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Items: 1 to 20 of 149


Dscam guides embryonic axons by Netrin-dependent and -independent functions.

Andrews GL, Tanglao S, Farmer WT, Morin S, Brotman S, Berberoglu MA, Price H, Fernandez GC, Mastick GS, Charron F, Kidd T.

Development. 2008 Dec;135(23):3839-48. doi: 10.1242/dev.023739. Epub 2008 Oct 23.


Mushroom body defect is required in parallel to Netrin for midline axon guidance in Drosophila.

Cate MS, Gajendra S, Alsbury S, Raabe T, Tear G, Mitchell KJ.

Development. 2016 Mar 15;143(6):972-7. doi: 10.1242/dev.129684. Epub 2016 Feb 18.


The Abelson tyrosine kinase, the Trio GEF and Enabled interact with the Netrin receptor Frazzled in Drosophila.

Forsthoefel DJ, Liebl EC, Kolodziej PA, Seeger MA.

Development. 2005 Apr;132(8):1983-94.


DSCAM is a netrin receptor that collaborates with DCC in mediating turning responses to netrin-1.

Ly A, Nikolaev A, Suresh G, Zheng Y, Tessier-Lavigne M, Stein E.

Cell. 2008 Jun 27;133(7):1241-54. doi: 10.1016/j.cell.2008.05.030.


Flamingo, a seven-pass transmembrane cadherin, cooperates with Netrin/Frazzled in Drosophila midline guidance.

Organisti C, Hein I, Grunwald Kadow IC, Suzuki T.

Genes Cells. 2015 Jan;20(1):50-67. doi: 10.1111/gtc.12202. Epub 2014 Nov 30.


In the absence of frazzled over-expression of Abelson tyrosine kinase disrupts commissure formation and causes axons to leave the embryonic CNS.

Dorsten JN, Varughese BE, Karmo S, Seeger MA, VanBerkum MF.

PLoS One. 2010 Mar 23;5(3):e9822. doi: 10.1371/journal.pone.0009822.


Coordinated interaction of Down syndrome cell adhesion molecule and deleted in colorectal cancer with dynamic TUBB3 mediates Netrin-1-induced axon branching.

Huang H, Shao Q, Qu C, Yang T, Dwyer T, Liu G.

Neuroscience. 2015 May 7;293:109-22. doi: 10.1016/j.neuroscience.2015.02.042. Epub 2015 Mar 6.


Sema-1a Reverse Signaling Promotes Midline Crossing in Response to Secreted Semaphorins.

Hernandez-Fleming M, Rohrbach EW, Bashaw GJ.

Cell Rep. 2017 Jan 3;18(1):174-184. doi: 10.1016/j.celrep.2016.12.027.


DSCAM functions as a netrin receptor in commissural axon pathfinding.

Liu G, Li W, Wang L, Kar A, Guan KL, Rao Y, Wu JY.

Proc Natl Acad Sci U S A. 2009 Feb 24;106(8):2951-6. doi: 10.1073/pnas.0811083106. Epub 2009 Feb 5.


Drosophila melanogaster Hedgehog cooperates with Frazzled to guide axons through a non-canonical signalling pathway.

Ricolo D, Butí E, Araújo SJ.

Mech Dev. 2015 Aug;137:11-22. doi: 10.1016/j.mod.2015.04.003. Epub 2015 Apr 30.


Robo and Frazzled/DCC mediate dendritic guidance at the CNS midline.

Furrer MP, Kim S, Wolf B, Chiba A.

Nat Neurosci. 2003 Mar;6(3):223-30.


A frazzled/DCC-dependent transcriptional switch regulates midline axon guidance.

Yang L, Garbe DS, Bashaw GJ.

Science. 2009 May 15;324(5929):944-7. doi: 10.1126/science.1171320. Epub 2009 Mar 26.


The Drosophila immunoglobulin gene turtle encodes guidance molecules involved in axon pathfinding.

Al-Anzi B, Wyman RJ.

Neural Dev. 2009 Aug 17;4:31. doi: 10.1186/1749-8104-4-31.


Src inhibits midline axon crossing independent of Frazzled/Deleted in Colorectal Carcinoma (DCC) receptor tyrosine phosphorylation.

O'Donnell MP, Bashaw GJ.

J Neurosci. 2013 Jan 2;33(1):305-14. doi: 10.1523/JNEUROSCI.2756-12.2013.


Down syndrome cell adhesion molecule (DSCAM) associates with uncoordinated-5C (UNC5C) in netrin-1-mediated growth cone collapse.

Purohit AA, Li W, Qu C, Dwyer T, Shao Q, Guan KL, Liu G.

J Biol Chem. 2012 Aug 3;287(32):27126-38. doi: 10.1074/jbc.M112.340174. Epub 2012 Jun 8.


The Drosophila Netrin receptor Frazzled guides axons by controlling Netrin distribution.

Hiramoto M, Hiromi Y, Giniger E, Hotta Y.

Nature. 2000 Aug 24;406(6798):886-9.


Frazzled cytoplasmic P-motifs are differentially required for axon pathway formation in the Drosophila embryonic CNS.

Dorsten JN, VanBerkum MF.

Int J Dev Neurosci. 2008 Nov;26(7):753-61. doi: 10.1016/j.ijdevneu.2008.07.004. Epub 2008 Jul 11.


Netrin1-DCC-Mediated Attraction Guides Post-Crossing Commissural Axons in the Hindbrain.

Shoja-Taheri F, DeMarco A, Mastick GS.

J Neurosci. 2015 Aug 19;35(33):11707-18. doi: 10.1523/JNEUROSCI.0613-15.2015.

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