Similar articles for PMID: 22805202

Year	Number of Results
1989	1
1991	1
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1999	3
2000	2
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2019	7
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2024	0

1

Ca2+ dysregulation in neurons from transgenic mice expressing mutant presenilin 2.

Kipanyula MJ, Contreras L, Zampese E, Lazzari C, Wong AK, Pizzo P, Fasolato C, Pozzan T. Kipanyula MJ, et al. Aging Cell. 2012 Oct;11(5):885-93. doi: 10.1111/j.1474-9726.2012.00858.x. Epub 2012 Aug 7. Aging Cell. 2012. PMID: 22805202 Free article.

2

Presenilin mutations linked to familial Alzheimer's disease reduce endoplasmic reticulum and Golgi apparatus calcium levels.

Zatti G, Burgo A, Giacomello M, Barbiero L, Ghidoni R, Sinigaglia G, Florean C, Bagnoli S, Binetti G, Sorbi S, Pizzo P, Fasolato C. Zatti G, et al. Cell Calcium. 2006 Jun;39(6):539-50. doi: 10.1016/j.ceca.2006.03.002. Epub 2006 Apr 18. Cell Calcium. 2006. PMID: 16620965

3

Neurons overexpressing mutant presenilin-1 are more sensitive to apoptosis induced by endoplasmic reticulum-Golgi stress.

Terro F, Czech C, Esclaire F, Elyaman W, Yardin C, Baclet MC, Touchet N, Tremp G, Pradier L, Hugon J. Terro F, et al. J Neurosci Res. 2002 Aug 15;69(4):530-9. doi: 10.1002/jnr.10312. J Neurosci Res. 2002. PMID: 12210846

4

Presenilin 1 regulates the processing of beta-amyloid precursor protein C-terminal fragments and the generation of amyloid beta-protein in endoplasmic reticulum and Golgi.

Xia W, Zhang J, Ostaszewski BL, Kimberly WT, Seubert P, Koo EH, Shen J, Selkoe DJ. Xia W, et al. Biochemistry. 1998 Nov 24;37(47):16465-71. doi: 10.1021/bi9816195. Biochemistry. 1998. PMID: 9843412

5

Increased intraneuronal resting [Ca2+] in adult Alzheimer's disease mice.

Lopez JR, Lyckman A, Oddo S, Laferla FM, Querfurth HW, Shtifman A. Lopez JR, et al. J Neurochem. 2008 Apr;105(1):262-71. doi: 10.1111/j.1471-4159.2007.05135.x. Epub 2007 Nov 16. J Neurochem. 2008. PMID: 18021291 Free article.

6

Mutations in amyloid precursor protein and presenilin-1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid beta-peptide (1-42), HO and kainic acid: implications for Alzheimer's disease.

Mohmmad Abdul H, Sultana R, Keller JN, St Clair DK, Markesbery WR, Butterfield DA. Mohmmad Abdul H, et al. J Neurochem. 2006 Mar;96(5):1322-35. doi: 10.1111/j.1471-4159.2005.03647.x. J Neurochem. 2006. PMID: 16478525 Free article.

7

Enhanced caffeine-induced Ca2+ release in the 3xTg-AD mouse model of Alzheimer's disease.

Smith IF, Hitt B, Green KN, Oddo S, LaFerla FM. Smith IF, et al. J Neurochem. 2005 Sep;94(6):1711-8. doi: 10.1111/j.1471-4159.2005.03332.x. J Neurochem. 2005. PMID: 16156741 Free article.

8

Calcium Signaling and Mitochondrial Function in Presenilin 2 Knock-Out Mice: Looking for Any Loss-of-Function Phenotype Related to Alzheimer's Disease.

Rossi A, Galla L, Gomiero C, Zentilin L, Giacca M, Giorgio V, Calì T, Pozzan T, Greotti E, Pizzo P. Rossi A, et al. Cells. 2021 Jan 21;10(2):204. doi: 10.3390/cells10020204. Cells. 2021. PMID: 33494218 Free PMC article.

9

Attenuated presenilin-1 endoproteolysis enhances store-operated calcium currents in neuronal cells.

Ryazantseva M, Skobeleva K, Glushankova L, Kaznacheyeva E. Ryazantseva M, et al. J Neurochem. 2016 Mar;136(5):1085-95. doi: 10.1111/jnc.13495. Epub 2016 Jan 11. J Neurochem. 2016. PMID: 26678016 Free article.

10

Presenilin 2 mutation accelerates the onset of impairment in trace eyeblink conditioning in a mouse model of Alzheimer's disease overexpressing human mutant amyloid precursor protein.

Kishimoto Y, Kirino Y. Kishimoto Y, et al. Neurosci Lett. 2013 Mar 22;538:15-9. doi: 10.1016/j.neulet.2013.01.025. Epub 2013 Jan 28. Neurosci Lett. 2013. PMID: 23370287

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