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Items: 1 to 20 of 308

1.

Increased expression of golli myelin basic proteins enhances calcium influx into oligodendroglial cells.

Paez PM, Spreuer V, Handley V, Feng JM, Campagnoni C, Campagnoni AT.

J Neurosci. 2007 Nov 14;27(46):12690-9.

2.

Region-specific myelin pathology in mice lacking the golli products of the myelin basic protein gene.

Jacobs EC, Pribyl TM, Feng JM, Kampf K, Spreur V, Campagnoni C, Colwell CS, Reyes SD, Martin M, Handley V, Hiltner TD, Readhead C, Jacobs RE, Messing A, Fisher RS, Campagnoni AT.

J Neurosci. 2005 Jul 27;25(30):7004-13. Erratum in: J Neurosci. 2005 Dec 14;25(50): table of contents. Feng, Ji-Ming [added]; Spreur, Vilma [added].

3.

Classical 18.5-and 21.5-kDa isoforms of myelin basic protein inhibit calcium influx into oligodendroglial cells, in contrast to golli isoforms.

Smith GS, Paez PM, Spreuer V, Campagnoni CW, Boggs JM, Campagnoni AT, Harauz G.

J Neurosci Res. 2011 Apr;89(4):467-80. doi: 10.1002/jnr.22570. Epub 2011 Jan 13.

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Golli protein negatively regulates store depletion-induced calcium influx in T cells.

Feng JM, Hu YK, Xie LH, Colwell CS, Shao XM, Sun XP, Chen B, Tang H, Campagnoni AT.

Immunity. 2006 Jun;24(6):717-27.

6.

Golli myelin basic proteins regulate oligodendroglial progenitor cell migration through voltage-gated Ca2+ influx.

Paez PM, Fulton DJ, Spreuer V, Handley V, Campagnoni CW, Macklin WB, Colwell C, Campagnoni AT.

J Neurosci. 2009 May 20;29(20):6663-76. doi: 10.1523/JNEUROSCI.5806-08.2009.

7.

Regulation of L-type Ca++ currents and process morphology in white matter oligodendrocyte precursor cells by golli-myelin proteins.

Fulton D, Paez PM, Fisher R, Handley V, Colwell CS, Campagnoni AT.

Glia. 2010 Aug 15;58(11):1292-303. doi: 10.1002/glia.21008.

PMID:
20607717
8.

Identification of a protein that interacts with the golli-myelin basic protein and with nuclear LIM interactor in the nervous system.

Fernandes AO, Campagnoni CW, Kampf K, Feng JM, Handley VW, Schonmann V, Bongarzone ER, Reyes S, Campagnoni AT.

J Neurosci Res. 2004 Feb 15;75(4):461-71.

PMID:
14743429
9.

Modulation of canonical transient receptor potential channel 1 in the proliferation of oligodendrocyte precursor cells by the golli products of the myelin basic protein gene.

Paez PM, Fulton D, Spreuer V, Handley V, Campagnoni AT.

J Neurosci. 2011 Mar 9;31(10):3625-37. doi: 10.1523/JNEUROSCI.4424-10.2011.

10.

Proline substitutions and threonine pseudophosphorylation of the SH3 ligand of 18.5-kDa myelin basic protein decrease its affinity for the Fyn-SH3 domain and alter process development and protein localization in oligodendrocytes.

Smith GS, De Avila M, Paez PM, Spreuer V, Wills MK, Jones N, Boggs JM, Harauz G.

J Neurosci Res. 2012 Jan;90(1):28-47. doi: 10.1002/jnr.22733. Epub 2011 Sep 1.

11.

Regulation of store-operated and voltage-operated Ca2+ channels in the proliferation and death of oligodendrocyte precursor cells by golli proteins.

Paez PM, Fulton DJ, Spreuer V, Handley V, Campagnoni CW, Campagnoni AT.

ASN Neuro. 2009 Apr 14;1(1). pii: e00003. doi: 10.1042/AN20090003.

13.

Expression and regulation of golli products of myelin basic protein gene during in vitro development of oligodendrocytes.

Givogri MI, Bongarzone ER, Schonmann V, Campagnoni AT.

J Neurosci Res. 2001 Nov 15;66(4):679-90.

PMID:
11746388
15.

Identification of Golli and myelin basic proteins in human brain during early development.

Tosic M, Rakic S, Matthieu J-, Zecevic N.

Glia. 2002 Mar 1;37(3):219-28.

PMID:
11857680
16.

Myelin basic protein gene expression in neurons: developmental and regional changes in protein targeting within neuronal nuclei, cell bodies, and processes.

Landry CF, Ellison JA, Pribyl TM, Campagnoni C, Kampf K, Campagnoni AT.

J Neurosci. 1996 Apr 15;16(8):2452-62.

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Subcellular heterogeneity of voltage-gated Ca2+ channels in cells of the oligodendrocyte lineage.

Kirischuk S, Scherer J, Möller T, Verkhratsky A, Kettenmann H.

Glia. 1995 Jan;13(1):1-12.

PMID:
7751051
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