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Items: 1 to 20 of 121

1.

Simultaneous absence of surfactant proteins A and D increases lung inflammation and injury after allogeneic HSCT in mice.

Gram K, Yang S, Steiner M, Somani A, Hawgood S, Blazar BR, Panoskaltsis-Mortari A, Haddad IY.

Am J Physiol Lung Cell Mol Physiol. 2009 Feb;296(2):L167-75. doi: 10.1152/ajplung.90253.2008. Epub 2008 Nov 7.

2.

Surfactant protein A decreases lung injury and mortality after murine marrow transplantation.

Yang S, Milla C, Panoskaltsis-Mortari A, Hawgood S, Blazar BR, Haddad IY.

Am J Respir Cell Mol Biol. 2002 Sep;27(3):297-305.

PMID:
12204891
3.

Surfactant protein A is a required mediator of keratinocyte growth factor after experimental marrow transplantation.

Haddad IY, Milla C, Yang S, Panoskaltsis-Mortari A, Hawgood S, Lacey DL, Blazar BR.

Am J Physiol Lung Cell Mol Physiol. 2003 Sep;285(3):L602-10. Epub 2003 May 9.

4.

Keratinocyte growth factor facilitates alloengraftment and ameliorates graft-versus-host disease in mice by a mechanism independent of repair of conditioning-induced tissue injury.

Panoskaltsis-Mortari A, Taylor PA, Rubin JS, Uren A, Welniak LA, Murphy WJ, Farrell CL, Lacey DL, Blazar BR.

Blood. 2000 Dec 15;96(13):4350-6.

5.

Tumor necrosis factor-alpha decreases surfactant protein B mRNA in murine lung.

Pryhuber GS, Bachurski C, Hirsch R, Bacon A, Whitsett JA.

Am J Physiol. 1996 May;270(5 Pt 1):L714-21.

PMID:
8967504
7.

Neutralization of tumor necrosis factor-alpha action delays but does not prevent lung injury induced by alloreactive T helper 1 cells.

Clark JG, Mandac JB, Dixon AE, Martin PJ, Hackman RC, Madtes DK.

Transplantation. 2000 Jul 15;70(1):39-43.

PMID:
10919572
8.

Increased surfactant protein a and d expression in acute ovalbumin-induced allergic airway inflammation in brown norway rats.

Schmiedl A, L├╝hrmann A, Pabst R, Koslowski R.

Int Arch Allergy Immunol. 2009;148(2):118-26. doi: 10.1159/000155742. Epub 2008 Sep 19.

PMID:
18802356
9.
10.

A role for tumor necrosis factor-alpha-mediated endothelial apoptosis in the development of experimental idiopathic pneumonia syndrome.

Gerbitz A, Nickoloff BJ, Olkiewicz K, Willmarth NE, Hildebrandt G, Liu C, Kobzik L, Eissner G, Holler E, Ferrara JL, Cooke KR.

Transplantation. 2004 Aug 27;78(4):494-502.

PMID:
15446306
14.

Post-BMT lung injury occurs independently of the expression of CCL2 or its receptor, CCR2, on host cells.

Panoskaltsis-Mortari A, Hermanson JR, Taras E, Wangensteen OD, Charo IF, Rollins BJ, Blazar BR.

Am J Physiol Lung Cell Mol Physiol. 2004 Feb;286(2):L284-92. Epub 2003 Oct 3.

15.
16.

Human surfactant protein a suppresses T cell-dependent inflammation and attenuates the manifestations of idiopathic pneumonia syndrome in mice.

Yang S, Milla C, Panoskaltsis-Mortari A, Ingbar DH, Blazar BR, Haddad IY.

Am J Respir Cell Mol Biol. 2001 May;24(5):527-36.

PMID:
11350821
17.

Blockade of CXCR3 receptor:ligand interactions reduces leukocyte recruitment to the lung and the severity of experimental idiopathic pneumonia syndrome.

Hildebrandt GC, Corrion LA, Olkiewicz KM, Lu B, Lowler K, Duffner UA, Moore BB, Kuziel WA, Liu C, Cooke KR.

J Immunol. 2004 Aug 1;173(3):2050-9.

18.

Cyclophosphamide prevents systemic keratinocyte growth factor-induced up-regulation of surfactant protein A after allogeneic transplant in mice.

Yang S, Panoskaltsis-Mortari A, Ingbar DH, Matalon S, Zhu S, Resnik ER, Farrell CL, Lacey DL, Blazar BR, Haddad IY.

Am J Respir Crit Care Med. 2000 Nov;162(5):1884-90.

PMID:
11069830
19.

GM-CSF mediates alveolar macrophage proliferation and type II cell hypertrophy in SP-D gene-targeted mice.

Hawgood S, Akiyama J, Brown C, Allen L, Li G, Poulain FR.

Am J Physiol Lung Cell Mol Physiol. 2001 Jun;280(6):L1148-56.

20.

Exuberant inflammation in nicotinamide adenine dinucleotide phosphate-oxidase-deficient mice after allogeneic marrow transplantation.

Yang S, Panoskaltsis-Mortari A, Shukla M, Blazar BR, Haddad IY.

J Immunol. 2002 Jun 1;168(11):5840-7.

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