Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 93

1.

The role of CREB-H transcription factor in triglyceride metabolism.

Lee AH.

Curr Opin Lipidol. 2012 Apr;23(2):141-6. doi: 10.1097/MOL.0b013e3283508fed. Review.

2.

The transcription factor cyclic AMP-responsive element-binding protein H regulates triglyceride metabolism.

Lee JH, Giannikopoulos P, Duncan SA, Wang J, Johansen CT, Brown JD, Plutzky J, Hegele RA, Glimcher LH, Lee AH.

Nat Med. 2011 Jun 12;17(7):812-5. doi: 10.1038/nm.2347.

3.

Deletion of the basement membrane heparan sulfate proteoglycan type XVIII collagen causes hypertriglyceridemia in mice and humans.

Bishop JR, Passos-Bueno MR, Fong L, Stanford KI, Gonzales JC, Yeh E, Young SG, Bensadoun A, Witztum JL, Esko JD, Moulton KS.

PLoS One. 2010 Nov 10;5(11):e13919. doi: 10.1371/journal.pone.0013919.

4.

Mice lacking prostaglandin E receptor subtype 4 manifest disrupted lipid metabolism attributable to impaired triglyceride clearance.

Cai Y, Ying F, Song E, Wang Y, Xu A, Vanhoutte PM, Tang EH.

FASEB J. 2015 Dec;29(12):4924-36. doi: 10.1096/fj.15-274597. Epub 2015 Aug 13.

5.

Amelioration of hypertriglyceridemia with hypo-alpha-cholesterolemia in LPL deficient mice by hematopoietic cell-derived LPL.

Ding Y, Zhang L, Wang Y, Huang W, Tang Y, Bai L, Ross CJ, Hayden MR, Liu G.

PLoS One. 2011;6(9):e25620. doi: 10.1371/journal.pone.0025620. Epub 2011 Sep 29.

6.

APOA5 and triglyceride metabolism, lesson from human APOA5 deficiency.

Calandra S, Priore Oliva C, Tarugi P, Bertolini S.

Curr Opin Lipidol. 2006 Apr;17(2):122-7. Review.

PMID:
16531747
7.

Severe hypertriglyceridemia, reduced high density lipoprotein, and neonatal death in lipoprotein lipase knockout mice. Mild hypertriglyceridemia with impaired very low density lipoprotein clearance in heterozygotes.

Weinstock PH, Bisgaier CL, Aalto-Setälä K, Radner H, Ramakrishnan R, Levak-Frank S, Essenburg AD, Zechner R, Breslow JL.

J Clin Invest. 1995 Dec;96(6):2555-68.

8.

Effects of six APOA5 variants, identified in patients with severe hypertriglyceridemia, on in vitro lipoprotein lipase activity and receptor binding.

Dorfmeister B, Zeng WW, Dichlberger A, Nilsson SK, Schaap FG, Hubacek JA, Merkel M, Cooper JA, Lookene A, Putt W, Whittall R, Lee PJ, Lins L, Delsaux N, Nierman M, Kuivenhoven JA, Kastelein JJ, Vrablik M, Olivecrona G, Schneider WJ, Heeren J, Humphries SE, Talmud PJ.

Arterioscler Thromb Vasc Biol. 2008 Oct;28(10):1866-71. doi: 10.1161/ATVBAHA.108.172866. Epub 2008 Jul 17.

9.
11.

Leu452His mutation in lipoprotein lipase gene transfer associated with hypertriglyceridemia in mice in vivo.

Sun K, Yang W, Huang Y, Wang Y, Xiang L, Qi J.

PLoS One. 2013 Sep 26;8(9):e75462. doi: 10.1371/journal.pone.0075462. eCollection 2013.

12.

Genetic deficiency of apolipoprotein D in the mouse is associated with nonfasting hypertriglyceridemia and hyperinsulinemia.

Jiménez-Palomares M, Cózar-Castellano I, Ganfornina MD, Sánchez D, Perdomo G.

Metabolism. 2011 Dec;60(12):1767-74. doi: 10.1016/j.metabol.2011.04.013. Epub 2011 May 31.

PMID:
21632073
13.

ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis.

Schaap FG, Rensen PC, Voshol PJ, Vrins C, van der Vliet HN, Chamuleau RA, Havekes LM, Groen AK, van Dijk KW.

J Biol Chem. 2004 Jul 2;279(27):27941-7. Epub 2004 Apr 16.

14.

The role and mode of action of apolipoproteins CIII and AV: synergistic actors in triglyceride metabolism?

van Dijk KW, Rensen PC, Voshol PJ, Havekes LM.

Curr Opin Lipidol. 2004 Jun;15(3):239-46. Review.

PMID:
15166778
15.

Apolipoprotein AV accelerates plasma hydrolysis of triglyceride-rich lipoproteins by interaction with proteoglycan-bound lipoprotein lipase.

Merkel M, Loeffler B, Kluger M, Fabig N, Geppert G, Pennacchio LA, Laatsch A, Heeren J.

J Biol Chem. 2005 Jun 3;280(22):21553-60. Epub 2005 Mar 17.

16.

Physiological regulation of lipoprotein lipase.

Kersten S.

Biochim Biophys Acta. 2014 Jul;1841(7):919-33. doi: 10.1016/j.bbalip.2014.03.013. Epub 2014 Apr 8. Review.

PMID:
24721265
17.

[Genetic basis of primary hypertriglyceridemia].

Gotoda T.

Nihon Rinsho. 2013 Sep;71(9):1558-64. Review. Japanese.

PMID:
24205714
18.

Post-heparin LPL activity measurement using VLDL as a substrate: a new robust method for routine assessment of plasma triglyceride lipolysis defects.

Di Filippo M, Marçais C, Charrière S, Marmontel O, Broyer M, Delay M, Merlin M, Nollace A, Valéro R, Lagarde M, Pruneta-Deloche V, Moulin P, Sassolas A.

PLoS One. 2014 May 2;9(5):e96482. doi: 10.1371/journal.pone.0096482. eCollection 2014. Erratum in: PLoS One. 2014;9(5):e99721.

19.

Very low density lipoprotein (VLDL) receptor-deficient mice have reduced lipoprotein lipase activity. Possible causes of hypertriglyceridemia and reduced body mass with VLDL receptor deficiency.

Yagyu H, Lutz EP, Kako Y, Marks S, Hu Y, Choi SY, Bensadoun A, Goldberg IJ.

J Biol Chem. 2002 Mar 22;277(12):10037-43. Epub 2002 Jan 14.

20.

Inherited apolipoprotein A-V deficiency in severe hypertriglyceridemia.

Priore Oliva C, Pisciotta L, Li Volti G, Sambataro MP, Cantafora A, Bellocchio A, Catapano A, Tarugi P, Bertolini S, Calandra S.

Arterioscler Thromb Vasc Biol. 2005 Feb;25(2):411-7. Epub 2004 Dec 9.

Supplemental Content

Support Center