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Items: 1 to 20 of 90

1.
2.

Senescence delay of human diploid fibroblast induced by anti-sense p16INK4a expression.

Duan J, Zhang Z, Tong T.

J Biol Chem. 2001 Dec 21;276(51):48325-31. Epub 2001 Oct 17.

3.
4.

Evidence that high telomerase activity may induce a senescent-like growth arrest in human fibroblasts.

Gorbunova V, Seluanov A, Pereira-Smith OM.

J Biol Chem. 2003 Feb 28;278(9):7692-8. Epub 2002 Dec 19.

5.

Senescence occurs with hTERT repression and limited telomere shortening in human oral keratinocytes cultured with feeder cells.

Kang MK, Kameta A, Shin KH, Baluda MA, Park NH.

J Cell Physiol. 2004 Jun;199(3):364-70.

PMID:
15095283
6.
7.

Increased stability of the p16 mRNA with replicative senescence.

Wang W, Martindale JL, Yang X, Chrest FJ, Gorospe M.

EMBO Rep. 2005 Feb;6(2):158-64.

8.

Reversal of human cellular senescence: roles of the p53 and p16 pathways.

Beauséjour CM, Krtolica A, Galimi F, Narita M, Lowe SW, Yaswen P, Campisi J.

EMBO J. 2003 Aug 15;22(16):4212-22.

9.

p16/cyclin-dependent kinase inhibitor 2A deficiency in human melanocyte senescence, apoptosis, and immortalization: possible implications for melanoma progression.

Sviderskaya EV, Gray-Schopfer VC, Hill SP, Smit NP, Evans-Whipp TJ, Bond J, Hill L, Bataille V, Peters G, Kipling D, Wynford-Thomas D, Bennett DC.

J Natl Cancer Inst. 2003 May 21;95(10):723-32.

PMID:
12759390
10.
11.

Features of replicative senescence induced by direct addition of antennapedia-p16INK4A fusion protein to human diploid fibroblasts.

Kato D, Miyazawa K, Ruas M, Starborg M, Wada I, Oka T, Sakai T, Peters G, Hara E.

FEBS Lett. 1998 May 8;427(2):203-8.

12.
13.

Induction of extracellular matrix-remodeling genes by the senescence-associated protein APA-1.

Benanti JA, Williams DK, Robinson KL, Ozer HL, Galloway DA.

Mol Cell Biol. 2002 Nov;22(21):7385-97.

14.

Telomerase induces immortalization of human esophageal keratinocytes without p16INK4a inactivation.

Harada H, Nakagawa H, Oyama K, Takaoka M, Andl CD, Jacobmeier B, von Werder A, Enders GH, Opitz OG, Rustgi AK.

Mol Cancer Res. 2003 Aug;1(10):729-38.

15.

Telomere-based proliferative lifespan barriers in Werner-syndrome fibroblasts involve both p53-dependent and p53-independent mechanisms.

Davis T, Singhrao SK, Wyllie FS, Haughton MF, Smith PJ, Wiltshire M, Wynford-Thomas D, Jones CJ, Faragher RG, Kipling D.

J Cell Sci. 2003 Apr 1;116(Pt 7):1349-57.

16.

Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence.

Ohtani N, Zebedee Z, Huot TJ, Stinson JA, Sugimoto M, Ohashi Y, Sharrocks AD, Peters G, Hara E.

Nature. 2001 Feb 22;409(6823):1067-70.

PMID:
11234019
17.

Putative telomere-independent mechanisms of replicative aging reflect inadequate growth conditions.

Ramirez RD, Morales CP, Herbert BS, Rohde JM, Passons C, Shay JW, Wright WE.

Genes Dev. 2001 Feb 15;15(4):398-403.

18.

A genomics approach identifies senescence-specific gene expression regulation.

Lackner DH, Hayashi MT, Cesare AJ, Karlseder J.

Aging Cell. 2014 Oct;13(5):946-50. doi: 10.1111/acel.12234. Epub 2014 May 23.

19.
20.

Evidence for a CDK4-dependent checkpoint in a conditional model of cellular senescence.

Brookes S, Gagrica S, Sanij E, Rowe J, Gregory FJ, Hara E, Peters G.

Cell Cycle. 2015;14(8):1164-73. doi: 10.1080/15384101.2015.1010866.

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