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2.

Latency-associated transcript (LAT) exon 1 controls herpes simplex virus species-specific phenotypes: reactivation in the guinea pig genital model and neuron subtype-specific latent expression of LAT.

Bertke AS, Patel A, Imai Y, Apakupakul K, Margolis TP, Krause PR.

J Virol. 2009 Oct;83(19):10007-15. doi: 10.1128/JVI.00559-09. Epub 2009 Jul 29.

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Neuronal control of herpes simplex virus latency.

Tenser RB, Edris WA, Hay KA.

Virology. 1993 Aug;195(2):337-47.

PMID:
8393231
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11.

The latency-associated transcript gene of herpes simplex virus type 1 (HSV-1) is required for efficient in vivo spontaneous reactivation of HSV-1 from latency.

Perng GC, Dunkel EC, Geary PA, Slanina SM, Ghiasi H, Kaiwar R, Nesburn AB, Wechsler SL.

J Virol. 1994 Dec;68(12):8045-55.

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A gene capable of blocking apoptosis can substitute for the herpes simplex virus type 1 latency-associated transcript gene and restore wild-type reactivation levels.

Perng GC, Maguen B, Jin L, Mott KR, Osorio N, Slanina SM, Yukht A, Ghiasi H, Nesburn AB, Inman M, Henderson G, Jones C, Wechsler SL.

J Virol. 2002 Feb;76(3):1224-35.

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20.

Repression of the HSV-1 latency-associated transcript (LAT) promoter by the early growth response (EGR) proteins: involvement of a binding site immediately downstream of the TATA box.

Tatarowicz WA, Martin CE, Pekosz AS, Madden SL, Rauscher FJ 3rd, Chiang SY, Beerman TA, Fraser NW.

J Neurovirol. 1997 Jun;3(3):212-24.

PMID:
9200069

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