Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 148

1.

ALS: a disease of motor neurons and their nonneuronal neighbors.

Boillée S, Vande Velde C, Cleveland DW.

Neuron. 2006 Oct 5;52(1):39-59. Review.

2.

Amyotrophic lateral sclerosis: from current developments in the laboratory to clinical implications.

Cozzolino M, Ferri A, Carrì MT.

Antioxid Redox Signal. 2008 Mar;10(3):405-43. doi: 10.1089/ars.2007.1760. Review.

PMID:
18370853
3.

Current hypotheses for the underlying biology of amyotrophic lateral sclerosis.

Rothstein JD.

Ann Neurol. 2009 Jan;65 Suppl 1:S3-9. doi: 10.1002/ana.21543. Review.

PMID:
19191304
5.

Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice.

Clement AM, Nguyen MD, Roberts EA, Garcia ML, Boillée S, Rule M, McMahon AP, Doucette W, Siwek D, Ferrante RJ, Brown RH Jr, Julien JP, Goldstein LS, Cleveland DW.

Science. 2003 Oct 3;302(5642):113-7. Erratum in: Science. 2003 Oct 24;302(5645):568.

6.

Familial amyotrophic lateral sclerosis-linked SOD1 mutants perturb fast axonal transport to reduce axonal mitochondria content.

De Vos KJ, Chapman AL, Tennant ME, Manser C, Tudor EL, Lau KF, Brownlees J, Ackerley S, Shaw PJ, McLoughlin DM, Shaw CE, Leigh PN, Miller CC, Grierson AJ.

Hum Mol Genet. 2007 Nov 15;16(22):2720-8. Epub 2007 Aug 28.

7.

Interaction between familial amyotrophic lateral sclerosis (ALS)-linked SOD1 mutants and the dynein complex.

Zhang F, Ström AL, Fukada K, Lee S, Hayward LJ, Zhu H.

J Biol Chem. 2007 Jun 1;282(22):16691-9. Epub 2007 Apr 2.

8.

Mitochondrial dysfunction and its role in motor neuron degeneration in ALS.

Manfredi G, Xu Z.

Mitochondrion. 2005 Apr;5(2):77-87. Review.

PMID:
16050975
9.

Deficits in axonal transport precede ALS symptoms in vivo.

Bilsland LG, Sahai E, Kelly G, Golding M, Greensmith L, Schiavo G.

Proc Natl Acad Sci U S A. 2010 Nov 23;107(47):20523-8. doi: 10.1073/pnas.1006869107. Epub 2010 Nov 8.

10.
11.

Transgenics, toxicity and therapeutics in rodent models of mutant SOD1-mediated familial ALS.

Turner BJ, Talbot K.

Prog Neurobiol. 2008 May;85(1):94-134. doi: 10.1016/j.pneurobio.2008.01.001. Epub 2008 Jan 16. Review.

PMID:
18282652
12.

Mitochondria-targeted catalase reverts the neurotoxicity of hSOD1G⁹³A astrocytes without extending the survival of ALS-linked mutant hSOD1 mice.

Pehar M, Beeson G, Beeson CC, Johnson JA, Vargas MR.

PLoS One. 2014 Jul 23;9(7):e103438. doi: 10.1371/journal.pone.0103438. eCollection 2014.

13.

SOD1 in neurotoxicity and its controversial roles in SOD1 mutation-negative ALS.

Hayashi Y, Homma K, Ichijo H.

Adv Biol Regul. 2016 Jan;60:95-104. doi: 10.1016/j.jbior.2015.10.006. Epub 2015 Oct 31. Review.

PMID:
26563614
14.

Extracellular wildtype and mutant SOD1 induces ER-Golgi pathology characteristic of amyotrophic lateral sclerosis in neuronal cells.

Sundaramoorthy V, Walker AK, Yerbury J, Soo KY, Farg MA, Hoang V, Zeineddine R, Spencer D, Atkin JD.

Cell Mol Life Sci. 2013 Nov;70(21):4181-95. doi: 10.1007/s00018-013-1385-2. Epub 2013 Jun 14.

PMID:
23765103
15.

Therapeutic AAV9-mediated suppression of mutant SOD1 slows disease progression and extends survival in models of inherited ALS.

Foust KD, Salazar DL, Likhite S, Ferraiuolo L, Ditsworth D, Ilieva H, Meyer K, Schmelzer L, Braun L, Cleveland DW, Kaspar BK.

Mol Ther. 2013 Dec;21(12):2148-59. doi: 10.1038/mt.2013.211. Epub 2013 Sep 6.

18.

A drosophila model for amyotrophic lateral sclerosis reveals motor neuron damage by human SOD1.

Watson MR, Lagow RD, Xu K, Zhang B, Bonini NM.

J Biol Chem. 2008 Sep 5;283(36):24972-81. doi: 10.1074/jbc.M804817200. Epub 2008 Jul 2.

19.

Mutant SOD1 in cell types other than motor neurons and oligodendrocytes accelerates onset of disease in ALS mice.

Yamanaka K, Boillee S, Roberts EA, Garcia ML, McAlonis-Downes M, Mikse OR, Cleveland DW, Goldstein LS.

Proc Natl Acad Sci U S A. 2008 May 27;105(21):7594-9. doi: 10.1073/pnas.0802556105. Epub 2008 May 20.

20.

Mitochondrial dysfunction in familial amyotrophic lateral sclerosis.

Faes L, Callewaert G.

J Bioenerg Biomembr. 2011 Dec;43(6):587-92. doi: 10.1007/s10863-011-9393-0. Review.

PMID:
22072073

Supplemental Content

Support Center