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Items: 1 to 20 of 125

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PHAS proteins as mediators of the actions of insulin, growth factors and cAMP on protein synthesis and cell proliferation.

Lawrence JC Jr, Fadden P, Haystead TA, Lin TA.

Adv Enzyme Regul. 1997;37:239-67. Review.

PMID:
9381973
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cAMP- and rapamycin-sensitive regulation of the association of eukaryotic initiation factor 4E and the translational regulator PHAS-I in aortic smooth muscle cells.

Graves LM, Bornfeldt KE, Argast GM, Krebs EG, Kong X, Lin TA, Lawrence JC Jr.

Proc Natl Acad Sci U S A. 1995 Aug 1;92(16):7222-6.

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Multiple mechanisms control phosphorylation of PHAS-I in five (S/T)P sites that govern translational repression.

Mothe-Satney I, Yang D, Fadden P, Haystead TA, Lawrence JC Jr.

Mol Cell Biol. 2000 May;20(10):3558-67.

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Phosphorylation of the translational regulator, PHAS-I, by protein kinase CK2.

Fadden P, Haystead TA, Lawrence JC Jr.

FEBS Lett. 1998 Sep 11;435(1):105-9.

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Phosphorylation of the translational repressor PHAS-I by the mammalian target of rapamycin.

Brunn GJ, Hudson CC, Sekulić A, Williams JM, Hosoi H, Houghton PJ, Lawrence JC Jr, Abraham RT.

Science. 1997 Jul 4;277(5322):99-101.

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PHAS-I as a link between mitogen-activated protein kinase and translation initiation.

Lin TA, Kong X, Haystead TA, Pause A, Belsham G, Sonenberg N, Lawrence JC Jr.

Science. 1994 Oct 28;266(5185):653-6.

PMID:
7939721
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Insulin and diabetes cause reciprocal changes in the association of eIF-4E and PHAS-I in rat skeletal muscle.

Kimball SR, Jefferson LS, Fadden P, Haystead TA, Lawrence JC Jr.

Am J Physiol. 1996 Feb;270(2 Pt 1):C705-9.

PMID:
8779938
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