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Items: 1 to 20 of 95

1.

CD36 protein influences myocardial Ca2+ homeostasis and phospholipid metabolism: conduction anomalies in CD36-deficient mice during fasting.

Pietka TA, Sulkin MS, Kuda O, Wang W, Zhou D, Yamada KA, Yang K, Su X, Gross RW, Nerbonne JM, Efimov IR, Abumrad NA.

J Biol Chem. 2012 Nov 9;287(46):38901-12. doi: 10.1074/jbc.M112.413609. Epub 2012 Sep 27.

2.

Hearts lacking caveolin-1 develop hypertrophy with normal cardiac substrate metabolism.

Augustus AS, Buchanan J, Gutman E, Rengo G, Pestell RG, Fortina P, Koch WJ, Bensadoun A, Abel ED, Lisanti MP.

Cell Cycle. 2008 Aug 15;7(16):2509-18. Epub 2008 Aug 26.

PMID:
18719368
3.

Expression of SERCA isoform with faster Ca2+ transport properties improves postischemic cardiac function and Ca2+ handling and decreases myocardial infarction.

Talukder MA, Kalyanasundaram A, Zhao X, Zuo L, Bhupathy P, Babu GJ, Cardounel AJ, Periasamy M, Zweier JL.

Am J Physiol Heart Circ Physiol. 2007 Oct;293(4):H2418-28. Epub 2007 Jul 13.

4.

Compensatory mechanisms associated with the hyperdynamic function of phospholamban-deficient mouse hearts.

Chu G, Luo W, Slack JP, Tilgmann C, Sweet WE, Spindler M, Saupe KW, Boivin GP, Moravec CS, Matlib MA, Grupp IL, Ingwall JS, Kranias EG.

Circ Res. 1996 Dec;79(6):1064-76.

5.

Phosphodiesterase type 3A regulates basal myocardial contractility through interacting with sarcoplasmic reticulum calcium ATPase type 2a signaling complexes in mouse heart.

Beca S, Ahmad F, Shen W, Liu J, Makary S, Polidovitch N, Sun J, Hockman S, Chung YW, Movsesian M, Murphy E, Manganiello V, Backx PH.

Circ Res. 2013 Jan 18;112(2):289-97. doi: 10.1161/CIRCRESAHA.111.300003. Epub 2012 Nov 19.

6.

Role of protein phosphorylation in excitation-contraction coupling in taurine deficient hearts.

Ramila KC, Jong CJ, Pastukh V, Ito T, Azuma J, Schaffer SW.

Am J Physiol Heart Circ Physiol. 2015 Feb 1;308(3):H232-9. doi: 10.1152/ajpheart.00497.2014. Epub 2014 Nov 26.

7.

CD36 actions in the heart: Lipids, calcium, inflammation, repair and more?

Abumrad NA, Goldberg IJ.

Biochim Biophys Acta. 2016 Oct;1861(10):1442-9. doi: 10.1016/j.bbalip.2016.03.015. Epub 2016 Mar 21. Review.

8.

CD36 is indispensable for thermogenesis under conditions of fasting and cold stress.

Putri M, Syamsunarno MR, Iso T, Yamaguchi A, Hanaoka H, Sunaga H, Koitabashi N, Matsui H, Yamazaki C, Kameo S, Tsushima Y, Yokoyama T, Koyama H, Abumrad NA, Kurabayashi M.

Biochem Biophys Res Commun. 2015 Feb 20;457(4):520-5. doi: 10.1016/j.bbrc.2014.12.124. Epub 2015 Jan 13.

9.

Phospholamban ablation in hearts expressing the high affinity SERCA2b isoform normalizes global Ca²⁺ homeostasis but not Ca²⁺-dependent hypertrophic signaling.

Louch WE, Vangheluwe P, Bito V, Raeymaekers L, Wuytack F, Sipido KR.

Am J Physiol Heart Circ Physiol. 2012 Jun 15;302(12):H2574-82. doi: 10.1152/ajpheart.01166.2011. Epub 2012 Apr 13.

10.

Compromised myocardial energetics in hypertrophied mouse hearts diminish the beneficial effect of overexpressing SERCA2a.

Pinz I, Tian R, Belke D, Swanson E, Dillmann W, Ingwall JS.

J Biol Chem. 2011 Mar 25;286(12):10163-8. doi: 10.1074/jbc.M110.210757. Epub 2011 Jan 29.

11.

SERCA Cys674 sulphonylation and inhibition of L-type Ca2+ influx contribute to cardiac dysfunction in endotoxemic mice, independent of cGMP synthesis.

