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Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis.

Kreutmayer S, Csordas A, Kern J, Maass V, Almanzar G, Offterdinger M, Öllinger R, Maass M, Wick G.

Cell Stress Chaperones. 2013 May;18(3):259-68. doi: 10.1007/s12192-012-0378-7. Epub 2012 Nov 29.


Chlamydia pneumoniae stimulates proliferation of vascular smooth muscle cells through induction of endogenous heat shock protein 60.

Hirono S, Dibrov E, Hurtado C, Kostenuk A, Ducas R, Pierce GN.

Circ Res. 2003 Oct 17;93(8):710-6. Epub 2003 Sep 18.


Requirement for NF-kappaB in transcriptional activation of monocyte chemotactic protein 1 by Chlamydia pneumoniae in human endothelial cells.

Molestina RE, Miller RD, Lentsch AB, Ramirez JA, Summersgill JT.

Infect Immun. 2000 Jul;68(7):4282-8.


Chlamydia pneumoniae heat shock protein 60 is associated with apoptotic signaling pathway in human atheromatous plaques of coronary artery disease patients.

Jha HC, Srivastava P, Vardhan H, Singh LC, Bhengraj AR, Prasad J, Mittal A.

J Cardiol. 2011 Nov;58(3):216-25. doi: 10.1016/j.jjcc.2011.07.010. Epub 2011 Sep 1.


Chlamydia pneumoniae infection of aortic smooth muscle cells reduces platelet-derived growth factor receptor-beta expression.

Rödel J, Lehmann M, Vogelsang H, Straube E.

FEMS Immunol Med Microbiol. 2007 Nov;51(2):363-71. Epub 2007 Aug 29.


Infection of U937 monocytic cells with Chlamydia pneumoniae induces extensive changes in host cell gene expression.

Virok D, Loboda A, Kari L, Nebozhyn M, Chang C, Nichols C, Endresz V, Gonczol E, Berencsi K, Showe MK, Showe LC.

J Infect Dis. 2003 Nov 1;188(9):1310-21. Epub 2003 Oct 13.


Chlamydia pneumoniae disrupts lipid metabolism in human umbilical vein endothelial cells.

Sun S, Cheng B, Wu X, Wu Q, Qi B, Wu J, He P.

Mol Med Rep. 2014 Aug;10(2):1150-6. doi: 10.3892/mmr.2014.2295. Epub 2014 Jun 3.


Up-regulation of host cell genes during interferon-gamma-induced persistent Chlamydia pneumoniae infection in HL cells.

Mannonen L, Nikula T, Haveri A, Reinikainen A, Vuola JM, Lahesmaa R, Puolakkainen M.

J Infect Dis. 2007 Jan 15;195(2):212-9. Epub 2006 Dec 13.


GroEL1, a heat shock protein 60 of Chlamydia pneumoniae, induces lectin-like oxidized low-density lipoprotein receptor 1 expression in endothelial cells and enhances atherogenesis in hypercholesterolemic rabbits.

Lin FY, Lin YW, Huang CY, Chang YJ, Tsao NW, Chang NC, Ou KL, Chen TL, Shih CM, Chen YH.

J Immunol. 2011 Apr 1;186(7):4405-14. doi: 10.4049/jimmunol.1003116. Epub 2011 Mar 7.


Growth factor production in human endothelial cells after Chlamydia pneumoniae infection.

Prochnau D, Rödel J, Hartmann M, Straube E, Figulla HR.

Int J Med Microbiol. 2004 Jul;294(1):53-7.


Nod1-mediated endothelial cell activation by Chlamydophila pneumoniae.

Opitz B, Förster S, Hocke AC, Maass M, Schmeck B, Hippenstiel S, Suttorp N, Krüll M.

Circ Res. 2005 Feb 18;96(3):319-26. Epub 2005 Jan 13.


Study of heat shock response of human umbilical vein endothelial cells (HUVECs) using cDNA microarray.

Zhou M, Zhang A, Lin B, Liu J, Xu LX.

Int J Hyperthermia. 2007 May;23(3):225-58.


Recruitment of Chlamydia pneumoniae-infected macrophages to the carotid artery wall in noninfected, nonatherosclerotic mice.

May AE, Redecke V, Grüner S, Schmidt R, Massberg S, Miethke T, Ryba B, Prazeres da Costa C, Schömig A, Neumann FJ.

Arterioscler Thromb Vasc Biol. 2003 May 1;23(5):789-94. Epub 2003 Mar 27.


Delivery of Chlamydia pneumoniae to the vessel wall aggravates atherosclerosis in LDLr-/- mice.

Hauer AD, de Vos P, Peterse N, ten Cate H, van Berkel TJ, Stassen FR, Kuiper J.

Cardiovasc Res. 2006 Jan;69(1):280-8. Epub 2005 Aug 19.


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