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Items: 1 to 20 of 107

1.

Selective disruption of ER{alpha} DNA-binding activity alters uterine responsiveness to estradiol.

Hewitt SC, O'Brien JE, Jameson JL, Kissling GE, Korach KS.

Mol Endocrinol. 2009 Dec;23(12):2111-6. doi: 10.1210/me.2009-0356.

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Estren behaves as a weak estrogen rather than a nongenomic selective activator in the mouse uterus.

Hewitt SC, Collins J, Grissom S, Hamilton K, Korach KS.

Endocrinology. 2006 May;147(5):2203-14.

PMID:
16469803
4.

Novel DNA motif binding activity observed in vivo with an estrogen receptor α mutant mouse.

Hewitt SC, Li L, Grimm SA, Winuthayanon W, Hamilton KJ, Pockette B, Rubel CA, Pedersen LC, Fargo D, Lanz RB, DeMayo FJ, Schütz G, Korach KS.

Mol Endocrinol. 2014 Jun;28(6):899-911. doi: 10.1210/me.2014-1051.

5.

Diarylheptanoid phytoestrogens isolated from the medicinal plant Curcuma comosa: biologic actions in vitro and in vivo indicate estrogen receptor-dependent mechanisms.

Winuthayanon W, Piyachaturawat P, Suksamrarn A, Ponglikitmongkol M, Arao Y, Hewitt SC, Korach KS.

Environ Health Perspect. 2009 Jul;117(7):1155-61. doi: 10.1289/ehp.0900613.

6.

Estrogen receptor-dependent genomic responses in the uterus mirror the biphasic physiological response to estrogen.

Hewitt SC, Deroo BJ, Hansen K, Collins J, Grissom S, Afshari CA, Korach KS.

Mol Endocrinol. 2003 Oct;17(10):2070-83.

PMID:
12893882
7.

Regulation of gene expression by 17β-estradiol in the arcuate nucleus of the mouse through ERE-dependent and ERE-independent mechanisms.

Yang JA, Mamounis KJ, Yasrebi A, Roepke TA.

Steroids. 2016 Mar;107:128-38. doi: 10.1016/j.steroids.2016.01.003.

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Skeletal effects of estrogen are mediated by opposing actions of classical and nonclassical estrogen receptor pathways.

Syed FA, Mödder UI, Fraser DG, Spelsberg TC, Rosen CJ, Krust A, Chambon P, Jameson JL, Khosla S.

J Bone Miner Res. 2005 Nov;20(11):1992-2001.

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ERE-independent ERalpha target genes differentially expressed in human breast tumors.

Glidewell-Kenney C, Weiss J, Lee EJ, Pillai S, Ishikawa T, Ariazi EA, Jameson JL.

Mol Cell Endocrinol. 2005 Dec 21;245(1-2):53-9.

PMID:
16298037
14.

Examination of ERα signaling pathways in bone of mutant mouse models reveals the importance of ERE-dependent signaling.

Chokalingam K, Roforth MM, Nicks KM, McGregor U, Fraser D, Khosla S, Monroe DG.

Endocrinology. 2012 Nov;153(11):5325-33. doi: 10.1210/en.2012-1721.

15.

A DNA binding mutation in estrogen receptor-α leads to suppression of Wnt signaling via β-catenin destabilization in osteoblasts.

Mödder UI, Rudnik V, Liu G, Khosla S, Monroe DG.

J Cell Biochem. 2012 Jul;113(7):2248-55. doi: 10.1002/jcb.24095.

16.

Genomic responses from the estrogen-responsive element-dependent signaling pathway mediated by estrogen receptor alpha are required to elicit cellular alterations.

Nott SL, Huang Y, Li X, Fluharty BR, Qiu X, Welshons WV, Yeh S, Muyan M.

J Biol Chem. 2009 May 29;284(22):15277-88. doi: 10.1074/jbc.M900365200.

17.

Global uterine genomics in vivo: microarray evaluation of the estrogen receptor alpha-growth factor cross-talk mechanism.

Hewitt SC, Collins J, Grissom S, Deroo B, Korach KS.

Mol Endocrinol. 2005 Mar;19(3):657-68.

PMID:
15528273
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Estrogen-induced proliferation of uterine epithelial cells is independent of estrogen receptor alpha binding to classical estrogen response elements.

O'Brien JE, Peterson TJ, Tong MH, Lee EJ, Pfaff LE, Hewitt SC, Korach KS, Weiss J, Jameson JL.

J Biol Chem. 2006 Sep 8;281(36):26683-92.

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