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Items: 1 to 20 of 68

1.

Cross-talk between MAPKs and PI-3K pathways alters the functional density of I(K) channels in hypertrophied hearts.

Zhao A, Alvin Z, Laurence G, Li C, Haddad GE.

Ethn Dis. 2010 Winter;20(1 Suppl 1):S1-219-24.

2.

Basal and IGF-I-dependent regulation of potassium channels by MAP kinases and PI3-kinase during eccentric cardiac hypertrophy.

Teos LY, Zhao A, Alvin Z, Laurence GG, Li C, Haddad GE.

Am J Physiol Heart Circ Physiol. 2008 Nov;295(5):H1834-45. doi: 10.1152/ajpheart.321.2008. Epub 2008 Aug 29.

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5.

The cross-talk between angiotensin and insulin differentially affects phosphatidylinositol 3-kinase- and mitogen-activated protein kinase-mediated signaling in rat heart: implications for insulin resistance.

Carvalheira JB, Calegari VC, Zecchin HG, Nadruz W Jr, Guimarães RB, Ribeiro EB, Franchini KG, Velloso LA, Saad MJ.

Endocrinology. 2003 Dec;144(12):5604-14. Epub 2003 Sep 4.

PMID:
12960006
6.

Regulation of L-type inward calcium channel activity by captopril and angiotensin II via the phosphatidyl inositol 3-kinase pathway in cardiomyocytes from volume-overload hypertrophied rat hearts.

Alvin Z, Laurence GG, Coleman BR, Zhao A, Hajj-Moussa M, Haddad GE.

Can J Physiol Pharmacol. 2011 Mar;89(3):206-15. doi: 10.1139/Y11-011.

7.

Stage-specific differential activation of mitogen-activated protein kinases in hypertrophied and failing rat hearts.

Hayashida W, Kihara Y, Yasaka A, Inagaki K, Iwanaga Y, Sasayama S.

J Mol Cell Cardiol. 2001 Apr;33(4):733-44.

PMID:
11273726
8.

MEK, p38, and PI-3K mediate cross talk between EGFR and TNFR in enhancing hepatocyte growth factor production from human mesenchymal stem cells.

Wang Y, Weil BR, Herrmann JL, Abarbanell AM, Tan J, Markel TA, Kelly ML, Meldrum DR.

Am J Physiol Cell Physiol. 2009 Nov;297(5):C1284-93. doi: 10.1152/ajpcell.00183.2009. Epub 2009 Aug 19.

9.

Mitogen-activated protein kinases (p38 and c-Jun NH2-terminal kinase) are differentially regulated during cardiac volume and pressure overload hypertrophy.

Sopontammarak S, Aliharoob A, Ocampo C, Arcilla RA, Gupta MP, Gupta M.

Cell Biochem Biophys. 2005;43(1):61-76.

PMID:
16043884
10.
11.

Src and multiple MAP kinase activation in cardiac hypertrophy and congestive heart failure under chronic pressure-overload: comparison with acute mechanical stretch.

Takeishi Y, Huang Q, Abe J, Glassman M, Che W, Lee JD, Kawakatsu H, Lawrence EG, Hoit BD, Berk BC, Walsh RA.

J Mol Cell Cardiol. 2001 Sep;33(9):1637-48.

PMID:
11549343
13.

Angiotensin II upregulates K(Ca)3.1 channels and stimulates cell proliferation in rat cardiac fibroblasts.

Wang LP, Wang Y, Zhao LM, Li GR, Deng XL.

Biochem Pharmacol. 2013 May 15;85(10):1486-94. doi: 10.1016/j.bcp.2013.02.032. Epub 2013 Mar 7.

PMID:
23500546
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16.

Association of PI3K-Akt signaling pathway with digitalis-induced hypertrophy of cardiac myocytes.

Liu L, Zhao X, Pierre SV, Askari A.

Am J Physiol Cell Physiol. 2007 Nov;293(5):C1489-97. Epub 2007 Aug 29.

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18.

Differential activation of mitogen-activated protein kinases in smooth muscle cells by angiotensin II: involvement of p22phox and reactive oxygen species.

Viedt C, Soto U, Krieger-Brauer HI, Fei J, Elsing C, Kübler W, Kreuzer J.

Arterioscler Thromb Vasc Biol. 2000 Apr;20(4):940-8.

20.

Mitogen-activated protein kinases inhibit the ROMK (Kir 1.1)-like small conductance K channels in the cortical collecting duct.

Babilonia E, Li D, Wang Z, Sun P, Lin DH, Jin Y, Wang WH.

J Am Soc Nephrol. 2006 Oct;17(10):2687-96. Epub 2006 Sep 13.

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