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Items: 1 to 20 of 83

1.

Long term changes in phospho-APP and tau aggregation in the 3xTg-AD mice following cerebral ischemia.

Koike MA, Garcia FG, Kitazawa M, Green KN, Laferla FM.

Neurosci Lett. 2011 May 9;495(1):55-9. doi: 10.1016/j.neulet.2011.03.034. Epub 2011 Mar 23.

2.

Oligemic hypoperfusion differentially affects tau and amyloid-{beta}.

Koike MA, Green KN, Blurton-Jones M, Laferla FM.

Am J Pathol. 2010 Jul;177(1):300-10. doi: 10.2353/ajpath.2010.090750. Epub 2010 May 14.

3.

Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimer's disease.

Manczak M, Reddy PH.

Hum Mol Genet. 2012 Dec 1;21(23):5131-46. doi: 10.1093/hmg/dds360. Epub 2012 Aug 27.

4.

Chronic cerebral hypoperfusion enhances Tau hyperphosphorylation and reduces autophagy in Alzheimer's disease mice.

Qiu L, Ng G, Tan EK, Liao P, Kandiah N, Zeng L.

Sci Rep. 2016 Apr 6;6:23964. doi: 10.1038/srep23964.

5.

Fyn knock-down increases Aβ, decreases phospho-tau, and worsens spatial learning in 3×Tg-AD mice.

Minami SS, Clifford TG, Hoe HS, Matsuoka Y, Rebeck GW.

Neurobiol Aging. 2012 Apr;33(4):825.e15-24. doi: 10.1016/j.neurobiolaging.2011.05.014. Epub 2011 Jul 7.

6.

Mifepristone alters amyloid precursor protein processing to preclude amyloid beta and also reduces tau pathology.

Baglietto-Vargas D, Medeiros R, Martinez-Coria H, LaFerla FM, Green KN.

Biol Psychiatry. 2013 Sep 1;74(5):357-66. doi: 10.1016/j.biopsych.2012.12.003. Epub 2013 Jan 8.

7.
9.

Intraneuronal APP, not free Aβ peptides in 3xTg-AD mice: implications for tau versus Aβ-mediated Alzheimer neurodegeneration.

Winton MJ, Lee EB, Sun E, Wong MM, Leight S, Zhang B, Trojanowski JQ, Lee VM.

J Neurosci. 2011 May 25;31(21):7691-9. doi: 10.1523/JNEUROSCI.6637-10.2011. Retraction in: J Neurosci. 2015 Feb 25;35(8):3724.

10.

Involvement of WAVE accumulation in Abeta/APP pathology-dependent tangle modification in Alzheimer's disease.

Takata K, Kitamura Y, Nakata Y, Matsuoka Y, Tomimoto H, Taniguchi T, Shimohama S.

Am J Pathol. 2009 Jul;175(1):17-24. doi: 10.2353/ajpath.2009.080908. Epub 2009 Jun 4.

11.

Endogenous murine tau promotes neurofibrillary tangles in 3xTg-AD mice without affecting cognition.

Baglietto-Vargas D, Kitazawa M, Le EJ, Estrada-Hernandez T, Rodriguez-Ortiz CJ, Medeiros R, Green KN, LaFerla FM.

Neurobiol Dis. 2014 Feb;62:407-15. doi: 10.1016/j.nbd.2013.10.019. Epub 2013 Oct 29.

PMID:
24176788
12.

Brain ischemia with Alzheimer phenotype dysregulates Alzheimer's disease-related proteins.

Ułamek-Kozioł M, Pluta R, Bogucka-Kocka A, Januszewski S, Kocki J, Czuczwar SJ.

Pharmacol Rep. 2016 Jun;68(3):582-91. doi: 10.1016/j.pharep.2016.01.006. Epub 2016 Jan 27. Review.

PMID:
26940197
13.

Phosphorylated tau in neuritic plaques of APP(sw)/Tau (vlw) transgenic mice and Alzheimer disease.

Pérez M, Morán MA, Ferrer I, Avila J, Gómez-Ramos P.

Acta Neuropathol. 2008 Oct;116(4):409-18. doi: 10.1007/s00401-008-0420-0. Epub 2008 Aug 5.

PMID:
18679696
14.

Profile for amyloid-beta and tau expression in primary cortical cultures from 3xTg-AD mice.

Vale C, Alonso E, Rubiolo JA, Vieytes MR, LaFerla FM, Giménez-Llort L, Botana LM.

Cell Mol Neurobiol. 2010 May;30(4):577-90. doi: 10.1007/s10571-009-9482-3. Epub 2009 Nov 27.

PMID:
19943189
15.

Immunolesion-induced loss of cholinergic projection neurones promotes β-amyloidosis and tau hyperphosphorylation in the hippocampus of triple-transgenic mice.

Härtig W, Saul A, Kacza J, Grosche J, Goldhammer S, Michalski D, Wirths O.

Neuropathol Appl Neurobiol. 2014 Feb;40(2):106-20. doi: 10.1111/nan.12050.

PMID:
23566195
16.

High dietary consumption of trans fatty acids decreases brain docosahexaenoic acid but does not alter amyloid-beta and tau pathologies in the 3xTg-AD model of Alzheimer's disease.

Phivilay A, Julien C, Tremblay C, Berthiaume L, Julien P, Giguère Y, Calon F.

Neuroscience. 2009 Mar 3;159(1):296-307. doi: 10.1016/j.neuroscience.2008.12.006. Epub 2008 Dec 14.

PMID:
19135506
17.

Distinct temporal and anatomical distributions of amyloid-β and tau abnormalities following controlled cortical impact in transgenic mice.

Tran HT, Sanchez L, Esparza TJ, Brody DL.

PLoS One. 2011;6(9):e25475. doi: 10.1371/journal.pone.0025475. Epub 2011 Sep 29.

19.

Physiological mouse brain Abeta levels are not related to the phosphorylation state of threonine-668 of Alzheimer's APP.

Sano Y, Nakaya T, Pedrini S, Takeda S, Iijima-Ando K, Iijima K, Mathews PM, Itohara S, Gandy S, Suzuki T.

PLoS One. 2006 Dec 20;1:e51.

20.

Modeling Alzheimer's disease in transgenic mice: effect of age and of presenilin1 on amyloid biochemistry and pathology in APP/London mice.

Dewachter I, van Dorpe J, Spittaels K, Tesseur I, Van Den Haute C, Moechars D, Van Leuven F.

Exp Gerontol. 2000 Sep;35(6-7):831-41. Review.

PMID:
11053674

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