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Items: 1 to 20 of 95

1.

FOSB proteins in the orbitofrontal and dorsolateral prefrontal cortices of human alcoholics.

Watanabe H, Henriksson R, Ohnishi YN, Ohnishi YH, Harper C, Sheedy D, Garrick T, Nyberg F, Nestler EJ, Bakalkin G, Yakovleva T.

Addict Biol. 2009 Jul;14(3):294-7. doi: 10.1111/j.1369-1600.2009.00155.x.

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Region-specific induction of FosB/ΔFosB by voluntary alcohol intake: effects of naltrexone.

Li J, Cheng Y, Bian W, Liu X, Zhang C, Ye JH.

Alcohol Clin Exp Res. 2010 Oct;34(10):1742-50. doi: 10.1111/j.1530-0277.2010.01261.x.

4.

Induction of deltaFosB in reward-related brain structures after chronic stress.

Perrotti LI, Hadeishi Y, Ulery PG, Barrot M, Monteggia L, Duman RS, Nestler EJ.

J Neurosci. 2004 Nov 24;24(47):10594-602.

5.

Elevated synaptophysin I in the prefrontal cortex of human chronic alcoholics.

Henriksson R, Kuzmin A, Okvist A, Harper C, Sheedy D, Garrick T, Yakovleva T, Bakalkin G.

Synapse. 2008 Nov;62(11):829-33. doi: 10.1002/syn.20559.

6.

DeltaFosB induction in orbitofrontal cortex mediates tolerance to cocaine-induced cognitive dysfunction.

Winstanley CA, LaPlant Q, Theobald DE, Green TA, Bachtell RK, Perrotti LI, DiLeone RJ, Russo SJ, Garth WJ, Self DW, Nestler EJ.

J Neurosci. 2007 Sep 26;27(39):10497-507.

7.

Gene x disease interaction on orbitofrontal gray matter in cocaine addiction.

Alia-Klein N, Parvaz MA, Woicik PA, Konova AB, Maloney T, Shumay E, Wang R, Telang F, Biegon A, Wang GJ, Fowler JS, Tomasi D, Volkow ND, Goldstein RZ.

Arch Gen Psychiatry. 2011 Mar;68(3):283-94. doi: 10.1001/archgenpsychiatry.2011.10.

9.

Glial and glutamatergic markers in depression, alcoholism, and their comorbidity.

Miguel-Hidalgo JJ, Waltzer R, Whittom AA, Austin MC, Rajkowska G, Stockmeier CA.

J Affect Disord. 2010 Dec;127(1-3):230-40. doi: 10.1016/j.jad.2010.06.003.

10.

Induction of FosB/DeltaFosB in the brain stress system-related structures during morphine dependence and withdrawal.

Núñez C, Martín F, Földes A, Luisa Laorden M, Kovács KJ, Victoria Milanés M.

J Neurochem. 2010 Jul;114(2):475-87. doi: 10.1111/j.1471-4159.2010.06765.x.

11.

DeltaFosB induction in orbitofrontal cortex potentiates locomotor sensitization despite attenuating the cognitive dysfunction caused by cocaine.

Winstanley CA, Green TA, Theobald DE, Renthal W, LaPlant Q, DiLeone RJ, Chakravarty S, Nestler EJ.

Pharmacol Biochem Behav. 2009 Sep;93(3):278-84. doi: 10.1016/j.pbb.2008.12.007.

13.

Withdrawal induces distinct patterns of FosB/∆FosB expression in outbred Swiss mice classified as susceptible and resistant to ethanol-induced locomotor sensitization.

De Pauli RF, Coelhoso CC, Tesone-Coelho C, Linardi A, Mello LE, Silveira DX, Santos JG Jr.

Pharmacol Biochem Behav. 2014 Feb;117:70-8. doi: 10.1016/j.pbb.2013.12.007.

14.

Differences in basal and morphine-induced FosB/DeltaFosB and pCREB immunoreactivities in dopaminergic brain regions of alcohol-preferring AA and alcohol-avoiding ANA rats.

Kaste K, Kivinummi T, Piepponen TP, Kiianmaa K, Ahtee L.

Pharmacol Biochem Behav. 2009 Jun;92(4):655-62. doi: 10.1016/j.pbb.2009.03.004.

PMID:
19324071
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Opiate sensitization induces FosB/ΔFosB expression in prefrontal cortical, striatal and amygdala brain regions.

Kaplan GB, Leite-Morris KA, Fan W, Young AJ, Guy MD.

PLoS One. 2011;6(8):e23574. doi: 10.1371/journal.pone.0023574.

18.

Increased impulsivity during withdrawal from cocaine self-administration: role for DeltaFosB in the orbitofrontal cortex.

Winstanley CA, Bachtell RK, Theobald DE, Laali S, Green TA, Kumar A, Chakravarty S, Self DW, Nestler EJ.

Cereb Cortex. 2009 Feb;19(2):435-44. doi: 10.1093/cercor/bhn094.

19.

Effects of chronic intermittent ethanol exposure on orbitofrontal and medial prefrontal cortex-dependent behaviors in mice.

Badanich KA, Becker HC, Woodward JJ.

Behav Neurosci. 2011 Dec;125(6):879-91. doi: 10.1037/a0025922.

20.

Medial orbitofrontal cortex gray matter is reduced in abstinent substance-dependent individuals.

Tanabe J, Tregellas JR, Dalwani M, Thompson L, Owens E, Crowley T, Banich M.

Biol Psychiatry. 2009 Jan 15;65(2):160-4. doi: 10.1016/j.biopsych.2008.07.030.

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