Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 110


Protection against CD95-induced apoptosis by chlamydial infection at a mitochondrial step.

Fischer SF, Harlander T, Vier J, Häcker G.

Infect Immun. 2004 Feb;72(2):1107-15.


Differential role of caspase-8 and BID activation during radiation- and CD95-induced apoptosis.

Belka C, Rudner J, Wesselborg S, Stepczynska A, Marini P, Lepple-Wienhues A, Faltin H, Bamberg M, Budach W, Schulze-Osthoff K.

Oncogene. 2000 Feb 24;19(9):1181-90.


Requirement for aspartate-cleaved bid in apoptosis signaling by DNA-damaging anti-cancer regimens.

Werner AB, Tait SW, de Vries E, Eldering E, Borst J.

J Biol Chem. 2004 Jul 2;279(27):28771-80. Epub 2004 Apr 26.


Characterization of antiapoptotic activities of Chlamydia pneumoniae in human cells.

Fischer SF, Schwarz C, Vier J, Häcker G.

Infect Immun. 2001 Nov;69(11):7121-9.


Bovine ephemeral fever virus-induced apoptosis requires virus gene expression and activation of Fas and mitochondrial signaling pathway.

Lin CH, Shih WL, Lin FL, Hsieh YC, Kuo YR, Liao MH, Liu HJ.

Apoptosis. 2009 Jul;14(7):864-77. doi: 10.1007/s10495-009-0371-5. Erratum in: Apoptosis. 2009 Sep;14(9):1134. Liu, Hung-Jeng [corrected to Liu, Hung-Jen].


p53 potentiation of tumor cell susceptibility to CTL involves Fas and mitochondrial pathways.

Thiery J, Abouzahr S, Dorothee G, Jalil A, Richon C, Vergnon I, Mami-Chouaib F, Chouaib S.

J Immunol. 2005 Jan 15;174(2):871-8.


Fas/CD95-mediated apoptosis of type II cells is blocked by Toxoplasma gondii primarily via interference with the mitochondrial amplification loop.

Hippe D, Lytovchenko O, Schmitz I, Lüder CG.

Infect Immun. 2008 Jul;76(7):2905-12. doi: 10.1128/IAI.01546-07. Epub 2008 Apr 14.


Apoptotic effects of mahanine on human leukemic cells are mediated through crosstalk between Apo-1/Fas signaling and the Bid protein and via mitochondrial pathways.

Bhattacharya K, Samanta SK, Tripathi R, Mallick A, Chandra S, Pal BC, Shaha C, Mandal C.

Biochem Pharmacol. 2010 Feb 1;79(3):361-72. doi: 10.1016/j.bcp.2009.09.007. Epub 2009 Sep 12.


Mitochondria from TRAIL-resistant prostate cancer cells are capable of responding to apoptotic stimuli.

Liang Y, Eid MA, Lewis RW, Kumar MV.

Cell Signal. 2005 Feb;17(2):243-51.


Effects of Bcl-2 levels on Fas signaling-induced caspase-3 activation: molecular genetic tests of computational model predictions.

Hua F, Cornejo MG, Cardone MH, Stokes CL, Lauffenburger DA.

J Immunol. 2005 Jul 15;175(2):985-95. Erratum in: J Immunol. 2005 Nov 1;175(9):6235-7.


Functional dissociation of DeltaPsim and cytochrome c release defines the contribution of mitochondria upstream of caspase activation during granzyme B-induced apoptosis.

Waterhouse NJ, Sedelies KA, Sutton VR, Pinkoski MJ, Thia KY, Johnstone R, Bird PI, Green DR, Trapani JA.

Cell Death Differ. 2006 Apr;13(4):607-18.


A critical role of glutathione in determining apoptosis sensitivity and resistance in leukemia cells.

Friesen C, Kiess Y, Debatin KM.

Cell Death Differ. 2004 Jul;11 Suppl 1:S73-85.

Supplemental Content

Support Center