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Hemodynamic profile, responsiveness to anandamide, and baroreflex sensitivity of mice lacking fatty acid amide hydrolase.

Pacher P, Bátkai S, Osei-Hyiaman D, Offertáler L, Liu J, Harvey-White J, Brassai A, Járai Z, Cravatt BF, Kunos G.

Am J Physiol Heart Circ Physiol. 2005 Aug;289(2):H533-41. Epub 2005 Apr 8.


Decreased age-related cardiac dysfunction, myocardial nitrative stress, inflammatory gene expression, and apoptosis in mice lacking fatty acid amide hydrolase.

Bátkai S, Rajesh M, Mukhopadhyay P, Haskó G, Liaudet L, Cravatt BF, Csiszár A, Ungvári Z, Pacher P.

Am J Physiol Heart Circ Physiol. 2007 Aug;293(2):H909-18. Epub 2007 Apr 13.


Haemodynamic profile and responsiveness to anandamide of TRPV1 receptor knock-out mice.

Pacher P, Bátkai S, Kunos G.

J Physiol. 2004 Jul 15;558(Pt 2):647-57. Epub 2004 Apr 30.


Phenotypic assessment of THC discriminative stimulus properties in fatty acid amide hydrolase knockout and wildtype mice.

Walentiny DM, Vann RE, Wiley JL.

Neuropharmacology. 2015 Jun;93:237-42. doi: 10.1016/j.neuropharm.2015.02.004. Epub 2015 Feb 16.


FAAH-/- mice display differential tolerance, dependence, and cannabinoid receptor adaptation after delta 9-tetrahydrocannabinol and anandamide administration.

Falenski KW, Thorpe AJ, Schlosburg JE, Cravatt BF, Abdullah RA, Smith TH, Selley DE, Lichtman AH, Sim-Selley LJ.

Neuropsychopharmacology. 2010 Jul;35(8):1775-87. doi: 10.1038/npp.2010.44. Epub 2010 Mar 31.


Inhibition of fatty acid amide hydrolase unmasks CB1 receptor and TRPV1 channel-mediated modulation of glutamatergic synaptic transmission in midbrain periaqueductal grey.

Kawahara H, Drew GM, Christie MJ, Vaughan CW.

Br J Pharmacol. 2011 Jul;163(6):1214-22. doi: 10.1111/j.1476-5381.2010.01157.x.


Endocannabinoids acting at cannabinoid-1 receptors regulate cardiovascular function in hypertension.

Bátkai S, Pacher P, Osei-Hyiaman D, Radaeva S, Liu J, Harvey-White J, Offertáler L, Mackie K, Rudd MA, Bukoski RD, Kunos G.

Circulation. 2004 Oct 5;110(14):1996-2002. Epub 2004 Sep 27.


Assessment of anandamide's pharmacological effects in mice deficient of both fatty acid amide hydrolase and cannabinoid CB1 receptors.

Wise LE, Shelton CC, Cravatt BF, Martin BR, Lichtman AH.

Eur J Pharmacol. 2007 Feb 14;557(1):44-8. Epub 2006 Nov 10.


Anandamide transport is independent of fatty-acid amide hydrolase activity and is blocked by the hydrolysis-resistant inhibitor AM1172.

Fegley D, Kathuria S, Mercier R, Li C, Goutopoulos A, Makriyannis A, Piomelli D.

Proc Natl Acad Sci U S A. 2004 Jun 8;101(23):8756-61. Epub 2004 May 11.


Inhibition of fatty-acid amide hydrolase accelerates acquisition and extinction rates in a spatial memory task.

Varvel SA, Wise LE, Niyuhire F, Cravatt BF, Lichtman AH.

Neuropsychopharmacology. 2007 May;32(5):1032-41. Epub 2006 Oct 18.


Mice lacking fatty acid amide hydrolase exhibit a cannabinoid receptor-mediated phenotypic hypoalgesia.

Lichtman AH, Shelton CC, Advani T, Cravatt BF.

Pain. 2004 Jun;109(3):319-27.


Fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury.

Mukhopadhyay P, Horváth B, Rajesh M, Matsumoto S, Saito K, Bátkai S, Patel V, Tanchian G, Gao RY, Cravatt BF, Haskó G, Pacher P.

Free Radic Biol Med. 2011 Jan 1;50(1):179-95. doi: 10.1016/j.freeradbiomed.2010.11.002. Epub 2010 Nov 9.


Pharmacological activity of fatty acid amides is regulated, but not mediated, by fatty acid amide hydrolase in vivo.

Lichtman AH, Hawkins EG, Griffin G, Cravatt BF.

J Pharmacol Exp Ther. 2002 Jul;302(1):73-9.


The fatty acid amide hydrolase (FAAH) inhibitor PF-3845 acts in the nervous system to reverse LPS-induced tactile allodynia in mice.

Booker L, Kinsey SG, Abdullah RA, Blankman JL, Long JZ, Ezzili C, Boger DL, Cravatt BF, Lichtman AH.

Br J Pharmacol. 2012 Apr;165(8):2485-96. doi: 10.1111/j.1476-5381.2011.01445.x.


Comparison of anandamide transport in FAAH wild-type and knockout neurons: evidence for contributions by both FAAH and the CB1 receptor to anandamide uptake.

Ortega-Gutiérrez S, Hawkins EG, Viso A, López-Rodríguez ML, Cravatt BF.

Biochemistry. 2004 Jun 29;43(25):8184-90.


Attenuation of experimental autoimmune hepatitis by exogenous and endogenous cannabinoids: involvement of regulatory T cells.

Hegde VL, Hegde S, Cravatt BF, Hofseth LJ, Nagarkatti M, Nagarkatti PS.

Mol Pharmacol. 2008 Jul;74(1):20-33. doi: 10.1124/mol.108.047035. Epub 2008 Apr 3.


Fatty acid amide hydrolase: an emerging therapeutic target in the endocannabinoid system.

Cravatt BF, Lichtman AH.

Curr Opin Chem Biol. 2003 Aug;7(4):469-75. Review.


Increased seizure susceptibility and proconvulsant activity of anandamide in mice lacking fatty acid amide hydrolase.

Clement AB, Hawkins EG, Lichtman AH, Cravatt BF.

J Neurosci. 2003 May 1;23(9):3916-23.


Inhibitor of fatty acid amide hydrolase normalizes cardiovascular function in hypertension without adverse metabolic effects.

Godlewski G, Alapafuja SO, Bátkai S, Nikas SP, Cinar R, Offertáler L, Osei-Hyiaman D, Liu J, Mukhopadhyay B, Harvey-White J, Tam J, Pacak K, Blankman JL, Cravatt BF, Makriyannis A, Kunos G.

Chem Biol. 2010 Nov 24;17(11):1256-66. doi: 10.1016/j.chembiol.2010.08.013.

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