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Items: 1 to 20 of 120

1.

Melatonin improves inflammation processes in liver of senescence-accelerated prone male mice (SAMP8).

Cuesta S, Kireev R, Forman K, García C, Escames G, Ariznavarreta C, Vara E, Tresguerres JA.

Exp Gerontol. 2010 Dec;45(12):950-6. doi: 10.1016/j.exger.2010.08.016.

PMID:
20817086
2.

Effect of growth hormone treatment on pancreatic inflammation, oxidative stress, and apoptosis related to aging in SAMP8 mice.

Cuesta S, Kireev R, García C, Forman K, Vara E, Tresguerres JA.

Rejuvenation Res. 2011 Oct;14(5):501-12. doi: 10.1089/rej.2011.1166.

PMID:
21958002
3.

Beneficial effects of melatonin on cardiological alterations in a murine model of accelerated aging.

Forman K, Vara E, García C, Kireev R, Cuesta S, Acuña-Castroviejo D, Tresguerres JA.

J Pineal Res. 2010 Oct;49(3):312-20. doi: 10.1111/j.1600-079X.2010.00800.x.

PMID:
20738757
4.

Melatonin decreases the expression of inflammation and apoptosis markers in the lung of a senescence-accelerated mice model.

Puig Á, Rancan L, Paredes SD, Carrasco A, Escames G, Vara E, Tresguerres JA.

Exp Gerontol. 2016 Mar;75:1-7. doi: 10.1016/j.exger.2015.11.021.

PMID:
26656745
5.

Melatonin is able to prevent the liver of old castrated female rats from oxidative and pro-inflammatory damage.

Kireev RA, Tresguerres AC, Garcia C, Ariznavarreta C, Vara E, Tresguerres JA.

J Pineal Res. 2008 Nov;45(4):394-402. doi: 10.1111/j.1600-079X.2008.00606.x.

PMID:
18573161
6.

Melatonin can improve insulin resistance and aging-induced pancreas alterations in senescence-accelerated prone male mice (SAMP8).

Cuesta S, Kireev R, García C, Rancan L, Vara E, Tresguerres JA.

Age (Dordr). 2013 Jun;35(3):659-71. doi: 10.1007/s11357-012-9397-7.

7.

Beneficial effect of melatonin treatment on inflammation, apoptosis and oxidative stress on pancreas of a senescence accelerated mice model.

Cuesta S, Kireev R, García C, Forman K, Escames G, Vara E, Tresguerres JA.

Mech Ageing Dev. 2011 Nov-Dec;132(11-12):573-82. doi: 10.1016/j.mad.2011.10.005.

PMID:
22024129
8.

Effect of a combined treatment with growth hormone and melatonin in the cardiological aging on male SAMP8 mice.

Forman K, Vara E, García C, Kireev R, Cuesta S, Escames G, Tresguerres JA.

J Gerontol A Biol Sci Med Sci. 2011 Aug;66(8):823-34. doi: 10.1093/gerona/glr083.

PMID:
21665987
9.

The senescence-accelerated mouse-prone 8 is not a suitable model for the investigation of cardiac inflammation and oxidative stress and their modulation by dietary phytochemicals.

Schiborr C, Schwamm D, Kocher A, Rimbach G, Eckert GP, Frank J.

Pharmacol Res. 2013 Aug;74:113-20. doi: 10.1016/j.phrs.2013.06.004.

PMID:
23792082
10.

Amelioration of cognitive ability in senescence-accelerated mouse prone 8 (SAMP8) by intra-bone marrow-bone marrow transplantation.

Li M, Inaba M, Guo K, Abraham NG, Ikehara S.

Neurosci Lett. 2009 Nov 6;465(1):36-40. doi: 10.1016/j.neulet.2009.09.001.

PMID:
19733629
11.

Hormonal regulation of pro-inflammatory and lipid peroxidation processes in liver of old ovariectomized female rats.

Kireev RA, Tresguerres AC, Garcia C, Borras C, Ariznavarreta C, Vara E, Vina J, Tresguerres JA.

Biogerontology. 2010 Apr;11(2):229-43. doi: 10.1007/s10522-009-9242-2.

PMID:
19633997
12.

Cardiac oxidative stress and inflammation are similar in SAMP8 and SAMR1 mice and unaltered by curcumin and Ginkgo biloba extract intake.

Schiborr C, Eckert GP, Weissenberger J, Müller WE, Schwamm D, Grune T, Rimbach G, Frank J.

Curr Pharm Biotechnol. 2010 Dec;11(8):861-7.

PMID:
20874680
13.

Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice.

Caballero B, Vega-Naredo I, Sierra V, Huidobro-Fernández C, Soria-Valles C, De Gonzalo-Calvo D, Tolivia D, Gutierrez-Cuesta J, Pallas M, Camins A, Rodríguez-Colunga MJ, Coto-Montes A.

J Pineal Res. 2008 Oct;45(3):302-11. doi: 10.1111/j.1600-079X.2008.00591.x.

PMID:
18410310
14.

Regional age-related changes in neuronal nitric oxide synthase (nNOS), messenger RNA levels and activity in SAMP8 brain.

Colas D, Gharib A, Bezin L, Morales A, Guidon G, Cespuglio R, Sarda N.

BMC Neurosci. 2006 Dec 21;7:81.

15.

Effect of chronic melatonin administration on several physiological parameters from old Wistar rats and SAMP8 mice.

Tresguerres JA, Kireev R, Forman K, Cuesta S, Tresguerres AF, Vara E.

Curr Aging Sci. 2012 Dec;5(3):242-53. Review.

PMID:
23387890
16.

Modulation of Macrophage Polarization and HMGB1-TLR2/TLR4 Cascade Plays a Crucial Role for Cardiac Remodeling in Senescence-Accelerated Prone Mice.

Karuppagounder V, Giridharan VV, Arumugam S, Sreedhar R, Palaniyandi SS, Krishnamurthy P, Quevedo J, Watanabe K, Konishi T, Thandavarayan RA.

PLoS One. 2016 Apr 12;11(4):e0152922. doi: 10.1371/journal.pone.0152922.

18.

Changes in expressions of proinflammatory cytokines IL-1beta, TNF-alpha and IL-6 in the brain of senescence accelerated mouse (SAM) P8.

Tha KK, Okuma Y, Miyazaki H, Murayama T, Uehara T, Hatakeyama R, Hayashi Y, Nomura Y.

Brain Res. 2000 Dec 1;885(1):25-31.

PMID:
11121526
19.

[Effects of melatonin in the brain of the senescence-accelerated mice-prone 8 (SAMP8) model].

Gutierrez-Cuesta J, Tajes M, Jimenez A, Camins A, Pallas M.

Rev Neurol. 2011 May 16;52(10):618-22. Review. Spanish.

20.

Chronic melatonin treatment reduces the age-dependent inflammatory process in senescence-accelerated mice.

Rodríguez MI, Escames G, López LC, López A, García JA, Ortiz F, Acuña-Castroviejo D.

J Pineal Res. 2007 Apr;42(3):272-9.

PMID:
17349026
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