sep 15.

20.

Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

Finucane DM, Bossy-Wetzel E, Waterhouse NJ, Cotter TG, Green DR.

J Biol Chem. 1999 Jan 22;274(4):2225-33.

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