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Items: 1 to 20 of 1254

1.
2.

Altered sodium channel expression in second-order spinal sensory neurons contributes to pain after peripheral nerve injury.

Hains BC, Saab CY, Klein JP, Craner MJ, Waxman SG.

J Neurosci. 2004 May 19;24(20):4832-9.

3.

Changes in electrophysiological properties and sodium channel Nav1.3 expression in thalamic neurons after spinal cord injury.

Hains BC, Saab CY, Waxman SG.

Brain. 2005 Oct;128(Pt 10):2359-71. Epub 2005 Aug 18.

PMID:
16109750
4.

Upregulation of the voltage-gated sodium channel beta2 subunit in neuropathic pain models: characterization of expression in injured and non-injured primary sensory neurons.

Pertin M, Ji RR, Berta T, Powell AJ, Karchewski L, Tate SN, Isom LL, Woolf CJ, Gilliard N, Spahn DR, Decosterd I.

J Neurosci. 2005 Nov 23;25(47):10970-80.

5.

The Cav2.3 calcium channel antagonist SNX-482 reduces dorsal horn neuronal responses in a rat model of chronic neuropathic pain.

Matthews EA, Bee LA, Stephens GJ, Dickenson AH.

Eur J Neurosci. 2007 Jun;25(12):3561-9.

PMID:
17610575
6.

Neuropathic pain memory is maintained by Rac1-regulated dendritic spine remodeling after spinal cord injury.

Tan AM, Stamboulian S, Chang YW, Zhao P, Hains AB, Waxman SG, Hains BC.

J Neurosci. 2008 Dec 3;28(49):13173-83. doi: 10.1523/JNEUROSCI.3142-08.2008.

7.

Relationship between sodium channel NaV1.3 expression and neuropathic pain behavior in rats.

Lindia JA, Köhler MG, Martin WJ, Abbadie C.

Pain. 2005 Sep;117(1-2):145-53.

PMID:
16061326
8.

Sodium channel expression and the molecular pathophysiology of pain after SCI.

Hains BC, Waxman SG.

Prog Brain Res. 2007;161:195-203. Review.

PMID:
17618978
9.

TNF-α contributes to up-regulation of Nav1.3 and Nav1.8 in DRG neurons following motor fiber injury.

He XH, Zang Y, Chen X, Pang RP, Xu JT, Zhou X, Wei XH, Li YY, Xin WJ, Qin ZH, Liu XG.

Pain. 2010 Nov;151(2):266-79. doi: 10.1016/j.pain.2010.06.005. Epub 2010 Jul 17.

PMID:
20638792
10.

Expression of central glucocorticoid receptors after peripheral nerve injury contributes to neuropathic pain behaviors in rats.

Wang S, Lim G, Zeng Q, Sung B, Ai Y, Guo G, Yang L, Mao J.

J Neurosci. 2004 Sep 29;24(39):8595-605.

11.
12.

Involvement of the TTX-resistant sodium channel Nav 1.8 in inflammatory and neuropathic, but not post-operative, pain states.

Joshi SK, Mikusa JP, Hernandez G, Baker S, Shieh CC, Neelands T, Zhang XF, Niforatos W, Kage K, Han P, Krafte D, Faltynek C, Sullivan JP, Jarvis MF, Honore P.

Pain. 2006 Jul;123(1-2):75-82. Epub 2006 Mar 20.

PMID:
16545521
13.

Upregulation of persistent and ramp sodium current in dorsal horn neurons after spinal cord injury.

Lampert A, Hains BC, Waxman SG.

Exp Brain Res. 2006 Oct;174(4):660-6. Epub 2006 May 23.

PMID:
16718433
14.

Serotonergic neural precursor cell grafts attenuate bilateral hyperexcitability of dorsal horn neurons after spinal hemisection in rat.

Hains BC, Johnson KM, Eaton MJ, Willis WD, Hulsebosch CE.

Neuroscience. 2003;116(4):1097-110.

PMID:
12617951
15.

Bilateral downregulation of Nav1.8 in dorsal root ganglia of rats with bone cancer pain induced by inoculation with Walker 256 breast tumor cells.

Miao XR, Gao XF, Wu JX, Lu ZJ, Huang ZX, Li XQ, He C, Yu WF.

BMC Cancer. 2010 May 20;10:216. doi: 10.1186/1471-2407-10-216.

16.

Contralateral neuropathic pain and neuropathology in dorsal root ganglion and spinal cord following hemilateral nerve injury in rats.

Hatashita S, Sekiguchi M, Kobayashi H, Konno S, Kikuchi S.

Spine (Phila Pa 1976). 2008 May 20;33(12):1344-51. doi: 10.1097/BRS.0b013e3181733188.

PMID:
18496347
17.

Alterations in burst firing of thalamic VPL neurons and reversal by Na(v)1.3 antisense after spinal cord injury.

Hains BC, Saab CY, Waxman SG.

J Neurophysiol. 2006 Jun;95(6):3343-52. Epub 2006 Feb 15.

18.
19.

Role of the spinal cord NR2B-containing NMDA receptors in the development of neuropathic pain.

Qu XX, Cai J, Li MJ, Chi YN, Liao FF, Liu FY, Wan Y, Han JS, Xing GG.

Exp Neurol. 2009 Feb;215(2):298-307. doi: 10.1016/j.expneurol.2008.10.018. Epub 2008 Nov 12.

PMID:
19046970
20.

Inhibition of neuropathic pain by decreased expression of the tetrodotoxin-resistant sodium channel, NaV1.8.

Lai J, Gold MS, Kim CS, Bian D, Ossipov MH, Hunter JC, Porreca F.

Pain. 2002 Jan;95(1-2):143-52.

PMID:
11790477

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