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Items: 1 to 20 of 358

1.

The critical role of Notch ligand Delta-like 1 in the pathogenesis of influenza A virus (H1N1) infection.

Ito T, Allen RM, Carson WF 4th, Schaller M, Cavassani KA, Hogaboam CM, Lukacs NW, Matsukawa A, Kunkel SL.

PLoS Pathog. 2011 Nov;7(11):e1002341. doi: 10.1371/journal.ppat.1002341. Epub 2011 Nov 3.

2.
3.

Dengue virus up-regulates expression of notch ligands Dll1 and Dll4 through interferon-β signalling pathway.

Li Y, Wu S, Pu J, Huang X, Zhang P.

Immunology. 2015 Jan;144(1):127-38. doi: 10.1111/imm.12357.

4.

CLEC5A-Mediated Enhancement of the Inflammatory Response in Myeloid Cells Contributes to Influenza Virus Pathogenicity In Vivo.

Teng O, Chen ST, Hsu TL, Sia SF, Cole S, Valkenburg SA, Hsu TY, Zheng JT, Tu W, Bruzzone R, Peiris JS, Hsieh SL, Yen HL.

J Virol. 2016 Dec 16;91(1). pii: e01813-16. Print 2017 Jan 1.

5.

Unique type I interferon responses determine the functional fate of migratory lung dendritic cells during influenza virus infection.

Moltedo B, Li W, Yount JS, Moran TM.

PLoS Pathog. 2011 Nov;7(11):e1002345. doi: 10.1371/journal.ppat.1002345. Epub 2011 Nov 3.

6.

NK cells regulate CD8+ T cell priming and dendritic cell migration during influenza A infection by IFN-γ and perforin-dependent mechanisms.

Ge MQ, Ho AW, Tang Y, Wong KH, Chua BY, Gasser S, Kemeny DM.

J Immunol. 2012 Sep 1;189(5):2099-109. doi: 10.4049/jimmunol.1103474. Epub 2012 Aug 6.

7.

Severe influenza A(H1N1)pdm09 infection induces thymic atrophy through activating innate CD8(+)CD44(hi) T cells by upregulating IFN-γ.

Liu B, Zhang X, Deng W, Liu J, Li H, Wen M, Bao L, Qu J, Liu Y, Li F, An Y, Qin C, Cao B, Wang C.

Cell Death Dis. 2014 Oct 2;5:e1440. doi: 10.1038/cddis.2014.323.

8.

Impaired immune responses in the lungs of aged mice following influenza infection.

Toapanta FR, Ross TM.

Respir Res. 2009 Nov 18;10:112. doi: 10.1186/1465-9921-10-112.

9.

Cross-protective immunity against influenza A/H1N1 virus challenge in mice immunized with recombinant vaccine expressing HA gene of influenza A/H5N1 virus.

Yang S, Niu S, Guo Z, Yuan Y, Xue K, Liu S, Jin H.

Virol J. 2013 Sep 22;10:291. doi: 10.1186/1743-422X-10-291.

10.

Reduced interferon-α production by dendritic cells in type 1 diabetes does not impair immunity to influenza virus.

Kreuzer D, Nikoopour E, Au BC, Krougly O, Lee-Chan E, Summers KL, Haeryfar SM, Singh B.

Clin Exp Immunol. 2015 Feb;179(2):245-55. doi: 10.1111/cei.12462.

11.

Seasonal and pandemic influenza H1N1 viruses induce differential expression of SOCS-1 and RIG-I genes and cytokine/chemokine production in macrophages.

Ramírez-Martínez G, Cruz-Lagunas A, Jiménez-Alvarez L, Espinosa E, Ortíz-Quintero B, Santos-Mendoza T, Herrera MT, Canché-Pool E, Mendoza C, Bañales JL, García-Moreno SA, Morán J, Cabello C, Orozco L, Aguilar-Delfín I, Hidalgo-Miranda A, Romero S, Suratt BT, Selman M, Zúñiga J.

Cytokine. 2013 Apr;62(1):151-9. doi: 10.1016/j.cyto.2013.01.018. Epub 2013 Feb 21.

12.

TLR signaling fine-tunes anti-influenza B cell responses without regulating effector T cell responses.

Heer AK, Shamshiev A, Donda A, Uematsu S, Akira S, Kopf M, Marsland BJ.

J Immunol. 2007 Feb 15;178(4):2182-91.

13.

Memory CD4 T cells direct protective responses to influenza virus in the lungs through helper-independent mechanisms.

Teijaro JR, Verhoeven D, Page CA, Turner D, Farber DL.

J Virol. 2010 Sep;84(18):9217-26. doi: 10.1128/JVI.01069-10. Epub 2010 Jun 30.

14.

Autophagy is involved in regulating the immune response of dendritic cells to influenza A (H1N1) pdm09 infection.

Zang F, Chen Y, Lin Z, Cai Z, Yu L, Xu F, Wang J, Zhu W, Lu H.

Immunology. 2016 May;148(1):56-69. doi: 10.1111/imm.12587. Epub 2016 Feb 25.

15.

Highly pathogenic avian influenza A H5N1 and pandemic H1N1 virus infections have different phenotypes in Toll-like receptor 3 knockout mice.

Leung YH, Nicholls JM, Ho CK, Sia SF, Mok CK, Valkenburg SA, Cheung P, Hui KP, Chan RW, Guan Y, Akira S, Peiris JS.

J Gen Virol. 2014 Sep;95(Pt 9):1870-9. doi: 10.1099/vir.0.066258-0. Epub 2014 May 30.

16.

Enhanced viral clearance in interleukin-18 gene-deficient mice after pulmonary infection with influenza A virus.

Van Der Sluijs KF, Van Elden LJ, Arens R, Nijhuis M, Schuurman R, Florquin S, Kwakkel J, Akira S, Jansen HM, Lutter R, Van Der Polls T.

Immunology. 2005 Jan;114(1):112-20.

17.

Activation of the RIG-I pathway during influenza vaccination enhances the germinal center reaction, promotes T follicular helper cell induction, and provides a dose-sparing effect and protective immunity.

Kulkarni RR, Rasheed MA, Bhaumik SK, Ranjan P, Cao W, Davis C, Marisetti K, Thomas S, Gangappa S, Sambhara S, Murali-Krishna K.

J Virol. 2014 Dec;88(24):13990-4001. doi: 10.1128/JVI.02273-14. Epub 2014 Sep 24.

18.

Type I interferon plays opposing roles in cytotoxicity and interferon-γ production by natural killer and CD8 T cells after influenza A virus infection in mice.

Arimori Y, Nakamura R, Yamada H, Shibata K, Maeda N, Kase T, Yoshikai Y.

J Innate Immun. 2014;6(4):456-66. doi: 10.1159/000356824. Epub 2014 Jan 10.

19.

MyD88 signaling is indispensable for primary influenza A virus infection but dispensable for secondary infection.

Seo SU, Kwon HJ, Song JH, Byun YH, Seong BL, Kawai T, Akira S, Kweon MN.

J Virol. 2010 Dec;84(24):12713-22. doi: 10.1128/JVI.01675-10. Epub 2010 Oct 13.

20.

Andrographolide as an anti-H1N1 drug and the mechanism related to retinoic acid-inducible gene-I-like receptors signaling pathway.

Yu B, Dai CQ, Jiang ZY, Li EQ, Chen C, Wu XL, Chen J, Liu Q, Zhao CL, He JX, Ju DH, Chen XY.

Chin J Integr Med. 2014 Jul;20(7):540-5. doi: 10.1007/s11655-014-1860-0. Epub 2014 Jun 28.

PMID:
24972581

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