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Items: 1 to 20 of 436

1.

Increased expression and intramitochondrial translocation of cyclophilin-D associates with increased vulnerability of the permeability transition pore to stress-induced opening during compensated ventricular hypertrophy.

Matas J, Young NT, Bourcier-Lucas C, Ascah A, Marcil M, Deschepper CF, Burelle Y.

J Mol Cell Cardiol. 2009 Mar;46(3):420-30. doi: 10.1016/j.yjmcc.2008.10.020. Epub 2008 Nov 6.

PMID:
19094991
2.

Muscle denervation promotes opening of the permeability transition pore and increases the expression of cyclophilin D.

Csukly K, Ascah A, Matas J, Gardiner PF, Fontaine E, Burelle Y.

J Physiol. 2006 Jul 1;574(Pt 1):319-27. Epub 2006 May 4.

3.
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Mitochondrial Ca2+ uptake during simulated ischemia does not affect permeability transition pore opening upon simulated reperfusion.

Ruiz-Meana M, Garcia-Dorado D, Miró-Casas E, Abellán A, Soler-Soler J.

Cardiovasc Res. 2006 Sep 1;71(4):715-24. Epub 2006 Jun 21.

PMID:
16860295
5.

Stress-induced opening of the permeability transition pore in the dystrophin-deficient heart is attenuated by acute treatment with sildenafil.

Ascah A, Khairallah M, Daussin F, Bourcier-Lucas C, Godin R, Allen BG, Petrof BJ, Des Rosiers C, Burelle Y.

Am J Physiol Heart Circ Physiol. 2011 Jan;300(1):H144-53. doi: 10.1152/ajpheart.00522.2010. Epub 2010 Oct 22.

6.

Mitochondria and ischemia-reperfusion injury of the heart: fixing a hole.

Di Lisa F, Bernardi P.

Cardiovasc Res. 2006 May 1;70(2):191-9. Epub 2006 Feb 23. Review.

PMID:
16497286
7.

Resistance to Ca2+-induced opening of the permeability transition pore differs in mitochondria from glycolytic and oxidative muscles.

Picard M, Csukly K, Robillard ME, Godin R, Ascah A, Bourcier-Lucas C, Burelle Y.

Am J Physiol Regul Integr Comp Physiol. 2008 Aug;295(2):R659-68. doi: 10.1152/ajpregu.90357.2008. Epub 2008 May 21.

8.

Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

Basso E, Fante L, Fowlkes J, Petronilli V, Forte MA, Bernardi P.

J Biol Chem. 2005 May 13;280(19):18558-61. Epub 2005 Mar 25.

9.

Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury.

Halestrap AP, Connern CP, Griffiths EJ, Kerr PM.

Mol Cell Biochem. 1997 Sep;174(1-2):167-72. Review.

PMID:
9309682
10.
11.

Compensated volume overload increases the vulnerability of heart mitochondria without affecting their functions in the absence of stress.

Marcil M, Ascah A, Matas J, Bélanger S, Deschepper CF, Burelle Y.

J Mol Cell Cardiol. 2006 Dec;41(6):998-1009. Epub 2006 Oct 24.

PMID:
17064727
12.

Vanadium compounds induced mitochondria permeability transition pore (PTP) opening related to oxidative stress.

Zhao Y, Ye L, Liu H, Xia Q, Zhang Y, Yang X, Wang K.

J Inorg Biochem. 2010 Apr;104(4):371-8. doi: 10.1016/j.jinorgbio.2009.11.007. Epub 2009 Nov 27.

PMID:
20015552
13.

Genetic dissection of the permeability transition pore.

Forte M, Bernardi P.

J Bioenerg Biomembr. 2005 Jun;37(3):121-8. Review.

PMID:
16167169
14.

Cyclosporine A normalizes mitochondrial coupling, reactive oxygen species production, and inflammation and partially restores skeletal muscle maximal oxidative capacity in experimental aortic cross-clamping.

Pottecher J, Guillot M, Belaidi E, Charles AL, Lejay A, Gharib A, Diemunsch P, Geny B.

J Vasc Surg. 2013 Apr;57(4):1100-1108.e2. doi: 10.1016/j.jvs.2012.09.020. Epub 2013 Jan 18.

16.

Opening of mitochondrial permeability transition pore induces hypercontracture in Ca2+ overloaded cardiac myocytes.

Ruiz-Meana M, Abellán A, Miró-Casas E, Garcia-Dorado D.

Basic Res Cardiol. 2007 Nov;102(6):542-52. Epub 2007 Sep 24.

PMID:
17891523
17.

Mitochondrial defects and dysfunction in calcium regulation in glaucomatous trabecular meshwork cells.

He Y, Ge J, Tombran-Tink J.

Invest Ophthalmol Vis Sci. 2008 Nov;49(11):4912-22. doi: 10.1167/iovs.08-2192. Epub 2008 Jul 9.

PMID:
18614807
18.
19.

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death.

Baines CP, Kaiser RA, Purcell NH, Blair NS, Osinska H, Hambleton MA, Brunskill EW, Sayen MR, Gottlieb RA, Dorn GW, Robbins J, Molkentin JD.

Nature. 2005 Mar 31;434(7033):658-62.

PMID:
15800627
20.

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