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Items: 1 to 20 of 63

1.

Chlamydophila pneumoniae infection induces alterations in vascular contractile responses.

Deniset JF, Hedley TE, Dibrov E, Pierce GN.

Am J Pathol. 2012 Mar;180(3):1264-72. doi: 10.1016/j.ajpath.2011.11.005. Epub 2011 Dec 31.

PMID:
22214836
2.

Acute Chlamydia pneumoniae infection causes coronary endothelial dysfunction in pigs.

Liuba P, Pesonen E, Paakkari I, Batra S, Forslid A, Kovanen P, Pentikäinen M, Persson K, Sandström S.

Atherosclerosis. 2003 Apr;167(2):215-22.

PMID:
12818403
3.

Red wine prevents homocysteine-induced endothelial dysfunction in porcine coronary arteries.

Fu W, Conklin BS, Lin PH, Lumsden AB, Yao Q, Chen C.

J Surg Res. 2003 Nov;115(1):82-91.

PMID:
14572777
4.

Chlamydia heat shock protein 60 decreases expression of endothelial nitric oxide synthase in human and porcine coronary artery endothelial cells.

Chen C, Chai H, Wang X, Lin PH, Yao Q.

Cardiovasc Res. 2009 Sep 1;83(4):768-77. doi: 10.1093/cvr/cvp150. Epub 2009 May 14. Erratum in: Cardiovasc Res. 2009 Nov 1;84(2):336.

6.

Homocysteine decreases endothelium-dependent vasorelaxation in porcine arteries.

Chen C, Conklin BS, Ren Z, Zhong DS.

J Surg Res. 2002 Jan;102(1):22-30.

PMID:
11792147
9.
10.

GroEL1, a heat shock protein 60 of Chlamydia pneumoniae, induces lectin-like oxidized low-density lipoprotein receptor 1 expression in endothelial cells and enhances atherogenesis in hypercholesterolemic rabbits.

Lin FY, Lin YW, Huang CY, Chang YJ, Tsao NW, Chang NC, Ou KL, Chen TL, Shih CM, Chen YH.

J Immunol. 2011 Apr 1;186(7):4405-14. doi: 10.4049/jimmunol.1003116. Epub 2011 Mar 7.

11.

Chlamydophila pneumoniae infection leads to smooth muscle cell proliferation and thickening in the coronary artery without contributions from a host immune response.

Deniset JF, Cheung PK, Dibrov E, Lee K, Steigerwald S, Pierce GN.

Am J Pathol. 2010 Feb;176(2):1028-37. doi: 10.2353/ajpath.2010.090645. Epub 2009 Dec 17.

12.

Estrogen blocks homocysteine-induced endothelial dysfunction in porcine coronary arteries(1,2).

Spencer TA, Chai H, Fu W, Ramaswami G, Cox MW, Conklin BS, Lin PH, Lumsden AB, Yao Q, Chen C.

J Surg Res. 2004 May 1;118(1):83-90.

PMID:
15093721
13.

Mechanisms causing coronary microvascular dysfunction following crystalloid cardioplegia and reperfusion.

Sellke FW, Friedman M, Dai HB, Shafique T, Schoen FJ, Weintraub RM, Johnson RG.

Cardiovasc Res. 1993 Nov;27(11):1925-32.

PMID:
8287398
14.

Role of oxidative stress in multiparity-induced endothelial dysfunction.

Tawfik HE, Cena J, Schulz R, Kaufman S.

Am J Physiol Heart Circ Physiol. 2008 Oct;295(4):H1736-42. doi: 10.1152/ajpheart.87.2008. Epub 2008 Aug 29.

15.
16.

Role of endothelium and nitric oxide in histamine-induced responses in human cranial arteries and detection of mRNA encoding H1- and H2-receptors by RT-PCR.

Jansen-Olesen I, Ottosson A, Cantera L, Strunk S, Lassen LH, Olesen J, Mortensen A, Engel U, Edvinsson L.

Br J Pharmacol. 1997 May;121(1):41-8.

18.

Effects of progesterone and estrogen on endothelial dysfunction in porcine coronary arteries.

Cox MW, Fu W, Chai H, Paladugu R, Lin PH, Lumsden AB, Yao Q, Chen C.

J Surg Res. 2005 Mar;124(1):104-11.

PMID:
15734487
19.

Increased superoxide anion production by interleukin-1beta impairs nitric oxide-mediated relaxation in resistance arteries.

Jiménez-Altayó F, Briones AM, Giraldo J, Planas AM, Salaices M, Vila E.

J Pharmacol Exp Ther. 2006 Jan;316(1):42-52. Epub 2005 Sep 23.

20.

Testosterone relaxes rabbit coronary arteries and aorta.

Yue P, Chatterjee K, Beale C, Poole-Wilson PA, Collins P.

Circulation. 1995 Feb 15;91(4):1154-60.

PMID:
7850954

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