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Items: 1 to 20 of 193

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Relationship between K-ras mutation and the expression of p21WAF1/CIP1 and p53 in chronic pancreatitis and pancreatic adenocarcinoma.

Hermanová M, Lukás Z, Kroupová I, Kleibl Z, Novotný J, Nenutil R, Pazourková M, Brázdil J, Kren L, Díte P.

Neoplasma. 2003;50(5):319-25.

PMID:
14628083
3.

Lack of apoptosis in PanIN-1 and PanIN-2 lesions associated with pancreatic ductal adenocarcinoma is not dependent on K-ras status.

Lüttges J, Neumann S, Jesenofsky R, Borries V, Löhr M, Klöppel G.

Pancreas. 2003 Oct;27(3):e57-62. Erratum in: Pancreas. 2012 Mar;41(2):338. Jesnowski, Ralf [corrected to Jesenofsky, Ralf].

PMID:
14508142
4.

Detection of K-ras mutations in the stool of patients with pancreatic adenocarcinoma and pancreatic ductal hyperplasia.

Caldas C, Hahn SA, Hruban RH, Redston MS, Yeo CJ, Kern SE.

Cancer Res. 1994 Jul 1;54(13):3568-73.

5.

Neoplasms of the ampulla of vater with concurrent pancreatic intraductal neoplasia: a histological and molecular study.

Agoff SN, Crispin DA, Bronner MP, Dail DH, Hawes SE, Haggitt RC.

Mod Pathol. 2001 Mar;14(3):139-46.

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p16 Inactivation in pancreatic intraepithelial neoplasias (PanINs) arising in patients with chronic pancreatitis.

Rosty C, Geradts J, Sato N, Wilentz RE, Roberts H, Sohn T, Cameron JL, Yeo CJ, Hruban RH, Goggins M.

Am J Surg Pathol. 2003 Dec;27(12):1495-501.

PMID:
14657708
9.

The role of p21ras in pancreatic neoplasia and chronic pancreatitis.

Mulligan NJ, Yang S, Andry C, Klein M, O'Brien MJ.

Hum Pathol. 1999 Jun;30(6):602-10.

PMID:
10374765
11.

Predictive factors for pancreatic cancer in patients with chronic pancreatitis in association with K-ras gene mutation.

Arvanitakis M, Van Laethem JL, Parma J, De Maertelaer V, Delhaye M, Devière J.

Endoscopy. 2004 Jun;36(6):535-42.

PMID:
15202051
12.

K-ras mutations in the duodenal fluid of patients with pancreatic carcinoma.

Wilentz RE, Chung CH, Sturm PD, Musler A, Sohn TA, Offerhaus GJ, Yeo CJ, Hruban RH, Slebos RJ.

Cancer. 1998 Jan 1;82(1):96-103.

PMID:
9428484
13.
14.

Different amounts of K-ras mutant epithelial cells in pancreatic carcinoma and mass-forming pancreatitis.

Matsubayashi H, Watanabe H, Ajioka Y, Nishikura K, Yamano M, Seki T, Saito T, Matsubayashi T.

Pancreas. 2000 Jul;21(1):77-85.

PMID:
10881936
15.

Clonality and field cancerization in intraductal papillary-mucinous tumors of the pancreas.

Izawa T, Obara T, Tanno S, Mizukami Y, Yanagawa N, Kohgo Y.

Cancer. 2001 Oct 1;92(7):1807-17.

PMID:
11745253
16.

Common occurrence of multiple K-RAS mutations in pancreatic cancers with associated precursor lesions and in biliary cancers.

Laghi L, Orbetegli O, Bianchi P, Zerbi A, Di Carlo V, Boland CR, Malesci A.

Oncogene. 2002 Jun 20;21(27):4301-6.

17.

Prevalence of activating K-ras mutations in the evolutionary stages of neoplasia in intraductal papillary mucinous tumors of the pancreas.

Z'graggen K, Rivera JA, Compton CC, Pins M, Werner J, Fernández-del Castillo C, Rattner DW, Lewandrowski KB, Rustgi AK, Warshaw AL.

Ann Surg. 1997 Oct;226(4):491-8; discussion 498-500.

18.

Tumor angiogenesis in chronic pancreatitis and pancreatic adenocarcinoma: impact of K-ras mutations.

Banerjee SK, Zoubine MN, Mullick M, Weston AP, Cherian R, Campbell DR.

Pancreas. 2000 Apr;20(3):248-55.

PMID:
10766450
19.

Pathology analysis reveals that dysplastic pancreatic ductal lesions are frequent in patients with hereditary pancreatitis.

Rebours V, Lévy P, Mosnier JF, Scoazec JY, Soubeyrand MS, Fléjou JF, Turlin B, Hammel P, Ruszniewski P, Bedossa P, Couvelard A.

Clin Gastroenterol Hepatol. 2010 Feb;8(2):206-12. doi: 10.1016/j.cgh.2009.09.009. Epub 2009 Sep 16.

PMID:
19765677
20.

K-ras mutation in focal proliferative lesions of human pancreas.

Terhune PG, Phifer DM, Tosteson TD, Longnecker DS.

Cancer Epidemiol Biomarkers Prev. 1998 Jun;7(6):515-21.

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