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Items: 1 to 20 of 156

1.

MEKK3 and TAK1 synergize to activate IKK complex in Helicobacter pylori infection.

Sokolova O, Maubach G, Naumann M.

Biochim Biophys Acta. 2014 Apr;1843(4):715-24. doi: 10.1016/j.bbamcr.2014.01.006. Epub 2014 Jan 11.

2.

Helicobacter pylori CagA activates NF-kappaB by targeting TAK1 for TRAF6-mediated Lys 63 ubiquitination.

Lamb A, Yang XD, Tsang YH, Li JD, Higashi H, Hatakeyama M, Peek RM, Blanke SR, Chen LF.

EMBO Rep. 2009 Nov;10(11):1242-9. doi: 10.1038/embor.2009.210. Epub 2009 Oct 9.

3.
4.

MyD88 and TNF receptor-associated factor 6 are critical signal transducers in Helicobacter pylori-infected human epithelial cells.

Hirata Y, Ohmae T, Shibata W, Maeda S, Ogura K, Yoshida H, Kawabe T, Omata M.

J Immunol. 2006 Mar 15;176(6):3796-803.

5.

Lysine 63-linked polyubiquitination of TAK1 at lysine 158 is required for tumor necrosis factor alpha- and interleukin-1beta-induced IKK/NF-kappaB and JNK/AP-1 activation.

Fan Y, Yu Y, Shi Y, Sun W, Xie M, Ge N, Mao R, Chang A, Xu G, Schneider MD, Zhang H, Fu S, Qin J, Yang J.

J Biol Chem. 2010 Feb 19;285(8):5347-60. doi: 10.1074/jbc.M109.076976. Epub 2009 Dec 28.

6.

Two mechanistically and temporally distinct NF-kappaB activation pathways in IL-1 signaling.

Yamazaki K, Gohda J, Kanayama A, Miyamoto Y, Sakurai H, Yamamoto M, Akira S, Hayashi H, Su B, Inoue J.

Sci Signal. 2009 Oct 20;2(93):ra66. doi: 10.1126/scisignal.2000387.

7.

TLR8-mediated NF-kappaB and JNK activation are TAK1-independent and MEKK3-dependent.

Qin J, Yao J, Cui G, Xiao H, Kim TW, Fraczek J, Wightman P, Sato S, Akira S, Puel A, Casanova JL, Su B, Li X.

J Biol Chem. 2006 Jul 28;281(30):21013-21. Epub 2006 May 31.

8.

Helicobacter pylori activates NF-κB by inducing Ubc13-mediated ubiquitination of lysine 158 of TAK1.

Lamb A, Chen J, Blanke SR, Chen LF.

J Cell Biochem. 2013 Oct;114(10):2284-92. doi: 10.1002/jcb.24573.

9.

H. pylori activates NF-kappaB through a signaling pathway involving IkappaB kinases, NF-kappaB-inducing kinase, TRAF2, and TRAF6 in gastric cancer cells.

Maeda S, Yoshida H, Ogura K, Mitsuno Y, Hirata Y, Yamaji Y, Akanuma M, Shiratori Y, Omata M.

Gastroenterology. 2000 Jul;119(1):97-108.

PMID:
10889159
10.

Helicobacter pylori induces IkappaB kinase alpha nuclear translocation and chemokine production in gastric epithelial cells.

Hirata Y, Maeda S, Ohmae T, Shibata W, Yanai A, Ogura K, Yoshida H, Kawabe T, Omata M.

Infect Immun. 2006 Mar;74(3):1452-61.

11.

TAB2, TRAF6 and TAK1 are involved in NF-kappaB activation induced by the TNF-receptor, Edar and its adaptator Edaradd.

Morlon A, Munnich A, Smahi A.

Hum Mol Genet. 2005 Dec 1;14(23):3751-7. Epub 2005 Oct 26.

PMID:
16251197
12.

cIAP1, cIAP2, and XIAP act cooperatively via nonredundant pathways to regulate genotoxic stress-induced nuclear factor-kappaB activation.

Jin HS, Lee DH, Kim DH, Chung JH, Lee SJ, Lee TH.

Cancer Res. 2009 Mar 1;69(5):1782-91. doi: 10.1158/0008-5472.CAN-08-2256. Epub 2009 Feb 17.

13.

TAK1 lysine 158 is required for TGF-β-induced TRAF6-mediated Smad-independent IKK/NF-κB and JNK/AP-1 activation.

Mao R, Fan Y, Mou Y, Zhang H, Fu S, Yang J.

Cell Signal. 2011 Jan;23(1):222-7. doi: 10.1016/j.cellsig.2010.09.006. Epub 2010 Sep 16.

14.

Homeostatic interactions between MEKK3 and TAK1 involved in NF-kappaB signaling.

Di Y, Li S, Wang L, Zhang Y, Dorf ME.

Cell Signal. 2008 Apr;20(4):705-13. doi: 10.1016/j.cellsig.2007.12.007. Epub 2008 Jan 18.

15.

Kaposi's sarcoma associated herpesvirus encoded viral FLICE inhibitory protein K13 activates NF-κB pathway independent of TRAF6, TAK1 and LUBAC.

Matta H, Gopalakrishnan R, Graham C, Tolani B, Khanna A, Yi H, Suo Y, Chaudhary PM.

PLoS One. 2012;7(5):e36601. doi: 10.1371/journal.pone.0036601. Epub 2012 May 8.

16.

Helicobacter pylori induces type 4 secretion system-dependent, but CagA-independent activation of IκBs and NF-κB/RelA at early time points.

Sokolova O, Borgmann M, Rieke C, Schweitzer K, Rothkötter HJ, Naumann M.

Int J Med Microbiol. 2013 Dec;303(8):548-52. doi: 10.1016/j.ijmm.2013.07.008. Epub 2013 Jul 26.

PMID:
23972614
17.

Differential roles of ASK1 and TAK1 in Helicobacter pylori-induced cellular responses.

Hayakawa Y, Hirata Y, Kinoshita H, Sakitani K, Nakagawa H, Nakata W, Takahashi R, Sakamoto K, Maeda S, Koike K.

Infect Immun. 2013 Dec;81(12):4551-60. doi: 10.1128/IAI.00914-13. Epub 2013 Sep 30.

18.

HopQ impacts the integrin α5β1-independent NF-κB activation by Helicobacter pylori in CEACAM expressing cells.

Feige MH, Sokolova O, Pickenhahn A, Maubach G, Naumann M.

Int J Med Microbiol. 2018 Jul;308(5):527-533. doi: 10.1016/j.ijmm.2018.05.003. Epub 2018 May 14.

PMID:
29779861
19.

Site-specific Lys-63-linked tumor necrosis factor receptor-associated factor 6 auto-ubiquitination is a critical determinant of I kappa B kinase activation.

Lamothe B, Besse A, Campos AD, Webster WK, Wu H, Darnay BG.

J Biol Chem. 2007 Feb 9;282(6):4102-12. Epub 2006 Nov 29.

20.

Phosphoinositide-dependent kinase-1 inhibits TRAF6 ubiquitination by interrupting the formation of TAK1-TAB2 complex in TLR4 signaling.

Moon G, Kim J, Min Y, Wi SM, Shim JH, Chun E, Lee KY.

Cell Signal. 2015 Dec;27(12):2524-33. doi: 10.1016/j.cellsig.2015.09.018. Epub 2015 Sep 30.

PMID:
26432169

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