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Lyme arthritis synovial gammadelta T cells instruct dendritic cells via fas ligand.

Collins C, Wolfe J, Roessner K, Shi C, Sigal LH, Budd RC.

J Immunol. 2005 Nov 1;175(9):5656-65.


High expression of Fas ligand by synovial fluid-derived gamma delta T cells in Lyme arthritis.

Roessner K, Wolfe J, Shi C, Sigal LH, Huber S, Budd RC.

J Immunol. 2003 Mar 1;170(5):2702-10.


The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways.

Kataoka T, Budd RC, Holler N, Thome M, Martinon F, Irmler M, Burns K, Hahne M, Kennedy N, Kovacsovics M, Tschopp J.

Curr Biol. 2000 Jun 1;10(11):640-8.


Lyme arthritis synovial gamma delta T cells respond to Borrelia burgdorferi lipoproteins and lipidated hexapeptides.

Vincent MS, Roessner K, Sellati T, Huston CD, Sigal LH, Behar SM, Radolf JD, Budd RC.

J Immunol. 1998 Nov 15;161(10):5762-71.


Apoptosis of Fashigh CD4+ synovial T cells by borrelia-reactive Fas-ligand(high) gamma delta T cells in Lyme arthritis.

Vincent MS, Roessner K, Lynch D, Wilson D, Cooper SM, Tschopp J, Sigal LH, Budd RC.

J Exp Med. 1996 Dec 1;184(6):2109-17.


Regulation of the Fas death pathway by FLICE-inhibitory protein in primary human B cells.

Hennino A, Berard M, Casamayor-Pallejà M, Krammer PH, Defrance T.

J Immunol. 2000 Sep 15;165(6):3023-30.


Fas-associated death domain protein (FADD) and caspase-8 mediate up-regulation of c-Fos by Fas ligand and tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) via a FLICE inhibitory protein (FLIP)-regulated pathway.

Siegmund D, Mauri D, Peters N, Juo P, Thome M, Reichwein M, Blenis J, Scheurich P, Tschopp J, Wajant H.

J Biol Chem. 2001 Aug 31;276(35):32585-90. Epub 2001 May 30.


Latent sensitivity to Fas-mediated apoptosis after CD40 ligation may explain activity of CD154 gene therapy in chronic lymphocytic leukemia.

Chu P, Deforce D, Pedersen IM, Kim Y, Kitada S, Reed JC, Kipps TJ.

Proc Natl Acad Sci U S A. 2002 Mar 19;99(6):3854-9. Epub 2002 Mar 12.


Dendritic cells are resistant to apoptosis through the Fas (CD95/APO-1) pathway.

Ashany D, Savir A, Bhardwaj N, Elkon KB.

J Immunol. 1999 Nov 15;163(10):5303-11.


Pneumocystis cell wall beta-glucans induce dendritic cell costimulatory molecule expression and inflammatory activation through a Fas-Fas ligand mechanism.

Carmona EM, Vassallo R, Vuk-Pavlovic Z, Standing JE, Kottom TJ, Limper AH.

J Immunol. 2006 Jul 1;177(1):459-67.


Fas ligand (CD95L) and B7 expression on dendritic cells provide counter-regulatory signals for T cell survival and proliferation.

Lu L, Qian S, Hershberger PA, Rudert WA, Lynch DH, Thomson AW.

J Immunol. 1997 Jun 15;158(12):5676-84.


Human astrocytes are resistant to Fas ligand and tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis.

Song JH, Bellail A, Tse MC, Yong VW, Hao C.

J Neurosci. 2006 Mar 22;26(12):3299-308. Erratum in: J Neurosci. 2006 May 3;26(18):4983.


Synovial fluid transforming growth factor beta inhibits dendritic cell-T lymphocyte interactions in patients with chronic arthritis.

Summers KL, O'Donnell JL, Heiser A, Highton J, Hart DN.

Arthritis Rheum. 1999 Mar;42(3):507-18.


Activation of gamma delta T cells by Borrelia burgdorferi is indirect via a TLR- and caspase-dependent pathway.

Collins C, Shi C, Russell JQ, Fortner KA, Budd RC.

J Immunol. 2008 Aug 15;181(4):2392-8.


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