Hobai IA, Buys ES, Morse JC, Edgecomb J, Weiss EH, Armoundas AA, Hou X, Khandelwal AR, Siwik DA, Brouckaert P, Cohen RA, Colucci WS.

Am J Physiol Heart Circ Physiol. 2013 Oct 15;305(8):H1189-200. doi: 10.1152/ajpheart.00392.2012. Epub 2013 Aug 9.

12.

S-Nitrosylation of Calcium-Handling Proteins in Cardiac Adrenergic Signaling and Hypertrophy.

Irie T, Sips PY, Kai S, Kida K, Ikeda K, Hirai S, Moazzami K, Jiramongkolchai P, Bloch DB, Doulias PT, Armoundas AA, Kaneki M, Ischiropoulos H, Kranias E, Bloch KD, Stamler JS, Ichinose F.

Circ Res. 2015 Oct 9;117(9):793-803. doi: 10.1161/CIRCRESAHA.115.307157. Epub 2015 Aug 10.

13.

Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction.

Raake PW, Zhang X, Vinge LE, Brinks H, Gao E, Jaleel N, Li Y, Tang M, Most P, Dorn GW 2nd, Houser SR, Katus HA, Chen X, Koch WJ.

Circulation. 2012 May 1;125(17):2108-18. doi: 10.1161/CIRCULATIONAHA.111.044255. Epub 2012 Apr 10.

14.

PI3Kgamma is required for PDE4, not PDE3, activity in subcellular microdomains containing the sarcoplasmic reticular calcium ATPase in cardiomyocytes.

Kerfant BG, Zhao D, Lorenzen-Schmidt I, Wilson LS, Cai S, Chen SR, Maurice DH, Backx PH.

Circ Res. 2007 Aug 17;101(4):400-8. Epub 2007 Jul 5.

15.

Tumor necrosis factor alpha protects heart cultures against hypoxic damage via activation of PKA and phospholamban to prevent calcium overload.

El-Ani D, Philipchik I, Stav H, Levi M, Zerbib J, Shainberg A.

Can J Physiol Pharmacol. 2014 Nov;92(11):917-25. doi: 10.1139/cjpp-2014-0092. Epub 2014 Aug 28.

PMID:
25349921
16.

CD36-dependent signaling mediates fatty acid-induced gut release of secretin and cholecystokinin.

Sundaresan S, Shahid R, Riehl TE, Chandra R, Nassir F, Stenson WF, Liddle RA, Abumrad NA.

FASEB J. 2013 Mar;27(3):1191-202. doi: 10.1096/fj.12-217703. Epub 2012 Dec 11.

17.

Chronic SR Ca2+-ATPase inhibition causes adaptive changes in cellular Ca2+ transport.

Brittsan AG, Ginsburg KS, Chu G, Yatani A, Wolska BM, Schmidt AG, Asahi M, MacLennan DH, Bers DM, Kranias EG.

Circ Res. 2003 Apr 18;92(7):769-76. Epub 2003 Mar 13.

18.

Neuronostatin attenuates myocardial contractile function through inhibition of sarcoplasmic reticulum Ca2+-ATPase in murine heart.

Zhu X, Hu N, Chen X, Zhu MZ, Dong H, Xu X, Luo F, Hua Y, Nair S, Samson WK, Xiong L.

Cell Physiol Biochem. 2014;33(6):1921-32.

19.

Myocardial energy provision is preserved by increased utilization of glucose and ketone bodies in CD36 knockout mice.

Nakatani K, Watabe T, Masuda D, Imaizumi M, Shimosegawa E, Kobayashi T, Sairyo M, Zhu Y, Okada T, Kawase R, Nakaoka H, Naito A, Ohama T, Koseki M, Oka T, Akazawa H, Nishida M, Komuro I, Sakata Y, Hatazawa J, Yamashita S.

Metabolism. 2015 Sep;64(9):1165-74. doi: 10.1016/j.metabol.2015.05.017. Epub 2015 Jun 6.

PMID:
26130608
20.

Expression of a Gi-coupled receptor in the heart causes impaired Ca2+ handling, myofilament injury, and dilated cardiomyopathy.

McCloskey DT, Turcato S, Wang GY, Turnbull L, Zhu BQ, Bambino T, Nguyen AP, Lovett DH, Nissenson RA, Karliner JS, Baker AJ.

Am J Physiol Heart Circ Physiol. 2008 Jan;294(1):H205-12. Epub 2007 Oct 26.

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