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Bacterial virulence proteins as tools to rewire kinase pathways in yeast and immune cells.

Wei P, Wong WW, Park JS, Corcoran EE, Peisajovich SG, Onuffer JJ, Weiss A, Lim WA.

Nature. 2012 Aug 16;488(7411):384-8. doi: 10.1038/nature11259.


OspF and OspC1 are Shigella flexneri type III secretion system effectors that are required for postinvasion aspects of virulence.

Zurawski DV, Mitsuhata C, Mumy KL, McCormick BA, Maurelli AT.

Infect Immun. 2006 Oct;74(10):5964-76.


Lck dephosphorylation at Tyr-394 and inhibition of T cell antigen receptor signaling by Yersinia phosphatase YopH.

Alonso A, Bottini N, Bruckner S, Rahmouni S, Williams S, Schoenberger SP, Mustelin T.

J Biol Chem. 2004 Feb 6;279(6):4922-8.


Aurintricarboxylic acid blocks in vitro and in vivo activity of YopH, an essential virulent factor of Yersinia pestis, the agent of plague.

Liang F, Huang Z, Lee SY, Liang J, Ivanov MI, Alonso A, Bliska JB, Lawrence DS, Mustelin T, Zhang ZY.

J Biol Chem. 2003 Oct 24;278(43):41734-41.


Characterization of new substrates targeted by Yersinia tyrosine phosphatase YopH.

de la Puerta ML, Trinidad AG, del Carmen Rodríguez M, Bogetz J, Sánchez Crespo M, Mustelin T, Alonso A, Bayón Y.

PLoS One. 2009;4(2):e4431. doi: 10.1371/journal.pone.0004431.


Shigella flexneri type III secretion system effectors OspB and OspF target the nucleus to downregulate the host inflammatory response via interactions with retinoblastoma protein.

Zurawski DV, Mumy KL, Faherty CS, McCormick BA, Maurelli AT.

Mol Microbiol. 2009 Jan;71(2):350-68. doi: 10.1111/j.1365-2958.2008.06524.x.


Yeast functional genomic screens lead to identification of a role for a bacterial effector in innate immunity regulation.

Kramer RW, Slagowski NL, Eze NA, Giddings KS, Morrison MF, Siggers KA, Starnbach MN, Lesser CF.

PLoS Pathog. 2007 Feb;3(2):e21.


Shigella flexneri type III secreted effector OspF reveals new crosstalks of proinflammatory signaling pathways during bacterial infection.

Reiterer V, Grossniklaus L, Tschon T, Kasper CA, Sorg I, Arrieumerlou C.

Cell Signal. 2011 Jul;23(7):1188-96. doi: 10.1016/j.cellsig.2011.03.006.


Negative-feedback regulation of CD28 costimulation by a novel mitogen-activated protein kinase phosphatase, MKP6.

Marti F, Krause A, Post NH, Lyddane C, Dupont B, Sadelain M, King PD.

J Immunol. 2001 Jan 1;166(1):197-206.


Insight into bacterial virulence mechanisms against host immune response via the Yersinia pestis-human protein-protein interaction network.

Yang H, Ke Y, Wang J, Tan Y, Myeni SK, Li D, Shi Q, Yan Y, Chen H, Guo Z, Yuan Y, Yang X, Yang R, Du Z.

Infect Immun. 2011 Nov;79(11):4413-24. doi: 10.1128/IAI.05622-11.


Regulation of innate and adaptive immune responses by MAP kinase phosphatase 5.

Zhang Y, Blattman JN, Kennedy NJ, Duong J, Nguyen T, Wang Y, Davis RJ, Greenberg PD, Flavell RA, Dong C.

Nature. 2004 Aug 12;430(7001):793-7.


Using engineered scaffold interactions to reshape MAP kinase pathway signaling dynamics.

Bashor CJ, Helman NC, Yan S, Lim WA.

Science. 2008 Mar 14;319(5869):1539-43. doi: 10.1126/science.1151153.


Yersinia phosphatase induces mitochondrially dependent apoptosis of T cells.

Bruckner S, Rhamouni S, Tautz L, Denault JB, Alonso A, Becattini B, Salvesen GS, Mustelin T.

J Biol Chem. 2005 Mar 18;280(11):10388-94.


The Yeast Saccharomyces cerevisiae: a versatile model system for the identification and characterization of bacterial virulence proteins.

Siggers KA, Lesser CF.

Cell Host Microbe. 2008 Jul 17;4(1):8-15. doi: 10.1016/j.chom.2008.06.004. Review.

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