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Items: 1 to 20 of 75

1.

Brain connexins in demyelinating diseases: therapeutic potential of glial targets.

Cotrina ML, Nedergaard M.

Brain Res. 2012 Dec 3;1487:61-8. doi: 10.1016/j.brainres.2012.07.003. Epub 2012 Jul 10. Review.

2.

Gap junction communication in myelinating glia.

Nualart-Marti A, Solsona C, Fields RD.

Biochim Biophys Acta. 2013 Jan;1828(1):69-78. doi: 10.1016/j.bbamem.2012.01.024. Epub 2012 Feb 3. Review.

3.

Curious and contradictory roles of glial connexins and pannexins in epilepsy.

Carlen PL.

Brain Res. 2012 Dec 3;1487:54-60. doi: 10.1016/j.brainres.2012.06.059. Epub 2012 Jul 11. Review.

PMID:
22796594
4.

Connexin-based intercellular communication and astrocyte heterogeneity.

Theis M, Giaume C.

Brain Res. 2012 Dec 3;1487:88-98. doi: 10.1016/j.brainres.2012.06.045. Epub 2012 Jul 10. Review.

PMID:
22789907
5.
6.

Connexin32 mutations cause loss of function in Schwann cells and oligodendrocytes leading to PNS and CNS myelination defects.

Sargiannidou I, Vavlitou N, Aristodemou S, Hadjisavvas A, Kyriacou K, Scherer SS, Kleopa KA.

J Neurosci. 2009 Apr 15;29(15):4736-49. doi: 10.1523/JNEUROSCI.0325-09.2009.

7.

Connexins are critical for normal myelination in the CNS.

Menichella DM, Goodenough DA, Sirkowski E, Scherer SS, Paul DL.

J Neurosci. 2003 Jul 2;23(13):5963-73.

8.

Connexins and gap junctions of astrocytes and oligodendrocytes in the CNS.

Nagy JI, Rash JE.

Brain Res Brain Res Rev. 2000 Apr;32(1):29-44. Review.

PMID:
10751655
9.

Oligodendrocytes are damaged by neuromyelitis optica immunoglobulin G via astrocyte injury.

Marignier R, Nicolle A, Watrin C, Touret M, Cavagna S, Varrin-Doyer M, Cavillon G, Rogemond V, Confavreux C, Honnorat J, Giraudon P.

Brain. 2010 Sep;133(9):2578-91. doi: 10.1093/brain/awq177. Epub 2010 Aug 5.

PMID:
20688809
10.

Molecular mechanisms of gap junction mutations in myelinating cells.

Sargiannidou I, Markoullis K, Kleopa KA.

Histol Histopathol. 2010 Sep;25(9):1191-206. doi: 10.14670/HH-25.1191. Review.

PMID:
20607661
11.

Nitric oxide as a potential pathological mechanism in demyelination: its differential effects on primary glial cells in vitro.

Mitrovic B, Ignarro LJ, Montestruque S, Smoll A, Merrill JE.

Neuroscience. 1994 Aug;61(3):575-85.

PMID:
7969931
13.

Connexin32-null mice develop demyelinating peripheral neuropathy.

Scherer SS, Xu YT, Nelles E, Fischbeck K, Willecke K, Bone LJ.

Glia. 1998 Sep;24(1):8-20.

PMID:
9700485
14.

Myelination by transplanted fetal and neonatal oligodendrocytes in a dysmyelinating mutant.

Friedman E, Nilaver G, Carmel P, Perlow M, Spatz L, Latov N.

Brain Res. 1986 Jul 16;378(1):142-6.

PMID:
2427154
15.

Transgenic replacement of Cx32 in gap junction-deficient oligodendrocytes rescues the phenotype of a hypomyelinating leukodystrophy model.

Schiza N, Sargiannidou I, Kagiava A, Karaiskos C, Nearchou M, Kleopa KA.

Hum Mol Genet. 2015 Apr 1;24(7):2049-64. doi: 10.1093/hmg/ddu725. Epub 2014 Dec 18.

PMID:
25524707
16.

Biology of oligodendrocyte and myelin in the mammalian central nervous system.

Baumann N, Pham-Dinh D.

Physiol Rev. 2001 Apr;81(2):871-927. Review.

17.

Early disruption of glial communication via connexin gap junction in multiple sclerosis, Baló's disease and neuromyelitis optica.

Masaki K.

Neuropathology. 2015 Oct;35(5):469-80. doi: 10.1111/neup.12211. Epub 2015 May 28. Review.

PMID:
26016402
18.

Connexin47 protein phosphorylation and stability in oligodendrocytes depend on expression of Connexin43 protein in astrocytes.

May D, Tress O, Seifert G, Willecke K.

J Neurosci. 2013 May 1;33(18):7985-96. doi: 10.1523/JNEUROSCI.5874-12.2013.

19.

Connexin and pannexin hemichannels in inflammatory responses of glia and neurons.

Bennett MV, Garré JM, Orellana JA, Bukauskas FF, Nedergaard M, Sáez JC.

Brain Res. 2012 Dec 3;1487:3-15. doi: 10.1016/j.brainres.2012.08.042. Epub 2012 Sep 10. Review.

20.

Olig2-lineage cells preferentially differentiate into oligodendrocytes but their processes degenerate at the chronic demyelinating stage of proteolipid protein-overexpressing mouse.

Shimizu T, Tanaka KF, Takebayashi H, Higashi M, Wisesmith W, Ono K, Hitoshi S, Ikenaka K.

J Neurosci Res. 2013 Feb;91(2):178-86. doi: 10.1002/jnr.23153. Epub 2012 Nov 22.

PMID:
23172790

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