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Items: 1 to 20 of 115

1.

Assessing the link between BACH1 and BRCA1 in the FA pathway.

Cantor SB, Andreassen PR.

Cell Cycle. 2006 Jan;5(2):164-7. Epub 2006 Jan 16. Review.

PMID:
16357529
2.
3.

BRCA1-mediated repression of mutagenic end-joining of DNA double-strand breaks requires complex formation with BACH1.

Dohrn L, Salles D, Siehler SY, Kaufmann J, Wiesmüller L.

Biochem J. 2012 Feb 1;441(3):919-26. doi: 10.1042/BJ20110314.

PMID:
22032289
4.

The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells.

Peng M, Litman R, Xie J, Sharma S, Brosh RM Jr, Cantor SB.

EMBO J. 2007 Jul 11;26(13):3238-49. Epub 2007 Jun 21.

5.

FANCM-FAAP24 and FANCJ: FA proteins that metabolize DNA.

Ali AM, Singh TR, Meetei AR.

Mutat Res. 2009 Jul 31;668(1-2):20-6. doi: 10.1016/j.mrfmmm.2009.04.002. Epub 2009 Apr 18. Review.

6.

Activation of BRCA1/BRCA2-associated helicase BACH1 is required for timely progression through S phase.

Kumaraswamy E, Shiekhattar R.

Mol Cell Biol. 2007 Oct;27(19):6733-41. Epub 2007 Jul 30.

7.

Hereditary breast cancer and the BRCA1-associated FANCJ/BACH1/BRIP1.

Cantor SB, Guillemette S.

Future Oncol. 2011 Feb;7(2):253-61. doi: 10.2217/fon.10.191. Review.

8.
9.

FANCD2, FANCJ and BRCA2 cooperate to promote replication fork recovery independently of the Fanconi Anemia core complex.

Raghunandan M, Chaudhury I, Kelich SL, Hanenberg H, Sobeck A.

Cell Cycle. 2015;14(3):342-53. doi: 10.4161/15384101.2014.987614.

10.

BACH1 is a DNA repair protein supporting BRCA1 damage response.

Peng M, Litman R, Jin Z, Fong G, Cantor SB.

Oncogene. 2006 Apr 6;25(15):2245-53.

PMID:
16462773
11.

Assessing the link between BACH1/FANCJ and MLH1 in DNA crosslink repair.

Cantor SB, Xie J.

Environ Mol Mutagen. 2010 Jul;51(6):500-7. doi: 10.1002/em.20568. Review.

PMID:
20658644
12.

Mutational analysis of the breast cancer susceptibility gene BRIP1 /BACH1/FANCJ in high-risk non-BRCA1/BRCA2 breast cancer families.

Guénard F, Labrie Y, Ouellette G, Joly Beauparlant C, Simard J, Durocher F; INHERIT BRCAs.

J Hum Genet. 2008;53(7):579-91. doi: 10.1007/s10038-008-0285-z. Epub 2008 Apr 15.

PMID:
18414782
13.

BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ.

Litman R, Peng M, Jin Z, Zhang F, Zhang J, Powell S, Andreassen PR, Cantor SB.

Cancer Cell. 2005 Sep;8(3):255-65.

14.

Crosstalk between BRCA-Fanconi anemia and mismatch repair pathways prevents MSH2-dependent aberrant DNA damage responses.

Peng M, Xie J, Ucher A, Stavnezer J, Cantor SB.

EMBO J. 2014 Aug 1;33(15):1698-712. doi: 10.15252/embj.201387530. Epub 2014 Jun 25.

15.

Regulation of the activation of the Fanconi anemia pathway by the p21 cyclin-dependent kinase inhibitor.

Rego MA, Harney JA, Mauro M, Shen M, Howlett NG.

Oncogene. 2012 Jan 19;31(3):366-75. doi: 10.1038/onc.2011.237. Epub 2011 Jun 20.

16.

The Fanconi anemia proteins FANCD2 and FANCJ interact and regulate each other's chromatin localization.

Chen X, Wilson JB, McChesney P, Williams SA, Kwon Y, Longerich S, Marriott AS, Sung P, Jones NJ, Kupfer GM.

J Biol Chem. 2014 Sep 12;289(37):25774-82. doi: 10.1074/jbc.M114.552570. Epub 2014 Jul 28.

17.

siRNA depletion of BRCA1, but not BRCA2, causes increased genome instability in Fanconi anemia cells.

Bruun D, Folias A, Akkari Y, Cox Y, Olson S, Moses R.

DNA Repair (Amst). 2003 Sep 18;2(9):1007-13.

PMID:
12967657
18.

Direct interaction of the Fanconi anaemia protein FANCG with BRCA2/FANCD1.

Hussain S, Witt E, Huber PA, Medhurst AL, Ashworth A, Mathew CG.

Hum Mol Genet. 2003 Oct 1;12(19):2503-10. Epub 2003 Aug 5.

PMID:
12915460
19.

Inhibition of BACH1 (FANCJ) helicase by backbone discontinuity is overcome by increased motor ATPase or length of loading strand.

Gupta R, Sharma S, Doherty KM, Sommers JA, Cantor SB, Brosh RM Jr.

Nucleic Acids Res. 2006;34(22):6673-83. Epub 2006 Dec 1.

20.

HP1 regulates the localization of FANCJ at sites of DNA double-strand breaks.

Wu W, Togashi Y, Johmura Y, Miyoshi Y, Nobuoka S, Nakanishi M, Ohta T.

Cancer Sci. 2016 Oct;107(10):1406-1415. doi: 10.1111/cas.13008. Epub 2016 Sep 1.

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