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Items: 1 to 20 of 347

1.

Yeast Rad5 protein required for postreplication repair has a DNA helicase activity specific for replication fork regression.

Blastyák A, Pintér L, Unk I, Prakash L, Prakash S, Haracska L.

Mol Cell. 2007 Oct 12;28(1):167-75.

2.

Requirement of RAD5 and MMS2 for postreplication repair of UV-damaged DNA in Saccharomyces cerevisiae.

Torres-Ramos CA, Prakash S, Prakash L.

Mol Cell Biol. 2002 Apr;22(7):2419-26.

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Requirement of Rad5 for DNA polymerase zeta-dependent translesion synthesis in Saccharomyces cerevisiae.

Pagès V, Bresson A, Acharya N, Prakash S, Fuchs RP, Prakash L.

Genetics. 2008 Sep;180(1):73-82. doi: 10.1534/genetics.108.091066.

6.

The Rad5 helicase activity is dispensable for error-free DNA post-replication repair.

Ball LG, Xu X, Blackwell S, Hanna MD, Lambrecht AD, Xiao W.

DNA Repair (Amst). 2014 Apr;16:74-83. doi: 10.1016/j.dnarep.2014.02.016.

PMID:
24674630
7.

RAD6-RAD18-RAD5-pathway-dependent tolerance to chronic low-dose ultraviolet light.

Hishida T, Kubota Y, Carr AM, Iwasaki H.

Nature. 2009 Jan 29;457(7229):612-5. doi: 10.1038/nature07580.

PMID:
19079240
8.

Requirement of replication checkpoint protein kinases Mec1/Rad53 for postreplication repair in yeast.

Gangavarapu V, Santa Maria SR, Prakash S, Prakash L.

MBio. 2011 May 17;2(3):e00079-11. doi: 10.1128/mBio.00079-11.

9.

Reversal of fortune: Rad5 to the rescue.

Klein HL.

Mol Cell. 2007 Oct 26;28(2):181-3.

10.

Role of yeast Rad5 and its human orthologs, HLTF and SHPRH in DNA damage tolerance.

Unk I, Hajdú I, Blastyák A, Haracska L.

DNA Repair (Amst). 2010 Mar 2;9(3):257-67. doi: 10.1016/j.dnarep.2009.12.013. Review.

PMID:
20096653
11.

Rad5 plays a major role in the cellular response to DNA damage during chromosome replication.

Ortiz-Bazán MÁ, Gallo-Fernández M, Saugar I, Jiménez-Martín A, Vázquez MV, Tercero JA.

Cell Rep. 2014 Oct 23;9(2):460-8. doi: 10.1016/j.celrep.2014.09.005.

12.

Opposing effects of ubiquitin conjugation and SUMO modification of PCNA on replicational bypass of DNA lesions in Saccharomyces cerevisiae.

Haracska L, Torres-Ramos CA, Johnson RE, Prakash S, Prakash L.

Mol Cell Biol. 2004 May;24(10):4267-74.

13.

Strand invasion by HLTF as a mechanism for template switch in fork rescue.

Burkovics P, Sebesta M, Balogh D, Haracska L, Krejci L.

Nucleic Acids Res. 2014 Feb;42(3):1711-20. doi: 10.1093/nar/gkt1040.

14.

A non-catalytic function of Rev1 in translesion DNA synthesis and mutagenesis is mediated by its stable interaction with Rad5.

Kuang L, Kou H, Xie Z, Zhou Y, Feng X, Wang L, Wang Z.

DNA Repair (Amst). 2013 Jan 1;12(1):27-37. doi: 10.1016/j.dnarep.2012.10.003.

PMID:
23142547
15.

Post-replication repair suppresses duplication-mediated genome instability.

Putnam CD, Hayes TK, Kolodner RD.

PLoS Genet. 2010 May 6;6(5):e1000933. doi: 10.1371/journal.pgen.1000933.

16.

Error-free DNA-damage tolerance in Saccharomyces cerevisiae.

Xu X, Blackwell S, Lin A, Li F, Qin Z, Xiao W.

Mutat Res Rev Mutat Res. 2015 Apr-Jun;764:43-50. doi: 10.1016/j.mrrev.2015.02.001. Review.

PMID:
26041265
17.

The Bloom's syndrome helicase can promote the regression of a model replication fork.

Ralf C, Hickson ID, Wu L.

J Biol Chem. 2006 Aug 11;281(32):22839-46.

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